Evidence mapPaperPMID 2573554Full record

ArticleDiabetes1989

Insulin regulation of lipolysis in nondiabetic and IDDM subjects.

M D Jensen, M Caruso, V Heiling, J M Miles

Registry-linked trialAbstract read
PubMed Publisher
In one paragraph

Article in Diabetes, 1989. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT02045290 (Metformin Therapy for Overweight Adolescents With Type 1 Diabetes), which is not on this map. Cited by 73 papers.

0numbers the graph read from it
0cells of the map it votes in
73citing papers in PubMed
4.6field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT02045290 phase3completedstarted 2014, after this paper: background citation

Metformin Therapy for Overweight Adolescents With Type 1 Diabetes (T1D)--Insulin Clamp Ancillary Study for Assessment of Insulin Resistance

Ran2014Enrolled37Registered outcomes3Posted comparisons0ConditionsType 1 DiabetesArmsMetformin, Placebo
Open the trial in the graph
3 · Its place in the literature

Who cites it

73 citing papers in PubMed, 315 citations in OpenAlex.

  1. Optimizing measures of insulin-regulated lipolysis in humans.American journal of physiology. Endocrinology and metabolism · 2025
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  6. Effect of Dehydroepiandrosterone and Testosterone Supplementation on Systemic Lipolysis.The Journal of clinical endocrinology and metabolism · 2016
    Trial
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  11. Article
  12. Article
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  14. Human Adipose Tissue Metabolism in Obesity.Journal of obesity & metabolic syndrome · 2025
    Review
  15. Article
  16. Review
  17. Article
  18. Observational
  19. Article
  20. Article

13 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 2 countries.

M D JensenDepartment of Internal Medicine, Mayo Clinic, Rochester, Minnesota 55905.
M Caruso
V Heiling
J M Miles
Mayo Clinic in Florida · USInstitute of Nutrition, Metabolism and Diabetes · CA

Funding

ZESTRIL VS. VERAPAMIL IN THE TREATMENT OF HYPERTENSIONM01RR000585 · MAYO CLINIC COLL OF MEDICINE, ROCHESTER · 1985 to 2005
$26.6M
CONTROL OF BCAA METABOLISM IN NORMAL AND DIABETIC MANR01DK026989 · UNIVERSITY OF FLORIDA · 1986 to 1992
REGULATION OF FFA METABOLISM IN NORMAL &DIABETIC HUMANSR01DK038092 · SAINT LUKE'S HOSPITAL · 1987 to 1996
NCRR NIH HHS RR-0585NIDDK NIH HHS DK-26989NIDDK NIH HHS DK-38092
6 · The paper itself

Abstract

To determine whether insulin regulation of lipolysis is abnormal in subjects with poorly controlled insulin-dependent diabetes mellitus (IDDM), free-fatty acid flux ([1-14C]palmitate) was measured under conditions ranging from complete insulin withdrawal to hyperinsulinemia. Seven nondiabetic and seven IDDM subjects were studied with the pancreatic clamp technique to control plasma insulin, growth hormone, and glucagon concentrations at the desired levels. Preliminary studies were performed to validate the experimental design. The palmitate flux response to insulin withdrawal (2.5 +/- 0.2 vs. 2.5 +/- 0.2 mumol.kg-1.min-1) and maximally antilipolytic insulin concentrations (0.17 +/- 0.02 vs. 0.23 +/- 0.03 mumol.kg-1.min-1) were not different in nondiabetic and IDDM subjects, respectively. In contrast, IDDM subjects required significantly greater plasma free-insulin concentrations to result in equivalent suppression of palmitate flux compared with nondiabetic subjects. Lipolysis was found to be very sensitive to insulin in nondiabetic humans, with half-maximal suppression occurring at plasma free-insulin concentrations of approximately 12 pM (less than 2 microU/ml). We conclude that adipose tissue lipolysis is normally exquisitely sensitive to insulin and that sensitivity, but not responsiveness to insulin, is impaired in poorly controlled IDDM.

Indexed as

AdultBlood GlucoseC-PeptideDiabetes Mellitus, Type 1Fatty Acids, NonesterifiedFemaleGlucagonGrowth HormoneHumansHydrocortisoneInsulinKineticsLipolysisMalePalmitic AcidPalmitic AcidsBlood GlucoseC-PeptideFatty Acids, NonesterifiedGlucagonGrowth HormoneHydrocortisoneInsulinPalmitic AcidPalmitic AcidsSomatostatin

Identifiers

PMID2573554
OpenAlexW2003950656

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.