Evidence mapPaperPMID 17519305Full record

Trial reportThe Journal of clinical endocrinology and metabolism2007

Concomitant reduction of low-density lipoprotein-cholesterol and biomarkers of inflammation with low-dose simvastatin therapy in patients with type 1 diabetes.

Ishwarlal Jialal, Eric Miguelino, Steven C Griffen, Sridevi Devaraj

Open access · bronzeAbstract readRandomized Controlled Trial
In one paragraph

Trial report in The Journal of clinical endocrinology and metabolism, 2007. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 21 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
21citing papers in PubMed, 2 pooled it
2.4field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

21 citing papers in PubMed, 2 syntheses or guidelines pooled it, 49 citations in OpenAlex.

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  10. Repurposing auranofin for the treatment of cutaneous staphylococcal infections.International journal of antimicrobial agents · 2016
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 1 institution in 1 country.

Ishwarlal JialalLaboratory for Atherosclerosis and Metabolic Research, University of California, Davis, Medical Center, Sacramento, California 95817, USA. ishwarlal.jialal@ucdmc.ucdavis.edu
Eric Miguelino
Steven C Griffen
Sridevi Devaraj
University of California Davis Medical Center · US

Funding

Cellular pathways of inflammation in type 1 diabetesR21DK069801 · UNIVERSITY OF CALIFORNIA DAVIS · 2004 to 2005
$747k
CLINICAL STUDIES IN NUTRITION AND METABOLISMK24AT000596 · UNIVERSITY OF TEXAS SW MED CTR/DALLAS · 2000 to 2005
$733k
NCCIH NIH HHS K24 AT000596NCCIH NIH HHS K24 AT00596NIDDK NIH HHS R21 DK069801NIDDK NIH HHS R21DK69801
6 · The paper itself

Abstract

contextCardiovascular disease is a major cause of mortality in type 1 diabetes (TIDM). TIDM is a proinflammatory state. Whereas there is consensus on lipid management in type 2 diabetes, there is a lack of data in type 1 diabetes. In addition to benefits on the lipid profile, statin therapy is antiinflammatory.

objectiveThere are scant data on statin therapy in T1DM. Thus, we tested the effect of simvastatin, compared with placebo, on biomarkers of inflammation and monocyte function in TIDM patients.

designThis was a double-blind, randomized, placebo-controlled study of T1DM patients, randomized to placebo or simvastatin, 20 mg/d for 3 months.

settingThe study was conducted at the University of California, Davis, Medical Center.

participantsParticipants included patients with T1DM. METHODS AND

resultsAnalytes measured at baseline and 3 months included liver function tests, creatinine, hemoglobin AIC, high-sensitivity C-reactive protein, soluble CD40 ligand, monocyte O(2)(-), cytokines, nuclear factor-kappaB. Simvastatin therapy resulted in significant reduction in low-density lipoprotein and non-high-density lipoprotein cholesterol, high-sensitivity C-reactive protein (18% reduction, P < 0.001) and soluble CD40 ligand (22% reduction, P < 0.05), compared with placebo. Simvastatin therapy significantly inhibited lipopolysaccharide-activated monocyte release of O(2)(-) (P < 0.0005), IL-8 (P < 0.03), and TNF (P < 0.02). Simvastatin therapy significantly inhibited monocyte IL-6 release, compared with baseline (P = 0.02). Simvastatin therapy also significantly reduced monocytic nuclear factor-kappaB p65 activity, compared with placebo (P < 0.01).

conclusionsThis study demonstrates that simvastatin (20 mg/d) is safe in T1DM patients and has concomitant benefits on the lipid profile and biomarkers of inflammation. These novel findings could have implications for developing policy guidelines for statin therapy in forestalling vascular complications in young T1DM.

Indexed as

AdultBiomarkersCholesterolCholesterol, LDLCytokinesDiabetes Mellitus, Type 1Double-Blind MethodFemaleGlycated HemoglobinHumansHydroxymethylglutaryl-CoA Reductase InhibitorsInflammationLipoproteins, LDLMaleMonocytesSimvastatinBiomarkersCholesterolCholesterol, LDLCytokinesGlycated HemoglobinHydroxymethylglutaryl-CoA Reductase InhibitorsLipoproteins, LDLSimvastatinTriglycerides

Identifiers

PMID17519305
PMCPMC2677961
OpenAlexW2149220775

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.