Evidence mapPaperPMID 21042325Full record

Trial reportInternational journal of obesity (2005)2011

Phenotypic and genetic variation in leptin as determinants of weight regain.

G Erez, A Tirosh, A Rudich, V Meiner, D Schwarzfuchs, N Sharon, S Shpitzen, M Blüher, M Stumvoll, J Thiery and 4 more

Erratum issuedOpen access · greenAbstract readRandomized Controlled Trial
In one paragraph

Trial report in International journal of obesity (2005), 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. An erratum has been issued. Cited by 31 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
31citing papers in PubMed, 2 pooled it
2.0field-weighted citation impact, top 15% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

31 citing papers in PubMed, 2 syntheses or guidelines pooled it, 54 citations in OpenAlex.

  1. Pooled it
  2. Pooled it
  3. Trial
  4. Trial
  5. Trial
  6. Trial
  7. Article
  8. Review
  9. Review
  10. Article
  11. Leptin Signaling in Obesity and Colorectal Cancer.International journal of molecular sciences · 2022
    Review
  12. Article
  13. Article
  14. Article
  15. Article
  16. Article
  17. Review
  18. Article
  19. Article
  20. Personalized nutrition and obesity.Annals of medicine · 2014
    Review
4 · The record

Corrections and comments

  • Erratum issued
5 · Who and what money

Authors and funding

14 authors at 6 institutions in 3 countries.

G Erez *Obesity Clinic, Meuhedet Health Services, Jerusalem, Israel.
A Tirosh *Division of Endocrinology, Diabetes, and Hypertension, Brigham and Women's Hospital and Harvard Medical School, Boston, MA, USA.
A RudichThe S. Daniel Abraham Center for Health and Nutrition, Faculty of Health Sciences, Ben-Gurion University of the Negev, Beer-Sheva, Israel.
V MeinerDepartment of Human Genetics, Hadassah-University Hospital, Jerusalem, Israel.
D SchwarzfuchsNuclear Research Center Negev, Dimona, Israel.
N SharonUnit of Epidemiology, Hebrew University-Hadassah School of Public Health, Jerusalem, Israel.
S ShpitzenCenter for Research, Prevention and Treatment of Atherosclerosis, Hadassah-University Hospital, Jerusalem, Israel.
M BlüherDepartment of Medicine, University of Leipzig, Leipzig, Germany.
M StumvollDepartment of Medicine, University of Leipzig, Leipzig, Germany.
J ThieryInstitute of Laboratory Medicine, University of Leipzig, Leipzig, Germany.
G M FiedlerInstitute of Laboratory Medicine, University of Leipzig, Leipzig, Germany.
Y FriedlanderUnit of Epidemiology, Hebrew University-Hadassah School of Public Health, Jerusalem, Israel.
E LeiterstdorfCenter for Research, Prevention and Treatment of Atherosclerosis, Hadassah-University Hospital, Jerusalem, Israel.
I ShaiThe S. Daniel Abraham Center for Health and Nutrition, Faculty of Health Sciences, Ben-Gurion University of the Negev, Beer-Sheva, Israel.
Leipzig University · DEBen-Gurion University of the Negev · ILHebrew University of Jerusalem · ILClalit Health Services · ILHadassah Academic College · ILHarvard University · US

Funding

TRAINING GRANT IN ACADEMIC ENDOCRINOLOGYT32DK007529 · BRIGHAM AND WOMEN'S HOSPITAL · 1986 to 2025
$1.5M
Biotechnology and Biological Sciences Research CouncilNIDDK NIH HHS T32 DK007529
6 · The paper itself

Abstract

aimsOver 75% of obese subjects fail to maintain their weight following weight loss interventions. We aimed to identify phenotypic and genetic markers associated with weight maintenance/regain following a dietary intervention. SUBJECTS AND

methodsIn the 2-year Dietary Intervention Randomized Controlled Trial, we assessed potential predictors for weight changes during the 'weight loss phase' (0-6 months) and the 'weight maintenance/regain phase' (7-24 months). Genetic variation between study participants was studied using single-nucleotide polymorphisms in the leptin gene (LEP).

resultsMean weight reduction was -5.5% after 6 months, with a mean weight regain of 1.2% of baseline weight during the subsequent 7-24 months. In a multivariate regression model, higher baseline high-molecular-weight adiponectin was the only biomarker predictor of greater success in 0- to 6-month weight loss (β = -0.222, P-value = 0.044). In a multivariate regression model adjusted for 6-month changes in weight and various biomarkers, 6-month plasma leptin reduction exhibited the strongest positive association with 6-month weight loss (β = 0.505, P-value < 0.001). Conversely, 6-month plasma leptin reduction independently predicted weight regain during the following 18 months (β = -0.131, P-value < 0.013). Weight regain was higher among participants who had a greater (top tertiles) 6-month decrease in both weight and leptin (+3.4% (95% confidence interval 2.1-4.8)) as compared with those in the lowest combined tertiles (+0.2% (95% confidence interval -1.1 to 1.4)); P-value < 0.001. Weight regain was further significantly and independently associated with genetic variations in LEP (P = 0.006 for both rs4731426 and rs2071045). Adding genetic data to the phenotypic multivariate model increased its predictive value for weight regain by 34%.

conclusionAlthough greater reduction in leptin concentrations during the initial phase of a dietary intervention is associated with greater weight loss in the short term, plasma leptin reduction, combined with the degree of initial weight loss and with genetic variations in the LEP gene, constitutes a significant predictor of subsequent long-term weight regain.

Indexed as

BiomarkersBody Mass IndexDiet, ReducingFemaleGenetic VariationHumansLeptinMaleMiddle AgedObesityPhenotypeWeight GainBiomarkersLeptin

Identifiers

PMID21042325
PMCPMC4941231
OpenAlexW2039638074

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.