Evidence mapPaperPMID 22885332Full record

ReviewGastroenterology2012

Effects of bariatric surgery on glucose homeostasis and type 2 diabetes.

David Bradley, Faidon Magkos, Samuel Klein

3 registry-linked trialsAbstract readReview
In one paragraph

Review in Gastroenterology, 2012. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to 3 registered trials, which are not on this map. Cited by 74 papers.

0numbers the graph read from it
0cells of the map it votes in
74citing papers in PubMed
15.0field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT03165812 naunknown statusnot on this mapstarted 2017, after this paper: background citation

Single Anastomosis Duodeno Jejunal Bypass With Sleeve Gastrectomy (SADJB-SG) Versus Intensive Medical Therapy (IMT) in the Treatment of Type 2 Diabetes Mellitus Among Asian Patients With BMI 23.5 - 30 kg/m2: A Clinical Trial

TypeinterventionalSponsorUniversiti Putra MalaysiaRan2017 to 2019Enrolled84ConditionsDiabetes Mellitus, Type 2, OverweightArmsSADJB-SG group, IMT group
NCT03239782 nacompletednot on this mapstarted 2016, after this paper: background citation

The "Metabolically-obese Normal-weight" Phenotype in Two Asian Ethnic Groups and Its Reversal by Calorie Restriction

TypeinterventionalSponsorSingapore Institute of Food and Biotechnology InnovationRan2016 to 2017Enrolled77ConditionsGlucose Metabolism Disorders, Obesity, Visceral, Obesity, EndocrineArmsCalorie restriction
NCT03264001 naunknown statusnot on this mapstarted 2017, after this paper: background citation

Effects of Progressive Negative Energy Balance Induced by Diet or Exercise on Glucose Tolerance, Insulin Sensitivity, and Beta-cell Function

TypeinterventionalSponsorSingapore Institute of Food and Biotechnology InnovationRan2017 to 2018Enrolled61ConditionsInsulin Sensitivity, Glucose Intolerance, Insulin Resistance, Energy SupplyArmsNegative energy balance
3 · Its place in the literature

Who cites it

74 citing papers in PubMed, 150 citations in OpenAlex.

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14 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

David BradleyCenter for Human Nutrition, Washington University School of Medicine, St Louis, Missouri 63110, USA.
Faidon Magkos
Samuel Klein
Washington University in St. Louis · US

Funding

Washington University Nutrition Obesity Research CenterP30DK056341 · WASHINGTON UNIVERSITY · 1999 to 2025
$5.7M
THERMOGENESIS AND EXERCISE IN LEAN, OBESE, AND DIABETICR01DK037948 · WEILL MEDICAL COLLEGE OF CORNELL UNIV · 1987 to 2005
$1.7M
NUTRITION-BEHAVIORAL CARDIOVASCULAR DISEASE PREVENTIONT32HL007456 · WASHINGTON UNIVERSITY · 1985 to 2005
$797k
NCATS NIH HHS UL1 TR000448NCRR NIH HHS UL1 RR024992NHLBI NIH HHS T32 HL007456NIDDK NIH HHS DK37948NIDDK NIH HHS DK56341NIDDK NIH HHS P30 DK056341NIDDK NIH HHS R01 DK037948
6 · The paper itself

Abstract

Obesity is an important risk factor for type 2 diabetes mellitus (T2DM). Weight loss improves the major factors involved in the pathogenesis of T2DM, namely insulin action and beta cell function, and is considered a primary therapy for obese patients who have T2DM. Unfortunately, most patients with T2DM fail to achieve successful weight loss and adequate glycemic control from medical therapy. In contrast, bariatric surgery causes marked weight loss and complete remission of T2DM in most patients. Moreover, bariatric surgical procedures that divert nutrients away from the upper gastrointestinal tract are more successful in producing weight loss and remission of T2DM than those that simply restrict stomach capacity. Although upper gastrointestinal tract bypass procedures alter the metabolic response to meal ingestion, by increasing early postprandial plasma concentrations of glucagon-like peptide 1 and insulin, it is not clear whether these effects make an important contribution to long-term control of glycemia and T2DM once substantial surgery-induced weight loss has occurred. Nonetheless, the effects of surgery on body weight and metabolic function indicate that bariatric surgery should be part of the standard therapy for T2DM. More research is needed to advance our understanding of the physiological effects of different bariatric surgical procedures and possible weight loss-independent factors that improve metabolic function and contribute to the resolution of T2DM.

Indexed as

Bariatric SurgeryHomeostasisInsulin ResistanceAnimalsBlood GlucoseDiabetes Mellitus, Type 2HumansInsulinInsulin-Secreting CellsInsulin SecretionObesityBlood GlucoseInsulin

Identifiers

PMID22885332
PMCPMC3462491
OpenAlexW1976524366

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.