Evidence mapPaperPMID 18769499Full record

ArticleExperimental diabetes research2008

Phlorizin prevents glomerular hyperfiltration but not hypertrophy in diabetic rats.

Slava Malatiali, Issam Francis, Mario Barac-Nieto

Registry-linked trialOpen access · hybridAbstract read
In one paragraph

Article in Experimental diabetes research, 2008. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT02911792 (Effect of Farxiga on Renal Function and Size in Type 2 Diabetic Patients With Hyperfiltration), which is not on this map. Cited by 37 papers.

0numbers the graph read from it
0cells of the map it votes in
37citing papers in PubMed
1.8field-weighted citation impact, top 16% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT02911792 phase4completedstarted 2016, after this paper: background citation

Effect of Farxiga on Renal Function and Size in Type 2 Diabetic Patients With Hyperfiltration

Ran2016Enrolled72Registered outcomes1Posted comparisons0ConditionsDiabetes Mellitus, Type 2Armsdapagliflozin, Glipizide 5 MG, Metformin
Open the trial in the graph
3 · Its place in the literature

Who cites it

37 citing papers in PubMed, 103 citations in OpenAlex.

  1. Trial
  2. Review
  3. Article
  4. Article
  5. Article
  6. Bioactive Substances and Biological Functions inMolecules (Basel, Switzerland) · 2023
    Review
  7. Article
  8. Review
  9. Article
  10. Article
  11. Article
  12. Review
  13. Management of post-transplant diabetes: immunosuppression, early prevention, and novel antidiabetics.Transplant international : official journal of the European Society for Organ Transplantation · 2021
    Review
  14. Review
  15. Article
  16. Effect ofEXCLI journal · 2020
    Article
  17. Article
  18. Article
  19. Review
  20. SGLT2 Inhibitors as a Therapeutic Option for Diabetic Nephropathy.International journal of molecular sciences · 2017
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Slava MalatialiDepartment of Physiology, Faculty of Medicine, Kuwait University, P.O. Box 24923, Safat 13110, Kuwait. slava@hsc.edu.kw
Issam Francis
Mario Barac-Nieto
Kuwait University · KW

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The relationships of renal and glomerular hypertrophies to development of hyperfiltration and proteinuria early in streptozotocin-induced diabetes were explored. Control, diabetic, phlorizin-treated controls, and diabetic male Fischer rats were used. Phlorizin (an Na+-glucose cotransport inhibitor) was given at a dose sufficient to normalize blood glucose. Inulin clearance (C(inulin)) and protein excretion rate (PER) were measured. For morphometry, kidney sections were stained with periodic acid Schiff. At one week, diabetes PER increased 2.8-folds (P < .001), C(inulin) increased 80% (P < .01). Kidney wet and dry weights increased 10%-12% (P < .05), and glomerular tuft area increased 9.3% (P < .001). Phlorizin prevented proteinuria, hyperfiltration, and kidney hypertrophy, but not glomerular hypertrophy. Thus, hyperfiltration, proteinuria, and whole kidney hypertrophy were related to hyperglycemia but not to glomerular growth. Diabetic glomerular hypertrophy constitutes an early event in the progression of glomerular pathology which occurs in the absence of mesangial expansion and persists even after changes in protein excretion and GFR are reversed through glycemic control.

Indexed as

AnimalsBlood GlucoseDiabetes Mellitus, ExperimentalDiabetic NephropathiesGlomerular Filtration RateHypertrophyKidney GlomerulusMaleOrgan SizePhlorhizinProteinuriaRatsRats, Inbred F344Blood GlucosePhlorhizin

Identifiers

PMID18769499
PMCPMC2522335
OpenAlexW1973282559

What Socratic holds

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LicenceCC BY
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Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.