Evidence mapPaperPMID 23734218Full record

Trial reportPloS one2013

The association between progression of atherosclerosis and the methylated amino acids asymmetric dimethylarginine and trimethyllysine.

Kjetil H Løland, Oyvind Bleie, Heidi Borgeraas, Elin Strand, Per M Ueland, Asbjørn Svardal, Jan E Nordrehaug, Ottar Nygård

Registry-linked trialOpen access · goldAbstract readRandomized Controlled Trial
In one paragraph

Trial report in PloS one, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT00354081 (A Randomised Double Blind Study of the Effects of Homocysteine Lowering Therapy on Mortality and Cardiac Events in Patients Undergoing Coronary Angiography), which is not on this map. Cited by 16 papers.

0numbers the graph read from it
0cells of the map it votes in
16citing papers in PubMed
2.2field-weighted citation impact, top 11% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT00354081 phase3completednot on this map

A Randomised Double Blind Study of the Effects of Homocysteine Lowering Therapy on Mortality and Cardiac Events in Patients Undergoing Coronary Angiography

TypeinterventionalSponsorHaukeland University HospitalRan1999 to 2008Enrolled3,096ConditionsCoronary Artery Disease, Myocardial Infarction, Cerebrovascular StrokeArmsfolic acid, vitamin B12 (cyanocobalamin), vitamin B6 (pyridoxine), folic acid, vitamin B12 (cyanocobalamin), vitamin B6 (pyridoxine), placebo
3 · Its place in the literature

Who cites it

16 citing papers in PubMed, 34 citations in OpenAlex.

  1. Article
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  8. Advances in Stroke Prevention.Journal of translational internal medicine · 2018
    Article
  9. Review
  10. Article
  11. Observational
  12. Review
  13. Effect of renal impairment on atherosclerosis: only partially mediated by homocysteine.Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association · 2016
    Article
  14. Article
  15. Article
  16. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 3 institutions in 1 country.

Kjetil H LølandDepartment of Clinical Science, University of Bergen, Bergen, Norway.
Oyvind Bleie
Heidi Borgeraas
Elin Strand
Per M Ueland
Asbjørn Svardal
Jan E Nordrehaug
Ottar Nygård
University of Bergen · NOHaukeland University Hospital · NOSykehuset i Vestfold · NO

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

objectiveWe previously showed that treatment with folic acid (FA)/B12 was associated with more rapid progression of coronary artery disease (CAD). High doses of FA may induce methylation by increasing the availability of S-adenosyl-methionine (SAM). Asymmetric dimethylarginine (ADMA) and trimethyllysine (TML) are both produced through proteolytic release following post-translational SAM-dependent methylation of precursor amino acid. ADMA has previously been associated with CAD. We investigated if plasma levels of ADMA and TML were associated with progression of CAD as measured by quantitative coronary angiography (QCA).

methods183 patients from the Western Norway B Vitamin Intervention Trial (WENBIT) undergoing percutaneous coronary intervention (PCI) were randomized to daily treatment with 0.8 mg FA/0.4 mg B12 with and without 40 mg B6, B6 alone or placebo. Coronary angiograms and plasma samples of ADMA and TML were obtained at both baseline and follow-up (median 10.5 months). The primary end-point was progression of CAD as measured by diameter stenosis (DS) evaluated by linear quantile mixed models.

resultsA total of 309 coronary lesions not treated with PCI were identified. At follow-up median (95% CI) DS increased by 18.35 (5.22-31.49) percentage points per µmol/L ADMA increase (p-value 0.006) and 2.47 (0.37-4.58) percentage points per µmol/L TML increase (p-value 0.021) in multivariate modeling. Treatment with FA/B12 (±B6) was not associated with ADMA or TML levels.

conclusionIn patients with established CAD, baseline ADMA and TML was associated with angiographic progression of CAD. However, neither ADMA nor TML levels were altered by treatment with FA/B12 (±B6).

trial registrationControlled-Trials.com NCT00354081.

Indexed as

AdenosineAgedArginineCoronary AngiographyCoronary Artery DiseaseDisease ProgressionEthionineFemaleFolic AcidFollow-Up StudiesHumansLysineMaleMethylationMiddle AgedTreatment OutcomeAdenosineArginineEthionineFolic AcidLysineN,N-dimethylarginineS-adenosylethioninetrimethyllysineVitamin B 12Vitamin B 6Vitamin B Complex

Identifiers

PMID23734218
PMCPMC3666971
OpenAlexW2096851297

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.