Evidence map›Paper›PMID 26968954›Full record

ArticleOncotarget2016

ChIP-on-chip analysis of thyroid hormone-regulated genes and their physiological significance.

I-Hsiao Chung, Hsuan Liu, Yang-Hsiang Lin, Hsiang-Cheng Chi, Ya-Hui Huang, Chang-Ching Yang, Chau-Ting Yeh, Bertrand Chin-Ming Tan, Kwang-Huei Lin

Open access · diamondAbstract read
In one paragraph

Article in Oncotarget, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 13 papers.

0numbers the graph read from it
0cells of the map it votes in
13citing papers in PubMed
1.6field-weighted citation impact, top 17% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

13 citing papers in PubMed, 23 citations in OpenAlex.

  1. Article
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  5. The Role of hsa-miR-21 and Its Target Genes Involved in Nasopharyngeal Carcinoma.Asian Pacific journal of cancer prevention : APJCP · 2021
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 2 institutions in 1 country.

I-Hsiao ChungDepartment of Biochemistry, College of Medicine, Chang Gung University, Taoyuan, Taiwan.
Hsuan LiuDepartment of Biochemistry, College of Medicine, Chang Gung University, Taoyuan, Taiwan.
Yang-Hsiang LinDepartment of Biochemistry, College of Medicine, Chang Gung University, Taoyuan, Taiwan.
Hsiang-Cheng ChiDepartment of Biochemistry, College of Medicine, Chang Gung University, Taoyuan, Taiwan.
Ya-Hui HuangLiver Research Center, Chang Gung Memorial Hospital, Linkou, Taoyuan, Taiwan.
Chang-Ching YangMolecular Medicine Research Center, Chang Gung University, Taoyuan, Taiwan.
Chau-Ting YehLiver Research Center, Chang Gung Memorial Hospital, Linkou, Taoyuan, Taiwan.
Bertrand Chin-Ming TanMolecular Medicine Research Center, Chang Gung University, Taoyuan, Taiwan.
Kwang-Huei LinDepartment of Biochemistry, College of Medicine, Chang Gung University, Taoyuan, Taiwan.
Chang Gung University · TWChang Gung Memorial Hospital · TW

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Triiodothyronine (T3) and its receptor (TR) modulate several physiological processes, including cell development, proliferation, differentiation and metabolism. The regulatory mechanism of T3/TR involves binding to the thyroid hormone response element (TRE) within the target gene promoter. However, the number of target genes directly regulated by TRα1 and the specific pathways of TR-regulated target genes remain largely unknown. Here, we expressed TRα1 in a HepG2 cell line and used chromatin immunoprecipitation coupled with microarray to determine the genes that are directly regulated by TRα1 and also involved in cell metabolism and proliferation. Our analysis identified E74-like factor 2 (ELF2), a transcription factor associated with tumor growth, as a direct target downregulated by T3/TR. Overexpression of ELF2 enhanced tumor cell proliferation, and conversely, its knockdown suppressed tumor growth. Additionally, ELF2 restored the proliferative ability of hepatoma cells inhibited by T3/TR. Our findings collectively support a potential role of T3/TR in tumor growth inhibition through regulation of ELF2.

Indexed as

Carcinoma, HepatocellularChromatin ImmunoprecipitationGene Expression Regulation, NeoplasticHep G2 CellsHumansLiver NeoplasmsReceptors, Thyroid HormoneTranscription FactorsTriiodothyronineELF2 protein, humanReceptors, Thyroid HormoneTranscription FactorsTriiodothyroninecell growthChIP-on-chipELF2thyroid hormone receptor

Identifiers

PMID26968954
PMCPMC5008372
OpenAlexW2317479182

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.