Evidence map›Paper›PMID 26965314›Full record

ReviewLipids in health and disease2016

Gene polymorphisms associated with non-alcoholic fatty liver disease and coronary artery disease: a concise review.

Xiao-Lin Li, Jian-Qing Sui, Lin-Lin Lu, Nan-Nan Zhang, Xin Xu, Quan-Yong Dong, Yong-Ning Xin, Shi-Ying Xuan

Open access · goldAbstract readReview
In one paragraph

Review in Lipids in health and disease, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 25 papers.

0numbers the graph read from it
0cells of the map it votes in
25citing papers in PubMed
4.3field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

25 citing papers in PubMed, 50 citations in OpenAlex.

  1. Review
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  6. Associations ofWorld journal of hepatology · 2024
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  8. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 3 institutions in 1 country.

Xiao-Lin LiDepartment of Gastroenterology, Qingdao Municipal Hospital, Dalian Medical University, Qingdao, 266011, China.
Jian-Qing SuiDepartment of Gastroenterology, Qingdao Municipal Hospital, Qingdao, 266011, China.
Lin-Lin LuDigestive Disease Key Laboratory of Qingdao, Qingdao, 266071, China.
Nan-Nan ZhangDepartment of Gastroenterology, Qingdao Municipal Hospital, Dalian Medical University, Qingdao, 266011, China.
Xin XuDepartment of Gastroenterology, Qingdao Municipal Hospital, Dalian Medical University, Qingdao, 266011, China.
Quan-Yong DongDepartment of Gastroenterology, Qingdao Municipal Hospital, Dalian Medical University, Qingdao, 266011, China.
Yong-Ning XinDepartment of Gastroenterology, Qingdao Municipal Hospital, Dalian Medical University, Qingdao, 266011, China. xinyongning@163.com.
Shi-Ying XuanDepartment of Gastroenterology, Qingdao Municipal Hospital, Dalian Medical University, Qingdao, 266011, China. xuansydxy@163.com.
Qingdao University · CNDalian Medical University · CNQingdao Municipal Hospital · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Non-alcoholic fatty liver disease (NAFLD) is a common chronic liver disease which represents a wide spectrum of hepatic damage. Several studies have reported that NAFLD is a strong independent risk factor for coronary artery disease (CAD). And patients with NAFLD are at higher risk and suggested undergoperiodic cardiovascular risk assessment. Cardiovascular disease (CVD) is responsible for the main cause of death in patients with NAFLD, and is mostly influenced by genetic factors. Both NAFLD and CAD are heterogeneous disease. Common pathways involved in the pathogenesis of NAFLD and CAD includes insulin resistance (IR), atherogenic dyslipidemia, subclinical inflammation, oxidative stress, etc. Genomic characteristics of these two diseases have been widely studied, further research about the association of these two diseases draws attention. The gene polymorphisms of adiponectin-encoding gene (ADIPOQ), leptin receptor (LEPR), apolipoprotein C3 (APOC3), peroxisome proliferator-activated receptors (PPAR), sterol regulatory elementbinding proteins (SREBP), transmembrane 6 superfamily member 2 (TM6SF2), microsomal triglyceride transfer protein (MTTP), tumor necrosis factors-alpha (TNF-α) and manganese superoxide dismutase (MnSOD) have been reported to be related to NAFLD and CAD. In this review, we aimed to provide an overview of recent insights into the genetic basis of NAFLD and CAD.

Indexed as

Polymorphism, GeneticAdiponectinApolipoprotein C-IIICarrier ProteinsCoronary Artery DiseaseHumansMembrane ProteinsNon-alcoholic Fatty Liver DiseasePeroxisome Proliferator-Activated ReceptorsReceptors, LeptinSterol Regulatory Element Binding ProteinsSuperoxide DismutaseSuperoxide Dismutase 2Tumor Necrosis Factor-alphaAdiponectinADIPOQ protein, humanApolipoprotein C-IIICarrier ProteinsLEPR protein, humanMembrane Proteinsmicrosomal triglyceride transfer proteinPeroxisome Proliferator-Activated ReceptorsReceptors, LeptinSterol Regulatory Element Binding ProteinsSuperoxide DismutaseSuperoxide Dismutase 2TM6SF2 protein, humanTumor Necrosis Factor-alphaCoronary artery diseaseGene pathogenesisGenetic polymorphismsNon-alcoholic fatty liver disease

Identifiers

PMID26965314
PMCPMC4785616
OpenAlexW2296700616

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.