Evidence mapPaperPMID 21712835Full record

Trial reportEuropean journal of clinical nutrition2011

Inulin increases short-term markers for colonic fermentation similarly in healthy and hyperinsulinaemic humans.

J Fernandes, J Vogt, T M S Wolever

Abstract readRandomized Controlled Trial
In one paragraph

Trial report in European journal of clinical nutrition, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 18 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
18citing papers in PubMed, 1 pooled it
2.7field-weighted citation impact, top 11% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

18 citing papers in PubMed, 1 synthesis or guideline pooled it, 49 citations in OpenAlex.

  1. Pooled it
  2. Trial
  3. Trial
  4. Trial
  5. Article
  6. Review
  7. Article
  8. Article
  9. Central and peripheral regulations mediated by short-chain fatty acids on energy homeostasis.Translational research : the journal of laboratory and clinical medicine · 2022
    Review
  10. Nutritional strategies to alleviate oxidative stress in sows.Animal nutrition (Zhongguo xu mu shou yi xue hui) · 2022
    Review
  11. Article
  12. Article
  13. Article
  14. The metabolic fate of acetate in cancer.Nature reviews. Cancer · 2016
    Review
  15. Article
  16. Article
  17. Article
  18. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 1 country.

J FernandesDepartment of Nutritional Sciences, Faculty of Medicine, University of Toronto, Toronto, Ontario, Canada.
J Vogt
T M S Wolever
University of Toronto · CASt. Michael's Hospital · CA

Funding

CIHR 64648-1
6 · The paper itself

Abstract

BACKGROUND/

objectivesColonic fermentation of dietary fibre produces short-chain fatty acids (SCFAs), acetate, propionate and butyrate, which may protect against type 2 diabetes by reducing serum free-fatty acids (FFAs). Since hyperinsulinaemia is associated with insulin resistance and increased diabetes risk, the main objective was to compare markers of colonic fermentation after acute inulin ingestion in subjects with normal (<40  pmol/l, NI) and high (≥40  pmol/l, HI) plasma insulin. SUBJECTS/

methodsOvernight fasted NI (n=9) and HI (n=9) subjects were studied for 4  h on two separate days after consuming 300  ml drinks containing 75  g glucose (Glucose) or 75  g glucose plus 24  g inulin (Inulin) using a randomized, single-blind, crossover design.

resultsInulin elicited a higher breath hydrogen and methane areas under the curve (AUC), but the increases in SCFA responses were not statistically significant. Mean serum-acetate concentration over the 4-h study period was higher in NI than in HI subjects (44.3 ± 6.9 vs 22.5 ± 3.7  μmol/l, P=0.001). The rate of rebound of FFA was reduced by Inulin, with FFA at 4  h being less after Inulin than Glucose, regardless of insulin status (0.310 ± 0.028 vs 0.432 ± 0.042 mEq/l, P=0.008).

conclusionsThis suggests that inulin increases short-term markers for colonic fermentation, but a longer study period may be necessary to observe differences in SCFA production. The reason for the lower serum acetate in HI is unclear but may be due to reduced absorption, increased clearance or decreased endogenous production. This suggests the need to compare acetate kinetics in normal and hyperinsulinaemic subjects.

Indexed as

AcetatesAdultArea Under CurveBiomarkersBreath TestsColonCross-Over StudiesDietary CarbohydratesDietary FiberFatty Acids, VolatileFemaleFermentationGlucoseHumansHydrogenHyperinsulinismAcetatesBiomarkersDietary CarbohydratesDietary FiberFatty Acids, VolatileGlucoseHydrogenInulinMethane

Identifiers

PMID21712835
PMCPMC3937120
OpenAlexW1993326198

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.