Evidence mapPaperPMID 21955459Full record

ReviewDiabetes, obesity & metabolism2012

The role of the kidneys in glucose homeostasis: a new path towards normalizing glycaemia.

R A DeFronzo, J A Davidson, S Del Prato

3 registry-linked trialsAbstract readReview
PubMed Publisher
In one paragraph

Review in Diabetes, obesity & metabolism, 2012. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to 3 registered trials, which are not on this map. Cited by 189 papers, 10 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
189citing papers in PubMed, 10 pooled it
17.4field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT02887677 phase4terminatedstarted 2016, after this paper: background citation

A Study of the Effects of Dapagliflozin on Ambulatory Aortic Pressure, Arterial Stiffness and Urine Albumin Excretion in Patients With Type 2 Diabetes

Ran2016Enrolled85Registered outcomes9Posted comparisons0ConditionsDiabetes MellitusArmsdapagliflozin, Placebo
Open the trial in the graph
NCT06000462 phase4active not recruitingstarted 2024, after this paper: background citation

The Effect of Sodium Glucose Co-transporter 2 Inhibitor (dapagliflozin) on Weight Loss in Non-diabetic Adults with Obesity: Triple-blinded, Randomized, Placebo-controlled Trial.

Ran2024Enrolled150Registered outcomes4Posted comparisons0ConditionsDapagliflozin Adverse Reaction, Effect of Drug, Obesity, Weight LossArmsDapagliflozin 10mg Tab, Metformin 1000 mg (ttd), Placebo
Open the trial in the graph
NCT06421870 phase3unknown statusnot on this mapstarted 2024, after this paper: background citation

Renoprotective Effects of Dapagliflozin Versus Pentoxiphylline in Chronic Kidney Disease Patients

TypeinterventionalSponsorAin Shams UniversityRan2024 to 2025Enrolled210ConditionsChronic Kidney DiseasesArmsDapagliflozin 10mg Tab, Pentoxifylline 400 MG
3 · Its place in the literature

Who cites it

189 citing papers in PubMed, 10 syntheses or guidelines pooled it, 486 citations in OpenAlex.

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129 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 3 institutions in 2 countries.

R A DeFronzoDiabetes Division, Department of Medicine, University of Texas Health Science Center, San Antonio, TX 78229, USA. albarado@uthscsa.edu
J A Davidson
S Del Prato
Texas Diabetes Institute · USThe University of Texas Southwestern Medical Center · USUniversity of Pisa · IT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The maintenance of normal glucose homeostasis requires a complex, highly integrated interaction among the liver, muscle, adipocytes, pancreas and neuroendocrine system. Recent studies have showed that the kidneys also play a central role in glucose homeostasis by reabsorbing all the filtered glucose, an adaptive mechanism that ensures sufficient energy is available during fasting periods. This mechanism becomes maladaptive in diabetes, however, as hyperglycaemia augments the expression and activity of the sodium-glucose cotransporter (SGLT) 2 in the proximal tubule of the kidney. As a result, glucose reabsorption may be increased by as much as 20% in individuals with poorly controlled diabetes. SGLT2 is a low-affinity, high-capacity glucose transport protein that reabsorbs 90% of filtered glucose, while the high-affinity, low-capacity SGLT1 transporter reabsorbs the remaining 10%. SGLT2 represents a novel target for the treatment of diabetes. In animal studies, SGLT2 inhibition reduces plasma glucose levels, resulting in improved β-cell function and enhanced insulin sensitivity in liver and muscle. Human studies have confirmed the efficacy of SLGT2 inhibitors in improving glucose control and reducing the A1c. Because the mechanism of SGLT2 inhibition is independent of circulating insulin levels or insulin sensitivity, these agents can be combined with all other antidiabetic classes, including exogenous insulin. Although the long-term efficacy and safety of SGLT2 inhibitors remain under study, the class represents a novel therapeutic approach with potential for the treatment of both type 2 and 1 diabetes.

Indexed as

Sodium-Glucose Transporter 2 InhibitorsAnimalsBlood GlucoseDiabetes Mellitus, Type 1Diabetes Mellitus, Type 2Glucose Transport Proteins, FacilitativeHomeostasisHumansHypoglycemic AgentsKidneyRatsSodium-Glucose Transporter 2Blood GlucoseGlucose Transport Proteins, FacilitativeHypoglycemic AgentsSodium-Glucose Transporter 2Sodium-Glucose Transporter 2 Inhibitors

Identifiers

PMID21955459
OpenAlexW2118613762

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.