Evidence map›Paper›PMID 25908760›Full record

ArticleArteriosclerosis, thrombosis, and vascular biology2015

Forkhead box O-1 modulation improves endothelial insulin resistance in human obesity.

Shakun Karki, Melissa G Farb, Doan T M Ngo, Samantha Myers, Vishwajeet Puri, Naomi M Hamburg, Brian Carmine, Donald T Hess, Noyan Gokce

Open access · greenAbstract read
In one paragraph

Article in Arteriosclerosis, thrombosis, and vascular biology, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.

0numbers the graph read from it
0cells of the map it votes in
17citing papers in PubMed
3.4field-weighted citation impact, top 8% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

17 citing papers in PubMed, 38 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Article
  5. Review
  6. Fat-Specific Protein 27 Regulation of Vascular Function in Human Obesity.Journal of the American Heart Association · 2019
    Article
  7. Article
  8. Article
  9. Article
  10. Review
  11. WNT5A regulates adipose tissue angiogenesis via antiangiogenic VEGF-AAmerican journal of physiology. Heart and circulatory physiology · 2017
    Article
  12. Article
  13. Impact of Diabetes Mellitus.Arteriosclerosis, thrombosis, and vascular biology · 2016
    Review
  14. Review
  15. Ageing, metabolism and cardiovascular disease.The Journal of physiology · 2016
    Review
  16. Article
  17. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 3 institutions in 2 countries.

Shakun KarkiFrom the Evans Department of Medicine and Whitaker Cardiovascular Institute (S.K., M.G.F., D.T.M.N., S.M., V.P., N.M.H., N.G.) and Department of General Surgery (B.C., D.T.H.), Boston University School of Medicine, MA.
Melissa G FarbFrom the Evans Department of Medicine and Whitaker Cardiovascular Institute (S.K., M.G.F., D.T.M.N., S.M., V.P., N.M.H., N.G.) and Department of General Surgery (B.C., D.T.H.), Boston University School of Medicine, MA.
Doan T M NgoFrom the Evans Department of Medicine and Whitaker Cardiovascular Institute (S.K., M.G.F., D.T.M.N., S.M., V.P., N.M.H., N.G.) and Department of General Surgery (B.C., D.T.H.), Boston University School of Medicine, MA.
Samantha MyersFrom the Evans Department of Medicine and Whitaker Cardiovascular Institute (S.K., M.G.F., D.T.M.N., S.M., V.P., N.M.H., N.G.) and Department of General Surgery (B.C., D.T.H.), Boston University School of Medicine, MA.
Vishwajeet PuriFrom the Evans Department of Medicine and Whitaker Cardiovascular Institute (S.K., M.G.F., D.T.M.N., S.M., V.P., N.M.H., N.G.) and Department of General Surgery (B.C., D.T.H.), Boston University School of Medicine, MA.
Naomi M HamburgFrom the Evans Department of Medicine and Whitaker Cardiovascular Institute (S.K., M.G.F., D.T.M.N., S.M., V.P., N.M.H., N.G.) and Department of General Surgery (B.C., D.T.H.), Boston University School of Medicine, MA.
Brian CarmineFrom the Evans Department of Medicine and Whitaker Cardiovascular Institute (S.K., M.G.F., D.T.M.N., S.M., V.P., N.M.H., N.G.) and Department of General Surgery (B.C., D.T.H.), Boston University School of Medicine, MA.
Donald T HessFrom the Evans Department of Medicine and Whitaker Cardiovascular Institute (S.K., M.G.F., D.T.M.N., S.M., V.P., N.M.H., N.G.) and Department of General Surgery (B.C., D.T.H.), Boston University School of Medicine, MA.
Noyan GokceFrom the Evans Department of Medicine and Whitaker Cardiovascular Institute (S.K., M.G.F., D.T.M.N., S.M., V.P., N.M.H., N.G.) and Department of General Surgery (B.C., D.T.H.), Boston University School of Medicine, MA. Noyan.Gokce@bmc.org.
Norwegian Womens Public Health Association · NOMaine Farmland Trust · USCardiovascular Institute of the South · US

Funding

Redox regulation in Endothelial Progenitor CellsP01HL081587 · NHLBI · BOSTON UNIVERSITY MEDICAL CAMPUS · PI WALSH, KENNETH · 2005 to 2015
$21.5M
Multidisciplinary Training in Cardiovascular ResearchT32HL007224 · NHLBI · BOSTON UNIVERSITY MEDICAL CAMPUS · PI HAMBURG, NAOMI MIRIAM, WAINFORD, RICHARD DAVID · 1985 to 2020
$12.3M
Anti-Angiogenic Mechanisms in Human ObesityR01HL126141 · NHLBI · BOSTON UNIVERSITY MEDICAL CAMPUS · PI GOKCE, NOYAN, WALSH, KENNETH · 2015 to 2019
$2.4M
Adipose inflammation, mitochondrial function & endothelial phenotypes in obesityR01HL114675 · NHLBI · BOSTON UNIVERSITY MEDICAL CAMPUS · PI GOKCE, NOYAN · 2012 to 2016
$2.3M
Endothelial Insulin Resistance, Inflammation and Vascular Function in DiabetesR01HL102299 · NHLBI · BOSTON UNIVERSITY MEDICAL CAMPUS · PI HAMBURG, NAOMI MIRIAM · 2010 to 2014
$2.0M
NHLBI NIH HHS HL081587NHLBI NIH HHS HL114675NHLBI NIH HHS HL126141NHLBI NIH HHS P01 HL081587NHLBI NIH HHS R01 HL102299NHLBI NIH HHS R01 HL114675NHLBI NIH HHS R01 HL126141NHLBI NIH HHS T32 HL007224NHLBI NIH HHS T32 HL07224
6 · The paper itself

Abstract

objectiveIncreased visceral adiposity has been closely linked to insulin resistance, endothelial dysfunction, and cardiometabolic disease in obesity, but pathophysiological mechanisms are poorly understood. We sought to investigate mechanisms of vascular insulin resistance by characterizing depot-specific insulin responses and gain evidence that altered functionality of transcription factor forkhead box O-1 (FOXO-1) may play an important role in obesity-related endothelial dysfunction. APPROACH AND

resultsWe intraoperatively collected paired subcutaneous and visceral adipose tissue samples from 56 severely obese (body mass index, 43 ± 7 kg/m(2)) and 14 nonobese subjects during planned surgical operations, and characterized depot-specific insulin-mediated responses using Western blot and quantitative immunofluorescence techniques. Insulin signaling via phosphorylation of FOXO-1 and consequent endothelial nitric oxide synthase stimulation was selectively impaired in the visceral compared with subcutaneous adipose tissue and endothelial cells of obese subjects. In contrast, tissue actions of insulin were preserved in nonobese individuals. Pharmacological antagonism with AS1842856 and biological silencing using small interfering RNA-mediated FOXO-1 knockdown reversed insulin resistance and restored endothelial nitric oxide synthase activation in the obese.

conclusionsWe observed profound endothelial insulin resistance in the visceral adipose tissue of obese humans which improved with FOXO-1 inhibition. FOXO-1 modulation may represent a novel therapeutic target to diminish vascular insulin resistance. In addition, characterization of endothelial insulin resistance in the adipose microenvironment may provide clues to mechanisms of systemic disease in human obesity.

Indexed as

AdultDelayed-Action PreparationsEndothelial CellsEndothelium, VascularEnzyme ActivationFemaleForkhead Box Protein O1Forkhead Transcription FactorsHumansInsulinInsulin ResistanceMaleMiddle AgedNitric Oxide Synthase Type IIIObesityPhosphorylation5-amino-7-(cyclohexylamino)-1-ethyl-6-fluoro-4-oxo-1,4-dihydroquinoline-3-carboxylic acidDelayed-Action PreparationsForkhead Box Protein O1Forkhead Transcription FactorsFOXO1 protein, humanInsulinNitric Oxide Synthase Type IIIQuinolonesFOXO1 protein, humaninsulinnitric oxide synthase type IIIobesity

Identifiers

PMID25908760
PMCPMC4441602
OpenAlexW2152093002

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.