Evidence mapPaperPMID 22193383Full record

ArticleJournal of the American Society of Nephrology : JASN2012

Hyperglycemia and renin-dependent hypertension synergize to model diabetic nephropathy.

Bryan R Conway, Jillian Rennie, Matthew A Bailey, Donald R Dunbar, Jonathan R Manning, Christopher O Bellamy, Jeremy Hughes, John J Mullins

Open access · bronzeAbstract read
In one paragraph

Article in Journal of the American Society of Nephrology : JASN, 2012. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 33 papers.

0numbers the graph read from it
0cells of the map it votes in
33citing papers in PubMed
1.6field-weighted citation impact, top 15% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

33 citing papers in PubMed, 46 citations in OpenAlex.

  1. Article
  2. Review
  3. Review
  4. Review
  5. Article
  6. Article
  7. Article
  8. NOX4-dependent regulation of ENaC in hypertension and diabetic kidney disease.FASEB journal : official publication of the Federation of American Societies for Experimental Biology · 2020
    Article
  9. Revisiting Experimental Models of Diabetic Nephropathy.International journal of molecular sciences · 2020
    Review
  10. Frontiers in pharmacology · 2020
    Article
  11. Article
  12. Renal lesions in leptin receptor-deficient medaka (Journal of toxicologic pathology · 2019
    Article
  13. Article
  14. Review
  15. Article
  16. Article
  17. Review
  18. Article
  19. Article
  20. Modelling diabetic nephropathy in mice.Nature reviews. Nephrology · 2018
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 4 institutions in 1 country.

Bryan R ConwayMRC Centre for Inflammation Research, University of Edinburgh, Edinburgh, Scotland, UK. bryan.conway@ed.ac.uk
Jillian Rennie
Matthew A Bailey
Donald R Dunbar
Jonathan R Manning
Christopher O Bellamy
Jeremy Hughes
John J Mullins
University of Edinburgh · GBBritish Heart Foundation Centre for Cardiovascular Science · GBCentre for Inflammation Research · GBQueen's Medical Centre · GB

Funding

Medical Research Council G0501900Medical Research Council G0901697
6 · The paper itself

Abstract

Rodent models exhibit only the earliest features of human diabetic nephropathy, which limits our ability to investigate new therapies. Hypertension is a prerequisite for advanced diabetic nephropathy in humans, so its rarity in typical rodent models may partly explain their resistance to nephropathy. Here, we used the Cyp1a1mRen2 rat, in which the murine renin-2 gene is incorporated under the Cytochrome P4501a1 promoter. In this transgenic strain, administration of low-dose dietary indole-3-carbinol induces moderate hypertension. In the absence of hypertension, streptozotocin-induced diabetes resulted in a 14-fold increase in albuminuria but only mild changes in histology and gene expression despite 28 weeks of marked hyperglycemia. In the presence of induced hypertension, hyperglycemia resulted in a 500-fold increase in albuminuria, marked glomerulosclerosis and tubulointerstitial fibrosis, and induction of many of the same pathways that are upregulated in the tubulointerstitium in human diabetic nephropathy. In conclusion, although induction of diabetes alone in rodents has limited utility to model human diabetic nephropathy, renin-dependent hypertension and hyperglycemia synergize to recapitulate many of the clinical, histological, and gene expression changes observed in humans.

Indexed as

Disease Models, AnimalReninAlbuminuriaAnimalsComorbidityCytochrome P-450 CYP1A1Diabetes Mellitus, ExperimentalDiabetic NephropathiesFibrosisHumansHyperglycemiaHypertensionIndolesKidney TubulesMiceRatsCytochrome P-450 CYP1A1indole-3-carbinolIndolesRen2 protein, mouseReninStreptozocin

Identifiers

PMID22193383
PMCPMC3294297
OpenAlexW2155334659

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.