ArticleGenetic epidemiology2011
Lessons learned from Genetic Analysis Workshop 17: transitioning from genome-wide association studies to whole-genome statistical genetic analysis.
Article in Genetic epidemiology, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.
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Who cites it
15 citing papers in PubMed, 17 citations in OpenAlex.
- NIA Long Life Family Study: Objectives, Design, and Heritability of Cross-Sectional and Longitudinal Phenotypes.The journals of gerontology. Series A, Biological sciences and medical sciences · 2022Article
- Rediscovering the value of families for psychiatric genetics research.Molecular psychiatry · 2019Review
- ComPaSS-GWAS: A method to reduce type I error in genome-wide association studies when replication data are not available.Genetic epidemiology · 2019Article
- Robust Rare-Variant Association Tests For Quantitative Traits in General Pedigrees.Statistics in biosciences · 2018Article
- Cognitive, Linguistic, and Motor Abilities in a Multigenerational Family with Childhood Apraxia of Speech.Archives of clinical neuropsychology : the official journal of the National Academy of Neuropsychologists · 2016Article
- Family-based approaches: design, imputation, analysis, and beyond.BMC genetics · 2016Article
- The value of extended pedigrees for next-generation analysis of complex disease in the rhesus macaque.ILAR journal · 2013Review
- Assessing the impact of differential genotyping errors on rare variant tests of association.PloS one · 2013Article
- The role of large pedigrees in an era of high-throughput sequencing.Human genetics · 2012Review
- Study designs and methods post genome-wide association studies.Human genetics · 2012Article
- Identification of rare variants from exome sequence in a large pedigree with autism.Human heredity · 2012Article
- Identifying rare variants from exome scans: the GAW17 experience.BMC proceedings · 2011Article
- Regression and data mining methods for analyses of multiple rare variants in the Genetic Analysis Workshop 17 mini-exome data.Genetic epidemiology · 2011Article
- Brief review of regression-based and machine learning methods in genetic epidemiology: the Genetic Analysis Workshop 17 experience.Genetic epidemiology · 2011Review
- Linkage analysis in the next-generation sequencing era.Human heredity · 2011Review
Corrections and comments
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Authors and funding
2 authors at 2 institutions in 2 countries.
Funding
Abstract
Genetic Analysis Workshop 17 (GAW17) focused on the transition from genome-wide association study designs and methods to the study designs and statistical genetic methods that will be required for the analysis of next-generation sequence data including both common and rare sequence variants. In the 166 contributions to GAW17, a wide variety of statistical methods were applied to simulated traits in population- and family-based samples, and results from these analyses were compared to the known generating model. In general, many of the statistical genetic methods used in the population-based sample identified causal sequence variants (SVs) when the estimated locus-specific heritability, as measured in the population-based sample, was greater than about 0.08. However, SVs with locus-specific heritabilities less than 0.03 were rarely identified consistently. In the family-based samples, many of the methods detected SVs that were rarer than those detected in the population-based sample, but the estimated locus-specific heritabilities for these rare SVs, as measured in the family-based samples, were substantially higher (>0.2) than their corresponding heritabilities in the population-based samples. Substantial inflation of the type I error rate was observed across a wide variety of statistical methods. Although many of the contributions found little inflation in type I error for Q4, a trait with no causal SVs, type I error rates for Q1 and Q2 were well above their nominal levels with the inflation for Q1 being higher than that for Q2. It seems likely that this inflation in type I error is due to correlations among SVs.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.