Evidence mapPaperPMID 26920146Full record

ReviewBiological research2016

Carotid body chemoreceptors, sympathetic neural activation, and cardiometabolic disease.

Rodrigo Iturriaga, Rodrigo Del Rio, Juan Idiaquez, Virend K Somers

Registry-linked trialAbstract readReview
In one paragraph

Review in Biological research, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT06116357 (The Safety and Efficacy of Carotid Body Modulation Therapy in Hypertensive Patients by External Ultrasonic Micro-bubble), which is not on this map. Cited by 54 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
54citing papers in PubMed, 1 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT06116357 nanot yet recruitingnot on this mapstarted 2023, after this paper: background citation

The Safety and Efficacy of Carotid Body Modulation Therapy in Hypertensive Patients by External Ultrasonic Micro-bubble

TypeinterventionalSponsorJing HuangRan2023 to 2026Enrolled200ConditionsPrimary HypertensionArmsExternal Ultrasonic Micro-bubble
3 · Its place in the literature

Who cites it

54 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. Review
  3. Emerging roles of spexin in cardiovascular homeostasis.Frontiers in cardiovascular medicine · 2026
    Review
  4. Review
  5. Article
  6. Review
  7. Review
  8. Article
  9. Article
  10. Approach the Patient With Obstructive Sleep Apnea and Obesity.The Journal of clinical endocrinology and metabolism · 2024
    Article
  11. Review
  12. Article
  13. Review
  14. Article
  15. Carotid Body and Cell Therapy.Advances in anatomy, embryology, and cell biology · 2023
    Article
  16. Carotid Body Dysfunction and Mechanisms of Disease.Advances in anatomy, embryology, and cell biology · 2023
    Article
  17. Article
  18. Article
  19. Article
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Rodrigo IturriagaLaboratorio de Neurobiología, Facultad de Ciencias Biológicas, Pontificia Universidad Católica de Chile, Santiago, Chile. riturriaga@bio.puc.cl.
Rodrigo Del RioLaboratory of Cardiorespiratory Control, Centro de Investigación Biomédica, Universidad Autónoma de Chile, Santiago, Chile. rodrigo.delrio@uautonoma.cl.
Juan IdiaquezCatedra de Neurología, Escuela de Medicina, Universidad de Valparaíso, Valparaíso, Chile. idiaquez@123.cl.
Virend K SomersDivision of Cardiovascular Diseases, Mayo Clinic, Rochester, MN, USA. somers.virend@mayo.edu.

Funding

CARDIOVASCULAR DISEASE MECHANISMS IN SLEEP APNEAR01HL065176 · MAYO CLINIC COLL OF MEDICINE, ROCHESTER · 1999 to 2005
$1.6M
NHLBI NIH HHS R01 HL065176NHLBI NIH HHS R01 HL65176
6 · The paper itself

Abstract

The carotid body (CB) is the main peripheral chemoreceptor that senses the arterial PO2, PCO2 and pH. In response to hypoxemia, hypercapnia and acidosis, carotid chemosensory discharge elicits reflex respiratory, autonomic and cardiovascular adjustments. The classical construct considers the CB as the main peripheral oxygen sensor, triggering reflex physiological responses to acute hypoxemia and facilitating the ventilatory acclimation to chronic hypoxemia at high altitude. However, a growing body of experimental evidence supports the novel concept that an abnormally enhanced CB chemosensory input to the brainstem contributes to overactivation of the sympathetic nervous system, and consequent pathology. Indeed, the CB has been implicated in several diseases associated with increases in central sympathetic outflow. These include hypertension, heart failure, sleep apnea, chronic obstructive pulmonary disease and metabolic syndrome. Indeed, ablation of the CB has been proposed for the treatment of severe and resistant hypertension in humans. In this review, we will analyze and discuss new evidence supporting an important role for the CB chemoreceptor in the progression of autonomic and cardiorespiratory alterations induced by heart failure, obstructive sleep apnea, chronic obstructive pulmonary disease and metabolic syndrome.

Indexed as

Carotid BodyHeart FailureHumansMetabolic DiseasesPulmonary Disease, Chronic ObstructiveRisk FactorsSleep Apnea, ObstructiveSympathetic Nervous System

Identifiers

PMID26920146
PMCPMC4768417

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.