Evidence map›Paper›PMID 21704502›Full record

ArticleNutrition, metabolism, and cardiovascular diseases : NMCD2012

Metabolic adaptations to early life protein restriction differ by offspring sex and post-weaning diet in the mouse.

K W Whitaker, K Totoki, T M Reyes

Abstract read
In one paragraph

Article in Nutrition, metabolism, and cardiovascular diseases : NMCD, 2012. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 24 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
24citing papers in PubMed, 1 pooled it
3.9field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

24 citing papers in PubMed, 1 synthesis or guideline pooled it, 39 citations in OpenAlex.

  1. Pooled it
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  4. Review
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  16. Genetic approaches in comparative and evolutionary physiology.American journal of physiology. Regulatory, integrative and comparative physiology · 2015
    Review
  17. Dissociable deficits of executive function caused by gestational adversity are linked to specific transcriptional changes in the prefrontal cortex.Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology · 2015
    Article
  18. Article
  19. Obesity at conception programs the opioid system in the offspring brain.Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology · 2014
    Article
  20. Sex differences in the physiology of eating.American journal of physiology. Regulatory, integrative and comparative physiology · 2013
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

K W WhitakerDepartment of Pharmacology, Institute for Translational Medicine and Therapeutics, University of Pennsylvania School of Medicine, Philadelphia, PA 19104-5158, USA.
K Totoki
T M Reyes
University of Pennsylvania · US

Funding

In utero programming of the dopamine system: behavior, neuroanatomy & epigeneticsR01MH087978 · NIMH · UNIVERSITY OF PENNSYLVANIA · PI REYES, TERESA M · 2010 to 2014
$2.1M
Functional Neuroanatomy of Cytokine-Mediated AnorexiaK01DK064086 · NIDDK · UNIVERSITY OF PENNSYLVANIA · PI REYES, TERESA M · 2003 to 2007
$523k
NIDDK NIH HHS DK064086NIDDK NIH HHS K01 DK064086NIMH NIH HHS MH087978NIMH NIH HHS R01 MH087978
6 · The paper itself

Abstract

BACKGROUND AND

aimsLow birth weight affects 1 in every 7 babies born globally and can predict a lifetime of increased risk for adverse health outcomes, including cardiovascular disease, hypertension, obesity, diabetes, and metabolic syndrome. Maternal low protein diet during pregnancy and lactation is a well-characterized rat model for low birth weight and the subsequent increase in chronic disease risk. However, mice have been relatively understudied in this paradigm and represent a critical resource for investigating the underlying molecular mechanisms that link adverse early life experience and the development of chronic disease. METHODS AND

resultsThe present manuscript describes a mouse model of low birth weight (maternal consumption of low protein diet (8% protein) through pregnancy and lactation) and characterizes metabolic adaptations (food intake, locomotor activity, oxygen consumption, and glucose tolerance) in male and female offspring. At weaning, mice were maintained either on the control diet or a high fat diet. Notable sex differences were observed, with male mice from the low protein pregnancies showing increased food intake, hyperactivity and increased metabolic rate only when weaned to the high fat diet, while female mice consistently showed increased food intake and were hypometabolic, regardless of post-weaning diet.

conclusionThese data identify offspring sex and post-weaning diet as critical variables in the metabolic adaptations to early life protein deficiency, and suggest that females may be more vulnerable to the adverse long-term health consequences of low birth weight.

Indexed as

Adaptation, PhysiologicalBody WeightDiet, Protein-RestrictedWeaningAnimal FeedAnimalsDietary ProteinsDiet, High-FatEnergy IntakeFemaleGlucose Tolerance TestLactationMaleMiceMice, Inbred C57BLMice, Inbred DBADietary Proteins

Identifiers

PMID21704502
PMCPMC3183163
OpenAlexW2093428785

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.