Evidence mapPaperPMID 11270938Full record

ReviewDrugs2001

Safety profiles for the HMG-CoA reductase inhibitors: treatment and trust.

M H Davidson

Registry-linked trialAbstract readReview
PubMed Publisher
In one paragraph

Review in Drugs, 2001. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT06767774 (Comparison of Pitavastatin Plus Ezetimibe Versus High-Intensity Statin Therapy on Risk of New-Onset Diabetes Mellitus in Prediabetic Patients With Atherosclerotic Cardiovascular Disease), which is not on this map. Cited by 26 papers.

0numbers the graph read from it
0cells of the map it votes in
26citing papers in PubMed
11.0field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT06767774 phase4recruitingstarted 2025, after this paper: background citation

Comparison of Pitavastatin Plus Ezetimibe Versus High-Intensity Statin Therapy on Risk of New-Onset Diabetes Mellitus in Prediabetic Patients With Atherosclerotic Cardiovascular Disease

Ran2025Enrolled2,000Registered outcomes13Posted comparisons0ConditionsASCVD, Diabetes, StatinArmsCombination therapy, High intensity statin monotherapy
Open the trial in the graph
3 · Its place in the literature

Who cites it

26 citing papers in PubMed, 109 citations in OpenAlex.

  1. Cholesterol in Mitochondrial Diseases-Friend or Foe?International journal of molecular sciences · 2026
    Review
  2. Article
  3. Review
  4. Article
  5. Review
  6. Review
  7. Article
  8. Review
  9. HMGCR is necessary for the tumorigenecity of esophageal squamous cell carcinoma and is regulated by Myc.Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine · 2014
    Article
  10. Article
  11. Mevalonate pathway is a therapeutic target in esophageal squamous cell carcinoma.Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine · 2013
    Article
  12. Article
  13. Article
  14. Dysregulation of the mevalonate pathway promotes transformation.Proceedings of the National Academy of Sciences of the United States of America · 2010
    Article
  15. Statin adverse effects : a review of the literature and evidence for a mitochondrial mechanism.American journal of cardiovascular drugs : drugs, devices, and other interventions · 2008
    Review
  16. Review
  17. Review
  18. Effects of atorvastatin on higher functions.European journal of clinical pharmacology · 2006
    Article
  19. Review
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author at 1 institution in 1 country.

M H DavidsonChicago Center for Clinical Research, Illinois 60610, USA. mdavidson@protocare.com
Center for Clinical Research (United States) · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Hypercholesterolaemia is a chronic condition that often requires life-long treatment, making the safety of lipid-lowering drugs a critical issue. 3-hydroxy-3-methylglutaryl coenzyme A (HMG-CoA) reductase inhibitors ('statins') are commonly used as the pharmacotherapeutic treatment of choice for patients with hypercholesterolaemia. These agents have consistently demonstrated a positive safety and tolerability profile, and are recommended by the US National Cholesterol Education Program guidelines and by the European Joint Task Force for Prevention of Coronary Heart Disease to be used after, or in addition to, a first-line approach with diet. Several large-scale clinical trials have shown HMG-CoA reductase inhibitors to be efficacious and well tolerated, and to be associated with a low rate of treatment withdrawal due to adverse events. These studies included mortality and morbidity end-points, and comprised both primary- and secondary-prevention trials. Hepatic, renal and muscular systems are rarely affected during HMG-CoA reductase inhibitor therapy and the few drug interactions that can occur with concomitantly administered drugs are well documented. There is no conclusive evidence linking HMG-CoA reductase inhibitors to the development of cancer in humans. In long term studies with various HMG-CoA reductase inhibitors, there was no increase in cancer rates compared with placebo. Thus, it can be concluded that HMG-CoA reductase inhibitors are well tolerated, effective treatments for hypercholesterolaemia.

Indexed as

Anticholesteremic AgentsClinical Trials as TopicContraindicationsCoronary DiseaseHumansHydroxymethylglutaryl-CoA Reductase InhibitorsHypercholesterolemiaNaphthalenesStrokeViolenceAnticholesteremic AgentsHydroxymethylglutaryl-CoA Reductase InhibitorsNaphthalenes

Identifiers

PMID11270938
OpenAlexW2059510434

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.