Evidence mapPaperPMID 25213566Full record

Trial reportJournal of the American Heart Association2014

Effect of phosphodiesterase inhibition on insulin resistance in obese individuals.

Jennifer E Ho, Pankaj Arora, Geoffrey A Walford, Anahita Ghorbani, Derek P Guanaga, Bishnu P Dhakal, Daniel I Nathan, Emmanuel S Buys, Jose C Florez, Christopher Newton-Cheh and 2 more

Registry-linked trialOpen access · goldAbstract readRandomized Controlled Trial
In one paragraph

Trial report in Journal of the American Heart Association, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT01444651 (Phase 3 Randomized Trial of Tadalafil and Glycemic Traits), which is not on this map. Cited by 20 papers.

0numbers the graph read from it
0cells of the map it votes in
20citing papers in PubMed
3.7field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT01444651 phase3completednot on this map

Phase 3 Randomized Trial of Tadalafil and Glycemic Traits

TypeinterventionalSponsorThomas J. Wang, MDRan2011 to 2013Enrolled73ConditionsCardiovascular Disease, Insulin Resistance, Glucose Intolerance, ObesityArmsTadalafil, Placebo
3 · Its place in the literature

Who cites it

20 citing papers in PubMed, 38 citations in OpenAlex.

  1. Trial
  2. Trial
  3. Trial
  4. Article
  5. Endothelial Dysfunction in Obesity and Therapeutic Targets.Advances in experimental medicine and biology · 2024
    Review
  6. Review
  7. Article
  8. Review
  9. Role of Phosphodiesterase in the Biology and Pathology of Diabetes.International journal of molecular sciences · 2020
    Review
  10. Review
  11. 70-year legacy of the Framingham Heart Study.Nature reviews. Cardiology · 2019
    Review
  12. Review
  13. Article
  14. Article
  15. The Vasculature in Prediabetes.Circulation research · 2018
    Review
  16. Article
  17. Article
  18. Review
  19. Review
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors at 7 institutions in 1 country.

Jennifer E HoCardiovascular Medicine Section, Department of Medicine, Boston University School of Medicine, Boston, MA (J.E.H.).
Pankaj AroraDivision of Cardiology, Department of Medicine, University of Alabama at Birmingham, Birmingham, AL (P.A.).
Geoffrey A WalfordDiabetes Center, Massachusetts General Hospital, Boston, MA (G.A.W., J.C.F.).
Anahita GhorbaniDepartment of Medicine, Mount Auburn Hospital, Cambridge, MA (A.G.).
Derek P GuanagaDepartment of Surgery, Brigham and Women's Hospital, Boston, MA (D.P.G.).
Bishnu P DhakalCardiology Division, Department of Medicine, Massachusetts General Hospital, Boston, MA (B.P.D., C.N.C., G.D.L.).
Daniel I NathanDepartment of Anesthesia, Critical Care, and Pain Medicine, Massachusetts General Hospital, Boston, MA (D.I.N., E.S.B.).
Emmanuel S BuysDepartment of Anesthesia, Critical Care, and Pain Medicine, Massachusetts General Hospital, Boston, MA (D.I.N., E.S.B.).
Jose C FlorezDiabetes Center, Massachusetts General Hospital, Boston, MA (G.A.W., J.C.F.) Center for Human Genetic Research, Massachusetts General Hospital, Boston, MA (J.C.F.).
Christopher Newton-ChehCardiology Division, Department of Medicine, Massachusetts General Hospital, Boston, MA (B.P.D., C.N.C., G.D.L.) Cardiovascular Research Center, Massachusetts General Hospital, Boston, MA (C.N.C.) Broad Institute of Harvard and MIT, Cambridge, MA (C.N.C.).
Gregory D LewisCardiology Division, Department of Medicine, Massachusetts General Hospital, Boston, MA (B.P.D., C.N.C., G.D.L.).
Thomas J WangCardiovascular Medicine Division, Department of Medicine, Vanderbilt University, Nashville, TN (T.J.W.).
Massachusetts General Hospital · USBoston University · USBrigham and Women's Hospital · USCenter for Human Genetics · USMount Auburn Hospital · USUniversity of Alabama at Birmingham · USVanderbilt University · US

Funding

NHLBI NIH HHS K23 HL116780NHLBI NIH HHS K23-HL116780NHLBI NIH HHS R01 HL086875NHLBI NIH HHS R01-HL086875NHLBI NIH HHS R01 HL098283NHLBI NIH HHS R01-HL098283NHLBI NIH HHS R01 HL113933NHLBI NIH HHS R01-HL113933
6 · The paper itself

Abstract

backgroundObesity is associated with cardiometabolic disease, including insulin resistance (IR) and diabetes. Cyclic guanosine monophosphate (cGMP) signaling affects energy balance, IR, and glucose metabolism in experimental models. We sought to examine effects of phosphodiesterase-5 inhibition with tadalafil on IR in a pilot study of obese nondiabetic individuals. METHODS AND

resultsWe conducted a randomized, double-blinded, placebo-controlled trial of adults age 18 to 50 years with obesity and elevated fasting insulin levels (≥10 μU/mL). Participants were randomized to tadalafil 20 mg daily or placebo for 3 months. Oral glucose tolerance tests were performed, and the effect of tadalafil on IR was examined. A total of 53 participants (mean age, 33 years; body mass index [BMI], 38 kg/m(2)) were analyzed, 25 randomized to tadalafil and 28 to placebo. In the overall sample, measures of IR did not differ between tadalafil and placebo groups at 3 months. However, in individuals with severe obesity (BMI ≥36.2 kg/m(2)), tadalafil use was associated with improved IR (homeostatic model assessment for IR), compared to placebo (P=0.02, respectively). Furthermore, one measure of β-cell compensation for IR (oral disposition index) improved with tadalafil in the overall sample (P=0.009) and in the subgroup with severe obesity (P=0.01).

conclusionResults of this pilot study did not show improvements in IR with tadalafil, compared to placebo. However, tadalafil may have favorable effects on β-cell compensation, particularly in individuals with severe obesity. Future studies evaluating the potential metabolic benefits of cGMP modulation in obesity are warranted. CLINICAL TRIAL REGISTRATION URL: ClinicalTrials.gov. Unique Identifier: NCT01444651.

Indexed as

Insulin ResistanceAdolescentAdultBiomarkersBlood GlucoseBody Mass IndexBostonCarbolinesDouble-Blind MethodFemaleHumansInsulinInsulin-Secreting CellsMaleMiddle AgedObesityBiomarkersBlood GlucoseCarbolinesInsulinPhosphodiesterase 5 InhibitorsTadalafilcGMPinsulin resistanceobesityphosphodiesterase type 5 inhibition

Identifiers

PMID25213566
PMCPMC4323801
OpenAlexW2157833783

What Socratic holds

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LicenceCC BY-NC
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Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.