Evidence map›Paper›PMID 26567197›Full record

ReviewAdvances in nutrition (Bethesda, Md.)2015

Health implications of high-fructose intake and current research.

Waleska C Dornas, Wanderson G de Lima, Maria L Pedrosa, Marcelo E Silva

Open access · bronzeAbstract readReview
In one paragraph

Review in Advances in nutrition (Bethesda, Md.), 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 40 papers.

0numbers the graph read from it
0cells of the map it votes in
40citing papers in PubMed
4.5field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

40 citing papers in PubMed, 103 citations in OpenAlex.

  1. Trial
  2. D-Fructose Exposure Impairs Neuronal Development in Mouse Neural Stem Cells.International journal of molecular sciences · 2026
    Article
  3. Article
  4. Review
  5. Article
  6. Article
  7. The Effects of the Ethanol Extract ofJournal of experimental pharmacology · 2025
    Article
  8. Review
  9. Review
  10. Article
  11. Drug design, development and therapy · 2024
    Article
  12. Article
  13. Article
  14. Article
  15. Article
  16. Review
  17. Metabolism and Health Impacts of Dietary Sugars.Journal of lipid and atherosclerosis · 2022
    Review
  18. Article
  19. Article
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 1 institution in 1 country.

Waleska C DornasResearch in Biological Sciences-Center for Research in Biological Sciences (NUPEB), w.dornas@ig.com.br.
Wanderson G de LimaResearch in Biological Sciences-Center for Research in Biological Sciences (NUPEB), Department of Biological Sciences, Institute of Exact and Biological Sciences, and.
Maria L PedrosaResearch in Biological Sciences-Center for Research in Biological Sciences (NUPEB), Department of Biological Sciences, Institute of Exact and Biological Sciences, and.
Marcelo E SilvaResearch in Biological Sciences-Center for Research in Biological Sciences (NUPEB), Department of Foods, School of Nutrition, Federal University of Ouro Preto, Minas Gerais, Brazil.
Universidade Federal de Ouro Preto · BR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Although fructose consumption has dramatically increased and is suspected to be causally linked to metabolic abnormalities, the mechanisms involved are still only partially understood. We discuss the available data and investigate the effects of dietary fructose on risk factors associated with metabolic disorders. The evidence suggests that fructose may be a predisposing cause in the development of insulin resistance in association with the induction of hypertriglyceridemia. Experiments in animals have shown this relation when they are fed diets very high in fructose or sucrose, and human studies also show this relation, although with conflicting results due to the heterogeneity of the studies. The link between increased fructose consumption and increases in uric acid also has been confirmed as a potential risk factor for metabolic syndrome, and insulin resistance/hyperinsulinemia may be causally related to the development of hypertension. Collectively, these results suggest a link between high fructose intake and insulin resistance, although future studies must be of reasonable duration, use defined populations, and improve comparisons regarding the effects of relevant doses of nutrients on specific endpoints to fully understand the effect of fructose intake in the absence of potential confounding factors.

Indexed as

AnimalsDietDietary SucroseFructoseHumansHypertensionHypertriglyceridemiaInsulinInsulin ResistanceLipid MetabolismMetabolic DiseasesMiceObesityRatsRisk FactorsUric AcidDietary SucroseFructoseInsulinUric Acidantioxidantsfructosehypertensionoxidative stresssugar-sweetened beverages

Identifiers

PMID26567197
PMCPMC4642413
OpenAlexW2239083546

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.