Evidence mapPaperPMID 26609792Full record

Trial reportBritish journal of clinical pharmacology2016

Exenatide acutely increases heart rate in parallel with augmented sympathetic nervous system activation in healthy overweight males.

Mark M Smits, Marcel H A Muskiet, Lennart Tonneijck, Trynke Hoekstra, Mark H H Kramer, Michaela Diamant, Daniël H van Raalte

Open access · bronzeAbstract readClinical Trial
In one paragraph

Trial report in British journal of clinical pharmacology, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 31 papers, 3 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
31citing papers in PubMed, 3 pooled it
5.3field-weighted citation impact, top 4% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

31 citing papers in PubMed, 3 syntheses or guidelines pooled it, 57 citations in OpenAlex.

  1. Pooled it
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  4. Trial
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  8. Comment on "The effects of glucagon-like peptide-1 receptor agonists on sympathetic neuron activity".Hypertension research : official journal of the Japanese Society of Hypertension · 2026
    Article
  9. The effects of glucagon-like peptide-1 receptor agonists on sympathetic neuron activity.Hypertension research : official journal of the Japanese Society of Hypertension · 2026
    Article
  10. Review
  11. Observational
  12. Review
  13. Review
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  17. Observational
  18. Safety of Semaglutide.Frontiers in endocrinology · 2021 · on this map
    Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 3 institutions in 2 countries.

Mark M SmitsDiabetes Center, Department of Internal Medicine, VU University Medical Center, Amsterdam.
Marcel H A MuskietDiabetes Center, Department of Internal Medicine, VU University Medical Center, Amsterdam.
Lennart TonneijckDiabetes Center, Department of Internal Medicine, VU University Medical Center, Amsterdam.
Trynke HoekstraDepartment of Health Sciences and the EMGO Institute for Health and Care Research, VU University Amsterdam, Amsterdam.
Mark H H KramerDiabetes Center, Department of Internal Medicine, VU University Medical Center, Amsterdam.
Michaela DiamantDiabetes Center, Department of Internal Medicine, VU University Medical Center, Amsterdam.
Daniël H van RaalteDiabetes Center, Department of Internal Medicine, VU University Medical Center, Amsterdam.
Amsterdam UMC Location VUmc · NLUniversity Hospital and Clinics · USUniversity Medical Center · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

aimClinical use of glucagon-like peptide-1 receptor agonists (GLP-1RA) is consistently associated with heart rate (HR) acceleration in type 2 diabetes patients. We explored the mechanisms underlying this potential safety concern.

methodsTen healthy overweight males (aged 20-27 years) were examined in an open label, crossover study. Automated oscillometric blood pressure measurements and finger photoplethysmography were performed throughout intravenous administration of placebo (saline 0.9%), exenatide (targeting therapeutic concentrations) and a combination of exenatide and the nitric oxide synthase inhibitor L-N(G) -monomethyl arginine (L-NMMA). Sympathetic nervous system (SNS) activity was measured by heart rate variability and rate-pressure product.

resultsExenatide increased HR by a mean maximum of 6.8 (95% CI 1.7, 11.9) beats min(-1) (P < 0.05), systolic blood pressure (SBP) by 9.8 (95% CI 3.5, 16.1) mmHg (P < 0.01) and markers of SNS activity (P < 0.05). No changes in total peripheral resistance were observed. Increases in HR, SBP and sympathetic activity were preserved during concomitant L-NMMA infusion.

conclusionsOur data argue against exenatide-induced reflex tachycardia as a response to vasodilation and rather suggest the involvement of SNS activation in humans.

Indexed as

OverweightAdultBlood GlucoseBody Mass IndexCross-Over StudiesExenatideGlucagon-Like Peptide 1Healthy VolunteersHeart RateHumansLipidsMalePeptidesSympathetic Nervous SystemVascular ResistanceVenomsBlood GlucoseExenatideGlucagon-Like Peptide 1LipidsPeptidesVenomsGLP-1 receptor agonisthaemodynamicsheart ratesympathetic nervous system activity

Identifiers

PMID26609792
PMCPMC4799913
OpenAlexW2176491627

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.