Evidence map›Paper›PMID 14525676›Full record

ReviewCurrent atherosclerosis reports2003

Postprandial lipemia and cardiovascular disease.

Dianne Hyson, John C Rutledge, Lars Berglund

Abstract readReview
PubMed Publisher
In one paragraph

Review in Current atherosclerosis reports, 2003. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 54 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
54citing papers in PubMed, 2 pooled it
4.4field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

54 citing papers in PubMed, 2 syntheses or guidelines pooled it, 161 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Dianne HysonDepartment of Medicine, University of California, Davis, 4150 V Street, Suite G400, Sacramento, CA 95817, USA.
John C Rutledge
Lars Berglund
University of California, Berkeley · US

Funding

VITAMINS, AMINO ACIDS, AND RELATED PRODUCTS CORE LABORATORYP30DK035747 · NIDDK · UNIVERSITY OF CALIFORNIA DAVIS · PI HALSTED, CHARLES HOPKINSON · 1986 to 2005
$4.7M
Regulation of Lipoprotein(a) Metabolism in Humans with Reference to EthnicityR01HL062705 · NHLBI · UNIVERSITY OF CALIFORNIA DAVIS · PI BERGLUND, LARS F · 2000 to 2011
$2.9M
LIPOPROTEIN AND ADIPOSE TISSUE METABOLISM IN HIVR01HL065938 · NHLBI · UNIVERSITY OF CALIFORNIA DAVIS · PI BERGLUND, LARS F · 2000 to 2004
$1.6M
Effects of a high glycemic load diet on vascular systemR01HL071488 · NHLBI · UNIVERSITY OF CALIFORNIA DAVIS · PI RUTLEDGE, JOHN CALVERT · 2002 to 2003
$641k
NHLBI NIH HHS HL62705NHLBI NIH HHS HL65938NHLBI NIH HHS HL71488NIDDK NIH HHS DK35747
6 · The paper itself

Abstract

Postprandial lipemia, characterized by a rise in triglyceride-rich lipoproteins after eating, is a dynamic, nonsteady-state condition in which humans spend the majority of time. There are several lines of evidence suggesting that postprandial lipemia increases risk of atherogenesis. Clinical data show a correlation between postprandial lipoproteins and the presence/progression of coronary artery disease and carotid intimal thickness. Mechanistic studies demonstrate that triglyceride-rich lipoprotein remnants may have adverse effects on endothelium and can penetrate into the subendothelial space. Exchange of core lipids between postprandial lipoproteins and low-density lipoprotein (LDL)/high-density lipoprotein (HDL) is increased during prolonged lipemia, resulting in small, dense LDL particles and reduced HDL cholesterol levels. Hemostatic variables, including clotting factors, platelet reactivity, and monocyte cytokine expression, may be increased during postprandial lipemia. Collectively, these data suggest that assessment and treatment of atherosclerosis should include parameters related to postprandial lipemia.

Indexed as

ArteriosclerosisCardiovascular DiseasesCarotid ArteriesCholesterol, HDLClinical Trials as TopicHumansHyperlipoproteinemiasLipoproteinsLipoproteins, LDLPostprandial PeriodRisk FactorsTriglyceridesTunica IntimaCholesterol, HDLLipoproteinsLipoproteins, LDLTriglycerides

Identifiers

PMID14525676
OpenAlexW2005564177

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.