ReviewPancreas2008
Pancreatitis and calcium signalling: report of an international workshop.
Review in Pancreas, 2008. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 13 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
13 citing papers in PubMed.
- Acute Pancreatitis: Genetic Risk and Clinical Implications.Journal of clinical medicine · 2021Review
- The Orai CaThe Journal of physiology · 2019Article
- Pancreatic adaptive responses in alcohol abuse: Role of the unfolded protein response.Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.] · 2015Review
- Novel lipophilic probe for detecting near-membrane reactive oxygen species responses and its application for studies of pancreatic acinar cells: effects of pyocyanin and L-ornithine.Antioxidants & redox signaling · 2015Article
- The role of Ca2+ influx in endocytic vacuole formation in pancreatic acinar cells.The Biochemical journal · 2015Article
- Primary hyperparathyroidism with pancreatitis: experience of management in 5 patients with review of literature.Indian journal of gastroenterology : official journal of the Indian Society of Gastroenterology · 2014Article
- Article
- The association of primary hyperparathyroidism with pancreatitis.Journal of clinical gastroenterology · 2012Review
- Genetic and pharmacologic inhibition of the Ca2+ influx channel TRPC3 protects secretory epithelia from Ca2+-dependent toxicity.Gastroenterology · 2011Article
- Environmental and genetic stressors and the unfolded protein response in exocrine pancreatic function - a hypothesis.Frontiers in physiology · 2011Article
- Inhibitors of Bcl-2 protein family deplete ER Ca2+ stores in pancreatic acinar cells.Pflugers Archiv : European journal of physiology · 2010Article
- Deletion of TRPC3 in mice reduces store-operated Ca2+ influx and the severity of acute pancreatitis.Gastroenterology · 2009Article
- Acceleration of our understanding of recurrent acute and chronic pancreatitis.Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.]Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
Abstract
Pancreatitis and Calcium Signalling was an international research workshop organized by the authors and held at the Liverpool Medical Institution, Liverpool, United Kingdom, from Sunday 12th to Tuesday 14th November 2006. The overall goal of the workshop was to review progress and explore new opportunities for understanding the mechanisms of acute pancreatitis with an emphasis on the role of pathological calcium signaling. The participants included those with significant interest and expertise in pancreatitis research and others who are in fields outside gastroenterology but with significant expertise in areas of cell biology relevant to pancreatitis. The workshop was designed to enhance interchange of ideas and collaborations, to engage and encourage younger researchers in the field, and promote biomedical research through the participating and supporting organizations and societies. The workshop was divided into 8 topic-oriented sessions. The sessions were: (1) Physiology and pathophysiology of calcium signaling; (2) Interacting signaling mechanisms; (3) Premature digestive enzyme activation; (4) Physiology Society Lecture: Aberrant Ca2+ signaling, bicarbonate secretion, and pancreatitis; (5) NFkappaB, cytokines, and immune mechanisms; (6) Mitochondrial injury; (7) Cell death pathways; and (8) Overview of areas for future research. In each session, speakers presented work appropriate to the topic followed by discussion of the material presented by the group. The publication of these proceedings is intended to provide a platform for enhancing research and therapeutic development for acute pancreatitis.
Indexed as
Identifiers
What Socratic holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.