Evidence mapPaperPMID 21079387Full record

ArticleNeuroendocrinology2011

Perinatal exposure to high-fat diet programs energy balance, metabolism and behavior in adulthood.

Elinor L Sullivan, M Susan Smith, Kevin L Grove

Registry-linked trialOpen access · bronzeAbstract read
In one paragraph

Article in Neuroendocrinology, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT02211651 (Maternal Adipose Tissue and Placental Dysfunction Programs the Fetus for Type 2 Diabetes), which is not on this map. Cited by 92 papers.

0numbers the graph read from it
0cells of the map it votes in
92citing papers in PubMed
22.0field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT02211651 completednot on this mapstarted 2013, after this paper: background citation

Maternal Adipose Tissue and Placental Dysfunction Programs the Fetus for Type 2 Diabetes (PlacentA-DM)

TypeobservationalSponsorAdventHealth Translational Research InstituteRan2013 to 2017Enrolled3ConditionsObesity, Type 2 Diabetes
3 · Its place in the literature

Who cites it

92 citing papers in PubMed, 175 citations in OpenAlex.

  1. Trial
  2. Article
  3. Article
  4. Review
  5. Review
  6. Article
  7. Exploring the Optimal Diagnostic Thresholds of Hyperglycemia During Pregnancy.Maternal-fetal medicine (Wolters Kluwer Health, Inc.) · 2023
    Article
  8. Review
  9. Article
  10. Review
  11. Article
  12. Review
  13. Review
  14. Article
  15. Article
  16. Review
  17. Article
  18. Effects of pre- and postnatal protein restriction on maternal and offspring metabolism in the nonhuman primate.American journal of physiology. Regulatory, integrative and comparative physiology · 2020
    Article
  19. Article
  20. Article

32 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Elinor L SullivanDepartment of Neuroscience, Oregon National Primate Research Center, Beaverton, OR 97006, USA. sullivel@ohsu.edu
M Susan Smith
Kevin L Grove
Oregon National Primate Research Center · US

Funding

XENOGRAFTS OF RHESUS ENDOMETRIUM IN SEVERE COMBINED IMMUNODEFICIENT (SCID) MICEP51RR000163 · OREGON REGIONAL PRIMATE RESEARCH CENTER · 1985 to 2005
$61.0M
CONTROL OF GONADOTROPIN SECRETION DURING LACTATIONR01HD014643 · UNIVERSITY OF PITTSBURGH AT PITTSBURGH · 1985 to 2004
$992k
NCRR NIH HHS P51 RR000163NICHD NIH HHS R01 HD014643NIDDK NIH HHS R01 DK079194
6 · The paper itself

Abstract

The perinatal environment plays an important role in programming many aspects of physiology and behavior including metabolism, body weight set point, energy balance regulation and predisposition to mental health-related disorders such as anxiety, depression and attention deficit hyperactivity disorder. Maternal health and nutritional status heavily influence the early environment and have a long-term impact on critical central pathways, including the melanocortinergic, serotonergic system and dopaminergic systems. Evidence from a variety of animal models including rodents and nonhuman primates indicates that exposure to maternal high-fat diet (HFD) consumption programs offspring for increased risk of adult obesity. Hyperphagia and increased preference for fatty and sugary foods are implicated as mechanisms for the increased obesity risk. The effects of maternal HFD consumption on energy expenditure are unclear, and future studies need to address the impact of perinatal HFD exposure on this important component of energy balance regulation. Recent evidence from animal models also indicates that maternal HFD consumption increases the risk of offspring developing mental health-related disorders such as anxiety. Potential mechanisms for perinatal HFD programming of neural pathways include circulating factors, such as hormones (leptin, insulin), nutrients (fatty acids, triglycerides and glucose) and inflammatory cytokines. As maternal HFD consumption and obesity are common and rapidly increasing, we speculate that future generations will be at increased risk for both metabolic and mental health disorders. Thus, it is critical that future studies identify therapeutic strategies that are effective at preventing maternal HFD-induced malprogramming.

Indexed as

AdultAnimalsDietary FatsEatingEnergy MetabolismFeeding BehaviorFemaleHumansMental DisordersMetabolismModels, AnimalModels, BiologicalObesityPregnancyPrenatal Exposure Delayed EffectsDietary Fats

Identifiers

PMID21079387
PMCPMC3700139
OpenAlexW2042364545

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.