Evidence map›Paper›PMID 22077553›Full record

ReviewAnnual review of pathology2012

Pathogenesis of plexiform neurofibroma: tumor-stromal/hematopoietic interactions in tumor progression.

Karl Staser, Feng-Chun Yang, D Wade Clapp

Open access · greenAbstract readReview
In one paragraph

Review in Annual review of pathology, 2012. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 56 papers.

0numbers the graph read from it
0cells of the map it votes in
56citing papers in PubMed
3.2field-weighted citation impact, top 8% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

56 citing papers in PubMed, 108 citations in OpenAlex.

  1. Trial
  2. Management of plexiform neurofibromas in neurofibromatosis type 1: An Italian Delphi consensus.Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology · 2026
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 2 countries.

Karl StaserDepartment of Biochemistry, Indiana University School of Medicine, Indianapolis, Indiana 46202, USA.
Feng-Chun Yang
D Wade Clapp
Indiana University – Purdue University Indianapolis · USIndiana University School of Medicine

Funding

Transgenic CoreP50NS052606 · NINDS · UT SOUTHWESTERN MEDICAL CENTER · PI PARADA, LUIS FERNANDO · 2005 to 2014
$12.6M
Neurofibromatosis Type 1 Gene Regulates MyelopoiesisR01CA074177 · NCI · INDIANA UNIV-PURDUE UNIV AT INDIANAPOLIS · PI CLAPP, DAVID W · 2002 to 2012
$3.6M
Cancer Biology Training ProgramT32CA111198 · NCI · INDIANA UNIV-PURDUE UNIV AT INDIANAPOLIS · PI NAKSHATRI, HARIKRISHNA · 2005 to 2009
$1.8M
NCI NIH HHS R01 CA074177NCI NIH HHS R01 CA074177-11A1/DNCI NIH HHS T32 CA111198NINDS NIH HHS P50 NS052606NINDS NIH HHS P50 NS052606-04
6 · The paper itself

Abstract

Neurofibromatosis type 1 (NF1) is a genetic disease that results from either heritable or spontaneous autosomal dominant mutations in the NF1 gene. A second-hit mutation precedes the predominant NF1 neoplasms, which include myeloid leukemia, optic glioma, and plexiform neurofibroma. Despite this requisite NF1 loss of heterozygosity in the tumor cell of origin, nontumorigenic cells contribute to both generalized and specific disease manifestations. In mouse models of plexiform neurofibroma formation, Nf1 haploinsufficient mast cells promote inflammation, accelerating tumor formation and growth. These recruited mast cells, hematopoietic effector cells long known to permeate neurofibroma tissue, mediate key mitogenic signals that contribute to vascular ingrowth, collagen deposition, and tumor growth. Thus, the plexiform neurofibroma microenvironment involves a tumor/stromal interaction with the hematopoietic system that depends, at the molecular level, on a stem cell factor/c-kit-mediated signaling axis. These observations parallel findings in other NF1 disease manifestations and are clearly relevant to medical management of these neurofibromas.

Indexed as

AnimalsAntineoplastic AgentsBenzamidesHematopoietic SystemHumansImatinib MesylateMast CellsNeurofibroma, PlexiformNeurofibromatosis 1Neurofibromin 1PiperazinesProto-Oncogene Proteins c-kitPyrimidinesSignal TransductionStem Cell FactorTumor MicroenvironmentAntineoplastic AgentsBenzamidesImatinib MesylateNeurofibromin 1PiperazinesProto-Oncogene Proteins c-kitPyrimidinesStem Cell Factor

Identifiers

PMID22077553
PMCPMC3694738
OpenAlexW2139663053

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.