Evidence map›Paper›PMID 17384993›Full record

ReviewClinical and experimental nephrology2007

Transcription factor decoy oligonucleotide-based therapeutic strategy for renal disease.

Naruya Tomita, Naoki Kashihara, Ryuichi Morishita

Abstract readReview
PubMed Publisher
In one paragraph

Review in Clinical and experimental nephrology, 2007. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.

0numbers the graph read from it
0cells of the map it votes in
15citing papers in PubMed
0.9field-weighted citation impact, top 28% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

15 citing papers in PubMed, 32 citations in OpenAlex.

  1. Trial
  2. Review
  3. Gene therapy and kidney diseases.Molecular therapy. Methods & clinical development · 2024
    Review
  4. Article
  5. Review
  6. Improving Molecular Therapy in the Kidney.Molecular diagnosis & therapy · 2020
    Review
  7. Article
  8. Transcription factor decoy: a pre-transcriptional approach for gene downregulation purpose in cancer.Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine · 2015
    Review
  9. Article
  10. Article
  11. STAT inhibitors for cancer therapy.Journal of hematology & oncology · 2013
    Review
  12. Article
  13. Article
  14. Article
  15. Nucleic acid drugs for prevention of cardiac rejection.Journal of biomedicine & biotechnology · 2009
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 1 country.

Naruya TomitaDivision of Nephrology, Department of Internal Medicine, Kawasaki Medical School, 577 Matsushima, Kurashiki, 701-0192, Japan. tomita@med.kawasaki-m.ac.jp
Naoki Kashihara
Ryuichi Morishita
Kawasaki Medical School · JPOsaka University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Renal disease, including slight renal injuries, has come to be seen as one of the risk factors for cardiovascular events. At present, most conventional therapy is inefficient, and tends to treat the symptoms rather than the underlying causes of the disorder. Gene therapy based on oligonucleotides (ODN) offers a novel approach for the prevention and treatment of renal diseases. Gene transfer into somatic cells to interfere with the pathogenesis contributing to renal disease may provide such an approach, leading to the better prevention and treatment of renal disease. The major development of gene transfer methods has made an important contribution to an intense investigation of the potential of gene therapy in renal diseases. Amazing advances in molecular biology have provided the dramatic improvement in the technology that is necessary to transfer target genes into somatic cells. Gene transfer methods, especially when mediated by several viral vectors, have improved to a surprising extent. In fact, some (retroviral vectors, adenoviral vectors, or liposome-based vectors, etc.) have already been used in clinical trials. On the other hand, recent progress in molecular biology has provided new techniques to inhibit target gene expression. The transfer of cis-element double-stranded ODN (= decoy) has been reported to be a powerful novel tool in a new class of antigene strategies for gene therapy. The transfer of decoy ODN corresponding to the cis sequence will result in attenuation of the authentic cis-trans interaction, leading to the removal of trans-factors from the endogenous cis-elements with a subsequent modulation of gene expression.

Indexed as

AnimalsGenetic TherapyKidney DiseasesOligodeoxyribonucleotidesTranscription FactorsOligodeoxyribonucleotidesTranscription Factors

Identifiers

PMID17384993
OpenAlexW1966811995

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.