Evidence mapPaperPMID 24304170Full record

Trial reportXenobiotica; the fate of foreign compounds in biological systems2014

Absorption, metabolism and excretion of [14C]gemigliptin, a novel dipeptidyl peptidase 4 inhibitor, in humans.

Namtae Kim, Lorna Patrick, Stuart Mair, Lloyd Stevens, Gill Ford, Vicky Birks, Sung-Hack Lee

Registry-linked trialAbstract readClinical Trial
PubMed Publisher
In one paragraph

Trial report in Xenobiotica; the fate of foreign compounds in biological systems, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT03310749 (A Randomized, Open-label, Multiple Dosing, Three-way Crossover Clinical Trial to Investigate the Pharmacokinetic Drug-drug Interaction of Gemigliptin and Metformin After Oral Administration in Healthy Mexican Male Subjects), which is not on this map. Cited by 11 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed, 1 pooled it
1.6field-weighted citation impact, top 15% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT03310749 phase1completedstarted 2016, after this paper: background citation

A Randomized, Open-label, Multiple Dosing, Three-way Crossover Clinical Trial to Investigate the Pharmacokinetic Drug-drug Interaction of Gemigliptin and Metformin After Oral Administration in Healthy Mexican Male Subjects

Ran2016Enrolled34Registered outcomes11Posted comparisons0ConditionsDiabetes Mellitus, Type 2ArmsGemigliptin, Gemigliptin 50 mg q.d. + metformin 1000 mg twice a day, Metformin
Open the trial in the graph
3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 1 synthesis or guideline pooled it, 22 citations in OpenAlex.

  1. Pooled it
  2. Trial
  3. Trial
  4. Trial
  5. Article
  6. Review
  7. Review
  8. Review
  9. Article
  10. Review
  11. Gemigliptin: Newer Promising Gliptin for Type 2 Diabetes Mellitus.Indian journal of endocrinology and metabolism
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 1 institution in 1 country.

Namtae KimLG Life Sciences, Drug Metabolism and Pharmacokinetics , Daejeon , Republic of Korea .
Lorna Patrick
Stuart Mair
Lloyd Stevens
Gill Ford
Vicky Birks
Sung-Hack Lee
Quotient Clinical (United Kingdom) · GB

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

1. Gemigliptin (formerly known as LC15-0444) is a newly developed dipeptidyl peptidase 4 inhibitor for the treatment of type 2 diabetes. Following oral administration of 50 mg (5.4 MBq) [(14)C]gemigliptin to healthy male subjects, absorption, metabolism and excretion were investigated. 2. A total of 90.5% of administered dose was recovered over 192 hr postdose, with 63.4% from urine and 27.1% from feces. Based on urinary recovery of radioactivity, a minimum 63.4% absorption from gastrointestinal tract could be confirmed. 3. Twenty-three metabolites were identified in plasma, urine and feces. In plasma, gemigliptin was the most abundant component accounting for 67.2% ∼ 100% of plasma radioactivity. LC15-0636, a hydroxylated metabolite of gemigliptin, was the only human metabolite with systemic exposure more than 10% of total drug-related exposure. Unchanged gemigliptin accounted for 44.8% ∼ 67.2% of urinary radioactivity and 27.7% ∼ 51.8% of fecal radioactivity. The elimination of gemigliptin was balanced between metabolism and excretion through urine and feces. CYP3A4 was identified as the dominant CYP isozyme converting gemigliptin to LC15-0636 in recombinant CYP/FMO enzymes.

Indexed as

Gastrointestinal AbsorptionAdministration, OralAdultCarbon RadioisotopesChromatography, High Pressure LiquidDipeptidyl-Peptidase IV InhibitorsFecesHumansMaleMetabolic Networks and PathwaysMiddle AgedPiperidonesProtein BindingPyrimidinesCarbon RadioisotopesDipeptidyl-Peptidase IV InhibitorsLC15-0444PiperidonesPyrimidines

Identifiers

PMID24304170
OpenAlexW2128783945

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.