Evidence mapPaperPMID 23471985Full record

Trial reportProceedings of the National Academy of Sciences of the United States of America2013

Genome-wide scan of healthy human connectome discovers SPON1 gene variant influencing dementia severity.

Neda Jahanshad, Priya Rajagopalan, Xue Hua, Derrek P Hibar, Talia M Nir, Arthur W Toga, Clifford R Jack, Andrew J Saykin, Robert C Green, Michael W Weiner and 9 more

Open access · bronzeAbstract readClinical TrialMulticenter StudyTwin Study
In one paragraph

Trial report in Proceedings of the National Academy of Sciences of the United States of America, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 88 papers, 3 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
88citing papers in PubMed, 3 pooled it
17.2field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

88 citing papers in PubMed, 3 syntheses or guidelines pooled it, 164 citations in OpenAlex.

  1. Pooled it
  2. Pooled it
  3. Pooled it
  4. Article
  5. Article
  6. Connecting intermediate phenotypes to disease using multi-omics in heart failure.Pacific Symposium on Biocomputing. Pacific Symposium on Biocomputing · 2025
    Article
  7. Article
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  9. Biological Age, Chronological Age, and Survival in Pulmonary Fibrosis: A Causal Mediation Analysis.American journal of respiratory and critical care medicine · 2024
    Article
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  13. Review
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  15. Peripheral Expression ofPsychiatry and clinical psychopharmacology · 2021
    Article
  16. Review
  17. Article
  18. Article
  19. Article
  20. Journal of Alzheimer's disease reports · 2021
    Article

28 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

19 authors at 11 institutions in 2 countries.

Neda JahanshadImaging Genetics Center, Laboratory of Neuro Imaging, University of California at Los Angeles School of Medicine, Los Angeles, CA 90095, USA.
Priya Rajagopalan
Xue Hua
Derrek P Hibar
Talia M Nir
Arthur W Toga
Clifford R Jack
Andrew J Saykin
Robert C Green
Michael W Weiner
Sarah E Medland
Grant W Montgomery
Narelle K Hansell
Katie L McMahon
Greig I de Zubicaray
Nicholas G Martin
Margaret J Wright
Paul M Thompson
Alzheimer’s Disease Neuroimaging Initiative
University of California, Los Angeles · USQIMR Berghofer Medical Research Institute · AUUniversity of California, San Francisco · USHarvard University Press · USIndiana University – Purdue University Indianapolis · USIndiana University School of MedicineSan Francisco VA Medical Center · USBrigham and Women's Hospital · USMayo Clinic · USMayo Clinic in Arizona · USThe University of Queensland · AU

Funding

National Centralized Repository for Alzheimer's Disease and Related Dementias (NCRAD)U24AG021886 · INDIANA UNIV-PURDUE UNIV AT INDIANAPOLIS · 2002 to 2025
$19.9M
PROTEIN TRAFFICKING IN SPORADIC ALZHEIMER'S DISEASEP50AG008702 · COLUMBIA UNIV NEW YORK MORNINGSIDE · 1989 to 2005
$12.2M
VESICULAR LOCALIZATION AND FUNCTION OF PRESENILIN 1 FRAGMENTP50AG005138 · MOUNT SINAI SCHOOL OF MEDICINE OF NYU · 1985 to 2005
$11.9M
MOUSE BREEDIDNG COLONY FOR ALZHEIMER RESEARCHP30AG013846 · BOSTON UNIVERSITY MEDICAL CAMPUS · 1996 to 2005
$7.0M
Risk Evaluation and Education for Alzheimer's DiseaseR01HG002213 · BOSTON UNIVERSITY MEDICAL CAMPUS · 1999 to 2005
$3.5M
Training Program for Imaging Based Medical InformaticsT15LM007356 · UNIVERSITY OF CALIFORNIA LOS ANGELES · 2002 to 2005
$1.6M
NHGRI NIH HHS HG002213NHGRI NIH HHS HG006500NHGRI NIH HHS R01 HG002213NHGRI NIH HHS U01 HG006500NIA NIH HHS AG027841NIA NIH HHS K24 AG027841NIA NIH HHS P30 AG013846NIA NIH HHS P50 AG005138NIA NIH HHS P50 AG008702NIA NIH HHS R01 AG040060NIA NIH HHS U24 AG021886NIBIB NIH HHS P41 EB015922NIBIB NIH HHS R01 EB007813NIBIB NIH HHS R01 EB008281NIBIB NIH HHS R01 EB008432NICHD NIH HHS R01 HD050735NIMH NIH HHS R01 MH097268NINDS NIH HHS R01 NS080655NLM NIH HHS T15 LM007356NLM NIH HHS T15 LM07356
6 · The paper itself

Abstract

Aberrant connectivity is implicated in many neurological and psychiatric disorders, including Alzheimer's disease and schizophrenia. However, other than a few disease-associated candidate genes, we know little about the degree to which genetics play a role in the brain networks; we know even less about specific genes that influence brain connections. Twin and family-based studies can generate estimates of overall genetic influences on a trait, but genome-wide association scans (GWASs) can screen the genome for specific variants influencing the brain or risk for disease. To identify the heritability of various brain connections, we scanned healthy young adult twins with high-field, high-angular resolution diffusion MRI. We adapted GWASs to screen the brain's connectivity pattern, allowing us to discover genetic variants that affect the human brain's wiring. The association of connectivity with the SPON1 variant at rs2618516 on chromosome 11 (11p15.2) reached connectome-wide, genome-wide significance after stringent statistical corrections were enforced, and it was replicated in an independent subsample. rs2618516 was shown to affect brain structure in an elderly population with varying degrees of dementia. Older people who carried the connectivity variant had significantly milder clinical dementia scores and lower risk of Alzheimer's disease. As a posthoc analysis, we conducted GWASs on several organizational and topological network measures derived from the matrices to discover variants in and around genes associated with autism (MACROD2), development (NEDD4), and mental retardation (UBE2A) significantly associated with connectivity. Connectome-wide, genome-wide screening offers substantial promise to discover genes affecting brain connectivity and risk for brain diseases.

Indexed as

Genetic VariationTwins, DizygoticTwins, MonozygoticAdultAlzheimer DiseaseAutistic DisorderBrainChromosomes, Human, Pair 11Endosomal Sorting Complexes Required for TransportExtracellular Matrix ProteinsFemaleGenome-Wide Association StudyHumansMagnetic Resonance ImagingMaleNedd4 Ubiquitin Protein LigasesEndosomal Sorting Complexes Required for TransportExtracellular Matrix ProteinsNedd4 protein, humanNedd4 Ubiquitin Protein LigasesSPON1 protein, humanUBE2A protein, humanUbiquitin-Conjugating EnzymesUbiquitin-Protein Ligases

Identifiers

PMID23471985
PMCPMC3606977
OpenAlexW2067676324

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.