Evidence mapPaperPMID 18931099Full record

Trial reportDiabetes care2009

Dipeptidyl peptidase-4 inhibition by vildagliptin and the effect on insulin secretion and action in response to meal ingestion in type 2 diabetes.

Chiara Dalla Man, Gerlies Bock, Paula D Giesler, Denise B Serra, Monica Ligueros Saylan, James E Foley, Michael Camilleri, Gianna Toffolo, Claudio Cobelli, Robert A Rizza and 1 more

Abstract readRandomized Controlled Trial
In one paragraph

Trial report in Diabetes care, 2009. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 35 papers.

0numbers the graph read from it
0cells of the map it votes in
35citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

35 citing papers in PubMed.

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  20. Mechanisms Underlying the Pathogenesis of Isolated Impaired Glucose Tolerance in Humans.The Journal of clinical endocrinology and metabolism · 2016
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors.

Chiara Dalla ManDepartment of Information Engineering, University of Padua, Padua, Italy.
Gerlies Bock
Paula D Giesler
Denise B Serra
Monica Ligueros Saylan
James E Foley
Michael Camilleri
Gianna Toffolo
Claudio Cobelli
Robert A Rizza
Adrian Vella

Funding

Pathobiology of the Enteric SystemP01DK068055 · MAYO CLINIC · 2004 to 2005
$2.5M
MECHANISMS OF INSULIN RESISTANCE in ManR37DK029953 · MAYO CLINIC COLL OF MEDICINE, ROCHESTER · 1996 to 2003
$1.8M
MECHANISMS OF INSULIN RESISTANCE IN MANR01DK029953 · NIDDK · UNIVERSITY OF ALABAMA AT BIRMINGHAM · PI RITA BASU · 1986 to 2024
$1.6M
ALPHA 2 ADRENERGIC CONTROL IN IRRITABLE BOWEL SYNDROMEK24DK002638 · MAYO CLINIC COLL OF MEDICINE, ROCHESTER · 1999 to 2005
$709k
The regulation of fasting glucose metabolism in people with and without prediabetesR01DK078646 · MAYO CLINIC ROCHESTER · 2025 to 2025
$667k
NIDDK NIH HHS 01-DK068055NIDDK NIH HHS DK02638NIDDK NIH HHS DK078646NIDDK NIH HHS DK29953NIDDK NIH HHS K24 DK002638NIDDK NIH HHS P01 DK068055NIDDK NIH HHS R01 DK029953NIDDK NIH HHS R01 DK078646NIDDK NIH HHS R37 DK029953
6 · The paper itself

Abstract

objectiveThe purpose of this study was to determine the mechanism by which dipeptidyl peptidase-4 inhibitors lower postprandial glucose concentrations. RESEARCH DESIGN AND

methodsWe measured insulin secretion and action as well as glucose effectiveness in 14 subjects with type 2 diabetes who received vildagliptin (50 mg b.i.d.) or placebo for 10 days in random order separated by a 3-week washout. On day 9 of each period, subjects ate a mixed meal. Insulin sensitivity (S(I)), glucose effectiveness, and beta-cell responsivity indexes were estimated using the oral glucose and C-peptide minimal models. At 300 min 0.02 unit/kg insulin was administered intravenously.

resultsVildagliptin reduced postprandial glucose concentrations (905 +/- 94 vs. 1,008 +/- 104 mmol/6 h, P = 0.02). Vildagliptin did not alter net S(I) (7.71 +/- 1.28 vs. 6.41 +/- 0.84 10(-4) dl x kg(-1) x min(-1) x muU(-1) x ml(-1), P = 0.13) or glucose effectiveness (0.019 +/- 0.002 vs. 0.018 +/- 0.002 dl x kg(-1) x min(-1), P = 0.65). However, the net beta-cell responsivity index was increased (35.7 +/- 5.2 vs. 28.9 +/- 5.2 10(-9) min(-1), P = 0.03) as was total disposition index (381 +/- 48 vs. 261 +/- 35 10(-14) dl x kg(-1) x min(-2) x pmol(-1) x l(-1), P = 0.006). Vildagliptin lowered postprandial glucagon concentrations (27.0 +/- 1.1 vs. 29.7 +/- 1.5 microg x l(-1) x 6 h(-1), P = 0.03), especially after administration of exogenous insulin (81.5 +/- 6.4 vs. 99.3 +/- 5.6 ng/l, P = 0.02).

conclusionsVildagliptin lowers postprandial glucose concentrations by stimulating insulin secretion and suppressing glucagon secretion but not by altered insulin action or glucose effectiveness. A novel observation is that vildagliptin alters alpha-cell responsiveness to insulin administration, but the significance of this action is as yet unclear.

Indexed as

AdamantaneBlood GlucoseC-PeptideCross-Over StudiesDiabetes Mellitus, Type 2DigestionDipeptidyl-Peptidase IV InhibitorsDouble-Blind MethodGlucagonHumansHypoglycemic AgentsInsulinInsulin SecretionNitrilesPlacebosPostprandial PeriodAdamantaneBlood GlucoseC-PeptideDipeptidyl-Peptidase IV InhibitorsGlucagonHypoglycemic AgentsInsulinNitrilesPlacebosPyrrolidinesVildagliptin

Identifiers

PMID18931099
PMCPMC2606822

What Socratic holds

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LicenceCC BY-NC-ND
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.