Evidence map›Paper›PMID 26466326›Full record

ReviewJournal of cardiovascular pharmacology2016

Third-generation Mineralocorticoid Receptor Antagonists: Why Do We Need a Fourth?

Elise P Gomez-Sanchez

Abstract readReview
In one paragraph

Review in Journal of cardiovascular pharmacology, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 26 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
26citing papers in PubMed, 1 pooled it
3.1field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

26 citing papers in PubMed, 1 synthesis or guideline pooled it, 50 citations in OpenAlex.

  1. Pooled it
  2. Trial
  3. Review
  4. Article
  5. Article
  6. Article
  7. Review
  8. Review
  9. Article
  10. Article
  11. Review
  12. Article
  13. Review
  14. Article
  15. Review
  16. Article
  17. Mineralocorticoid Receptor Antagonists in Diabetic Kidney Disease.Pharmaceuticals (Basel, Switzerland) · 2021
    Review
  18. Review
  19. Article
  20. The renin-angiotensin system in cardiovascular autonomic control: recent developments and clinical implications.Clinical autonomic research : official journal of the Clinical Autonomic Research Society · 2019
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author at 1 institution in 2 countries.

Elise P Gomez-SanchezDepartments of *Pharmacology and Toxicology; †Medicine; and ‡Neurobiology and Anatomical Science, University of Mississippi Medical Center, Jackson, MS.
Institute of Pharmacology · CZ

Funding

ROLE OF MINERALOCORTICOIDS IN LOW-RENIN HYPERTENSIONR01HL027255 · NHLBI · UNIVERSITY OF SOUTH FLORIDA · PI GOMEZ-SANCHEZ, CELSO ENRIQUE · 1985 to 2017
$3.9M
STEROIDS IN EXPERIMENTAL HYPERTENSIONR01HL027737 · NHLBI · UNIVERSITY OF SOUTH FLORIDA · PI GOMEZ-SANCHEZ, ELISE PEERY · 1985 to 2003
$785k
Regulation of the late-pathway of aldosterone biosynthesisR21HL105383 · NHLBI · UNIVERSITY OF MISSISSIPPI MED CTR · PI GOMEZ-SANCHEZ, CELSO ENRIQUE · 2011 to 2012
$351k
Aldosterone Action In The Brain, Hypertension and InflammationI01BX000227 · VA · G V SONNY MONTGOMERY VA MEDCIAL CENTER · PI GOMEZ-SANCHEZ, ELISE PEERY · 2009 to 2012
–
BLRD VA I01 BX000227NHLBI NIH HHS HL105383NHLBI NIH HHS HL27255NHLBI NIH HHS R01 HL027255NHLBI NIH HHS R01 HL027737NHLBI NIH HHS R21 HL105383
6 · The paper itself

Abstract

The first mineralocorticoid receptor (MR) antagonist, spironolactone, was developed almost 60 years ago to treat primary aldosteronism and pathological edema. Its use waned in part because of its lack of selectivity. Subsequently, knowledge of the scope of MR function was expanded along with clinical evidence of the therapeutic importance of MR antagonists to prevent the ravages of inappropriate MR activation. Forty-two years elapsed between the first and MR-selective second generation of MR antagonists. Fifteen years later, despite serious shortcomings of the existing antagonists, a third-generation antagonist has yet to be marketed. Progress has been slowed by the lack of appreciation of the large variety of cell types that express the MR and its diverse cell-type-specific actions, and also its unique complex interaction actions at the molecular level. New MR antagonists should preferentially target the inflammatory and fibrotic effects of MR and perhaps its excitatory effects on sympathetic nervous system, but not the renal tubular epithelium or neurons of the cortex and hippocampus. This review briefly describes efforts to develop a third-generation MR antagonist and why fourth generation antagonists and selective agonists based on structural determinants of tissue and ligand-specific MR activation should be contemplated.

Indexed as

AnimalsDrug DiscoveryHumansHyperaldosteronismMineralocorticoid Receptor AntagonistsProtein BindingReceptors, MineralocorticoidSignal TransductionSpironolactoneMineralocorticoid Receptor AntagonistsReceptors, MineralocorticoidSpironolactone

Identifiers

PMID26466326
PMCPMC4703541
OpenAlexW2463628099

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.