Evidence mapPaperPMID 21411512Full record

Trial reportDiabetes2011

Recombinant human leptin treatment does not improve insulin action in obese subjects with type 2 diabetes.

Bettina Mittendorfer, Jeffrey F Horowitz, Alex M DePaoli, Mark A McCamish, Bruce W Patterson, Samuel Klein

Open access · bronzeAbstract readRandomized Controlled Trial
In one paragraph

Trial report in Diabetes, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 61 papers.

0numbers the graph read from it
0cells of the map it votes in
61citing papers in PubMed
3.5field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

61 citing papers in PubMed, 113 citations in OpenAlex.

  1. Trial
  2. Trial
  3. Review
  4. Review
  5. Review
  6. Research Progress on Leptin in Metabolic Dysfunction-associated Fatty Liver Disease.Journal of clinical and translational hepatology · 2025
    Review
  7. Review
  8. Review
  9. Review
  10. Article
  11. Review
  12. The role of leptin and low testosterone in obesity.International journal of impotence research · 2022
    Review
  13. Review
  14. Article
  15. Article
  16. Article
  17. Review
  18. Leptin in Leanness and Obesity: JACC State-of-the-Art Review.Journal of the American College of Cardiology · 2021
    Review
  19. Review
  20. Leptin and Obesity: Role and Clinical Implication.Frontiers in endocrinology · 2021
    Review

1 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 2 institutions in 1 country.

Bettina MittendorferCenter for Human Nutrition and Atkins Center of Excellence in Obesity Medicine, Washington University School of Medicine, St. Louis, Missouri, USA.
Jeffrey F Horowitz
Alex M DePaoli
Mark A McCamish
Bruce W Patterson
Samuel Klein
Atkins (United States) · USAmgen (United States) · US

Funding

WHOLE BODY PROTEIN KINETICS IN HIV INFECTED MALAWIAN CHILDRENP41RR000954 · WASHINGTON UNIVERSITY · 1985 to 2005
$6.6M
Washington University Nutrition Obesity Research CenterP30DK056341 · WASHINGTON UNIVERSITY · 1999 to 2025
$5.7M
THERMOGENESIS AND EXERCISE IN LEAN, OBESE, AND DIABETICR01DK037948 · WEILL MEDICAL COLLEGE OF CORNELL UNIV · 1987 to 2005
$1.7M
NCRR NIH HHS P41 RR000954NCRR NIH HHS RR-00954NCRR NIH HHS UL1 RR024992NCRR NIH HHS UL1-RR-024992NICHD NIH HHS HD-57796NICHD NIH HHS P50 HD057796NIDDK NIH HHS DK-37948NIDDK NIH HHS DK-56341NIDDK NIH HHS P30 DK056341NIDDK NIH HHS R01 DK037948
6 · The paper itself

Abstract

objectiveLeptin therapy improves insulin sensitivity in people with leptin deficiency, but it is not known whether it improves insulin action in people who are not leptin deficient. The purpose of the current study was to determine whether leptin treatment has weight loss-independent effects on insulin action in obese subjects with type 2 diabetes. RESEARCH DESIGN AND

methodsWe conducted a randomized, placebo-controlled trial in obese subjects (BMI: 35.4 ± 0.6 kg/m(2); mean ± SE) with newly diagnosed type 2 diabetes. Subjects were randomized to treatment with placebo (saline), low-dose (30 mg/day), or high-dose (80 mg/day) recombinant methionyl human (r-Met hu) leptin for 14 days. Multiorgan insulin sensitivity before and after treatment was evaluated by using the hyperinsulinemic-euglycemic clamp procedure in conjunction with stable isotopically labeled tracer infusions to measure glucose, glycerol, and fatty acid kinetics.

resultsLow-dose and high-dose leptin treatment resulted in a threefold (P < 0.01) and 150-fold (P < 0.001) increase in basal plasma leptin concentrations, respectively. However, neither low-dose nor high-dose therapy had an effect on insulin-mediated suppression of glucose, glycerol, or palmitate rates of appearance into plasma compared with placebo. In addition, leptin treatment did not increase insulin-mediated stimulation of glucose disposal compared with placebo (14.3 ± 3.1, 18.4 ± 3.6, 16.7 ± 2.4 vs. 17.5 ± 2.5, 20.7 ± 3.0, 19.1 ± 3.3 μmol/kg body wt/min before vs. after treatment in the placebo, low-dose, and high-dose leptin groups, respectively).

conclusionsr-Met hu leptin does not have weight loss-independent, clinically important effects on insulin sensitivity in obese people with type 2 diabetes.

Indexed as

Blood GlucoseBody CompositionDiabetes Mellitus, Type 2FemaleGlucose Clamp TechniqueGlycerolHumansInsulinLeptinMaleMiddle AgedObesityPalmitic AcidPlacebosBlood GlucoseGlycerolInsulinLeptinPalmitic AcidPlacebos

Identifiers

PMID21411512
PMCPMC3292320
OpenAlexW2133619548

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.