Evidence mapPaperPMID 26895605Full record

ArticleInternational journal of antimicrobial agents2016

Repurposing auranofin for the treatment of cutaneous staphylococcal infections.

Shankar Thangamani, Haroon Mohammad, Mostafa F N Abushahba, Tiago J P Sobreira, Mohamed N Seleem

Open access · greenAbstract read
In one paragraph

Article in International journal of antimicrobial agents, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 47 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
47citing papers in PubMed, 1 pooled it
7.9field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

47 citing papers in PubMed, 1 synthesis or guideline pooled it, 85 citations in OpenAlex.

  1. Pooled it
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  5. Review
  6. Review
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  11. Metal Complexes as Antiviral Agents for SARS-CoV-2.Chembiochem : a European journal of chemical biology · 2021
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  18. Anti-virulence strategies forGut microbes · 2020
    Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 2 countries.

Shankar ThangamaniDepartment of Comparative Pathobiology, Purdue University College of Veterinary Medicine, West Lafayette, IN, USA.
Haroon MohammadDepartment of Comparative Pathobiology, Purdue University College of Veterinary Medicine, West Lafayette, IN, USA.
Mostafa F N AbushahbaDepartment of Comparative Pathobiology, Purdue University College of Veterinary Medicine, West Lafayette, IN, USA; Department of Animal Hygiene and Zoonoses, Faculty of Veterinary Medicine, Assiut University, Assiut, Egypt.
Tiago J P SobreiraBindley Bioscience Center, Purdue University, West Lafayette, IN, USA.
Mohamed N SeleemDepartment of Comparative Pathobiology, Purdue University College of Veterinary Medicine, West Lafayette, IN, USA. Electronic address: mseleem@purdue.edu.
Purdue University West Lafayette · US

Funding

NIAID NIH HHS HHSN272200700055CNIAID NIH HHS R56 AI114861NIAID NIH HHS R56AI114861
6 · The paper itself

Abstract

The scourge of multidrug-resistant bacterial infections necessitates the urgent development of novel antimicrobials to address this public health challenge. Drug repurposing is a proven strategy to discover new antimicrobial agents; given that these agents have undergone extensive toxicological and pharmacological analysis, repurposing is an effective method to reduce the time, cost and risk associated with traditional antibiotic innovation. In this study, the in vitro and in vivo antibacterial activities of an antirheumatic drug, auranofin, was investigated against multidrug-resistant Staphylococcus aureus. The results indicated that auranofin possesses potent antibacterial activity against all tested strains of S. aureus, including meticillin-resistant S. aureus (MRSA), vancomycin-intermediate S. aureus (VISA) and vancomycin-resistant S. aureus (VRSA), with minimum inhibitory concentrations (MICs) ranging from 0.0625μg/mL to 0.125μg/mL. In vivo, topical auranofin proved superior to conventional antimicrobials, including fusidic acid and mupirocin, in reducing the mean bacterial load in infected wounds in a murine model of MRSA skin infection. In addition to reducing the bacterial load, topical treatment of auranofin greatly reduced the production of inflammatory cytokines, including tumour necrosis factor-α (TNFα), interleukin-6 (IL-6), interleukin-1 beta (IL-1β) and monocyte chemoattractant protein-1 (MCP-1), in infected skin lesions. Moreover, auranofin significantly disrupted established in vitro biofilms of S. aureus and Staphylococcus epidermidis, more so than the traditional antimicrobials linezolid and vancomycin. Taken together, these results support that auranofin has potential to be repurposed as a topical antimicrobial agent for the treatment of staphylococcal skin and wound infections.

Indexed as

Drug RepositioningAnimalsAnti-Bacterial AgentsAuranofinBiofilmsCell LineChemokine CCL2Drug Resistance, Multiple, BacterialDrug Therapy, CombinationFemaleFusidic AcidHumansInterleukin-1betaInterleukin-6Methicillin-Resistant Staphylococcus aureusMiceAnti-Bacterial AgentsAuranofinCcl2 protein, mouseChemokine CCL2Fusidic AcidIL1B protein, mouseInterleukin-1betaInterleukin-6MupirocinTumor Necrosis Factor-alphaAuranofinInflammatory cytokinesMultidrug resistanceRepurposingTopical antimicrobials

Identifiers

PMID26895605
PMCPMC4792765
OpenAlexW2275898223

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.