Evidence map›Paper›PMID 26837740›Full record

ArticleCirculation research2016

Coronary Artery Disease and Its Risk Factors: Leveraging Shared Genetics to Discover Novel Biology.

Thomas Quertermous, Erik Ingelsson

Abstract readEditorialComment
In one paragraph

Article in Circulation research, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed.

  1. Article
  2. Article
  3. Article
  4. Selective Inhibition of PKCJournal of diabetes research · 2020
    Article
  5. Article
  6. Article
  7. Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

2 authors.

Thomas QuertermousFrom the Division of Cardiovascular Medicine, Department of Medicine (T.Q., E.I.), Cardiovascular Institute (T.Q., E.I.), Stanford University School of Medicine, CA; and Molecular Epidemiology and Science for Life Laboratory, Department of Medical Sciences, Uppsala University, Uppsala, Sweden (E.I.). tomq1@stanford.edu.
Erik IngelssonFrom the Division of Cardiovascular Medicine, Department of Medicine (T.Q., E.I.), Cardiovascular Institute (T.Q., E.I.), Stanford University School of Medicine, CA; and Molecular Epidemiology and Science for Life Laboratory, Department of Medical Sciences, Uppsala University, Uppsala, Sweden (E.I.).

Funding

Identifying the gene networks of insulin resistance: the GENESIPS studyU01HL107388 · NHLBI · STANFORD UNIVERSITY · PI QUERTERMOUS, THOMAS, SCHADT, ERIC E · 2011 to 2015
$8.8M
Mechanism of the Coronary Heart Disease Association at Chromosome 6q23.2R01HL109512 · NHLBI · STANFORD UNIVERSITY · PI QUERTERMOUS, THOMAS · 2011 to 2019
$6.1M
Molecular Mechanisms of Insulin Resistance Associated LociR01DK107437 · NIDDK · STANFORD UNIVERSITY · PI QUERTERMOUS, THOMAS · 2016 to 2019
$2.2M
Identification of causal coronary heart disease variation in smooth muscle cellsR33HL120757 · NHLBI · STANFORD UNIVERSITY · PI QUERTERMOUS, THOMAS · 2016 to 2018
$1.6M
Identification of causal coronary heart disease variation in smooth muscle cellsR21HL120757 · NHLBI · STANFORD UNIVERSITY · PI QUERTERMOUS, THOMAS · 2014 to 2015
$434k
NHLBI NIH HHS HL109512NHLBI NIH HHS R01 HL109512NHLBI NIH HHS R21 HL120757NHLBI NIH HHS R21HL120757NHLBI NIH HHS R33 HL120757NHLBI NIH HHS U01 HL107388NHLBI NIH HHS U01HL107388NIDDK NIH HHS R01 DK107437
6 · The paper itself

Abstract

PubMed holds no abstract for this paper.

Indexed as

Coronary Artery DiseaseFemaleGenetic Predisposition to DiseaseGenetic VariationGenome-Wide Association StudyHumanscholesterol, LDLcoronary diseasediabetes mellitusEditorialsgeneticsgenome-wide association study

Identifiers

PMID26837740
PMCPMC4743893

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.