ArticleAmerican journal of human genetics2010
Principal-component analysis for assessment of population stratification in mitochondrial medical genetics.
Article in American journal of human genetics, 2010. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 31 papers, 2 of them syntheses that pooled it.
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Who cites it
31 citing papers in PubMed, 2 syntheses or guidelines pooled it.
- Mitochondrial genome-wide analysis of nuclear DNA methylation quantitative trait loci.Human molecular genetics · 2022Pooled it
- Meta-analysis identifies mitochondrial DNA sequence variants associated with walking speed.GeroScience · 2018Pooled it
- Trial
- Maternal genetic structure and population affinities of the Sichuan Han Chinese based on whole mitochondrial genomes.BMC genomics · 2026Article
- Mitochondrial-derived microproteins: from discovery to function.Trends in genetics : TIG · 2025Review
- Mitochondria-derived peptides in aging and healthspan.The Journal of clinical investigation · 2022Review
- Human genetic analyses of organelles highlight the nucleus in age-related trait heritability.eLife · 2021Article
- Examining the effect of mitochondrial DNA variants on blood pressure in two Finnish cohorts.Scientific reports · 2021Article
- Worldwide human mitochondrial haplogroup distribution from urban sewage.Scientific reports · 2019Article
- Article
- MitoIMP: A Computational Framework for Imputation of Missing Data in Low-Coverage Human Mitochondrial Genome.Bioinformatics and biology insights · 2019Article
- Complimentary Methods for Multivariate Genome-Wide Association Study Identify New Susceptibility Genes for Blood Cell Traits.Frontiers in genetics · 2019Article
- Article
- Complex spatio-temporal distribution and genomic ancestry of mitochondrial DNA haplogroups in 24,216 Danes.PloS one · 2018Article
- Mitochondrial DNA Sequence Variation Associated With Peripheral Nerve Function in the Elderly.The journals of gerontology. Series A, Biological sciences and medical sciences · 2015Article
- Mitonuclear linkage disequilibrium in human populations.Proceedings. Biological sciences · 2015Article
- Mitochondrial DNA sequence variation in multiple sclerosis.Neurology · 2015Article
- Genetic variation of oxidative phosphorylation genes in stroke and Alzheimer's disease.Neurobiology of aging · 2014Article
- Article
- Recent mitochondrial DNA mutations increase the risk of developing common late-onset human diseases.PLoS genetics · 2014Article
Corrections and comments
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Authors and funding
18 authors.
Funding
Abstract
Although inherited mitochondrial genetic variation can cause human disease, no validated methods exist for control of confounding due to mitochondrial population stratification (PS). We sought to identify a reliable method for PS assessment in mitochondrial medical genetics. We analyzed mitochondrial SNP data from 1513 European American individuals concomitantly genotyped with the use of a previously validated panel of 144 mitochondrial markers as well as the Affymetrix 6.0 (n = 432), Illumina 610-Quad (n = 458), or Illumina 660 (n = 623) platforms. Additional analyses were performed in 938 participants in the Human Genome Diversity Panel (HGDP) (Illumina 650). We compared the following methods for controlling for PS: haplogroup-stratified analyses, mitochondrial principal-component analysis (PCA), and combined autosomal-mitochondrial PCA. We computed mitochondrial genomic inflation factors (mtGIFs) and test statistics for simulated case-control and continuous phenotypes (10,000 simulations each) with varying degrees of correlation with mitochondrial ancestry. Results were then compared across adjustment methods. We also calculated power for discovery of true associations under each method, using a simulation approach. Mitochondrial PCA recapitulated haplogroup information, but haplogroup-stratified analyses were inferior to mitochondrial PCA in controlling for PS. Correlation between nuclear and mitochondrial principal components (PCs) was very limited. Adjustment for nuclear PCs had no effect on mitochondrial analysis of simulated phenotypes. Mitochondrial PCA performed with the use of data from commercially available genome-wide arrays correlated strongly with PCA performed with the use of an exhaustive mitochondrial marker panel. Finally, we demonstrate, through simulation, no loss in power for detection of true associations with the use of mitochondrial PCA.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.