Evidence mapPaperPMID 19434052Full record

ReviewCurrent opinion in nephrology and hypertension2009

Developmental programming and hypertension.

Anne Monique Nuyt, Barbara T Alexander

Open access · greenAbstract readReview
In one paragraph

Review in Current opinion in nephrology and hypertension, 2009. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 62 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
62citing papers in PubMed, 1 pooled it
23.0field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

62 citing papers in PubMed, 1 synthesis or guideline pooled it, 157 citations in OpenAlex.

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  11. Chronic hypoxia alters cardiac mitochondrial complex protein expression and activity in fetal guinea pigs in a sex-selective manner.American journal of physiology. Regulatory, integrative and comparative physiology · 2021
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2 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 2 institutions in 2 countries.

Anne Monique NuytDepartment of Pediatrics, Research Center, CHU Sainte-Justine, Université de Montréal, Canada. anne-monique.nuyt@recherche-ste-justine.qc.ca
Barbara T Alexander
Centre Hospitalier Universitaire Sainte-Justine · CAJackson Memorial Hospital · US

Funding

STRUCTURAL VASCULAR ADAPTATION OF THE MICROCIRCULATIONP01HL051971 · UNIVERSITY OF MISSISSIPPI MEDICAL CENTER · 1993 to 2005
$11.3M
Low birth weight, the kidney, and hypertensionR01HL074927 · UNIVERSITY OF MISSISSIPPI MEDICAL CENTER · 2004 to 2005
$666k
NHLBI NIH HHS HL074927NHLBI NIH HHS HL51971NHLBI NIH HHS P01 HL051971NHLBI NIH HHS R01 HL074927NIMHD NIH HHS MD002725NIMHD NIH HHS P20 MD002725
6 · The paper itself

Abstract

purpose of reviewThere is a growing body of evidence linking adverse events or exposures during early life and adult-onset diseases. After important epidemiological studies from many parts of the world, research now focuses on mechanisms of organ dysfunction and on refining the understanding of the interaction between common elements of adverse perinatal conditions, such as nutrition, oxidants, and toxins exposures. This review will focus on advances in our comprehension of developmental programming of hypertension. RECENT

findingsRecent studies have unraveled important mechanisms of oligonephronia and impaired renal function, altered vascular function and structure as well as sympathetic regulation of the cardiovascular system. Furthermore, interactions between prenatal insults and postnatal conditions are the subject of intensive research. Prematurity vs. intrauterine growth restriction modulate differently programming of high blood pressure. Along with antenatal exposure to glucocorticoids and imbalanced nutrition, a critical role for perinatal oxidative stress is emerging. SUMMARY: While the complexity of the interactions between antenatal and postnatal influences on adult blood pressure is increasingly recognized, the importance of postnatal life in (positively) modulating developmental programming offers the hope of a critical window of opportunity to reverse programming and prevent or reduce related adult-onset diseases.

Indexed as

AnimalsBirth WeightBlood VesselsDisease SusceptibilityFemaleFetal DevelopmentFetusGlucocorticoidsGonadal Steroid HormonesHumansHypertensionKidneyModels, AnimalNephronsPregnancyPremature BirthGlucocorticoidsGonadal Steroid HormonesSodium Chloride, Dietary

Identifiers

PMID19434052
PMCPMC2782679
OpenAlexW2014087264

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.