Evidence mapPaperPMID 27558696Full record

ArticleLipids in health and disease2016

Lipid composition of circulating multiple-modified low density lipoprotein.

E R Zakiev, V N Sukhorukov, A A Melnichenko, I A Sobenin, E A Ivanova, A N Orekhov

Open access · goldAbstract read
In one paragraph

Article in Lipids in health and disease, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 23 papers.

0numbers the graph read from it
0cells of the map it votes in
23citing papers in PubMed
1.2field-weighted citation impact, top 21% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

23 citing papers in PubMed, 45 citations in OpenAlex.

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  10. Sialic acid as the potential link between lipid metabolism and inflammation in the pathogenesis of atherosclerosis.Brazilian journal of medical and biological research = Revista brasileira de pesquisas medicas e biologica · 2023
    Review
  11. Review
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  13. Article
  14. Cinnamaldehyde Mitigates Atherosclerosis Induced by High-Fat DietOxidative medicine and cellular longevity · 2022
    Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 3 institutions in 2 countries.

E R ZakievLaboratory of Angiopathology, Institute of General Pathology and Pathophysiology, 125315, Moscow, Russia.
V N SukhorukovLaboratory of Angiopathology, Institute of General Pathology and Pathophysiology, 125315, Moscow, Russia.
A A MelnichenkoLaboratory of Angiopathology, Institute of General Pathology and Pathophysiology, 125315, Moscow, Russia.
I A SobeninLaboratory of Angiopathology, Institute of General Pathology and Pathophysiology, 125315, Moscow, Russia.
E A IvanovaDepartment of Development and Regeneration, KU Leuven, 3000, Leuven, Belgium.
A N OrekhovLaboratory of Angiopathology, Institute of General Pathology and Pathophysiology, 125315, Moscow, Russia. a.h.opexob@gmail.com.
Research Institute of General Pathology and Pathophysiology, the Russian Academy of Medical Sciences · RUInstitute for Atherosclerosis Research · RUKU Leuven · BE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Atherogenic modified low- density lipoprotein (LDL) induces pronounced accumulation of cholesterol and lipids in the arterial wall, while native LDL seems to lack such capability. Therefore, modified LDL appears to be a major causative agent in the pathogenesis of atherosclerosis. Possible modifications of LDL particles include changes in size and density, desialylation, oxidation and acquisition of negative charge. Total LDL isolated from pooled plasma of patients with coronary atherosclerosis, as well as from healthy subjects contains two distinct subfractions: normally sialylated LDL and desialylated LDL, which can be isolated by binding to a lectin affinity column. We called the desialylated LDL subfraction circulating modified LDL (cmLDL). In this study, we focused on lipid composition of LDL particles, analysing the total LDL preparation and two LDL subfractions: cmLDL and native LDL. The composition of LDL was studied using thin-layer chromatography. We found that cmLDL subfraction had decreased levels of free and esterified cholesterol, triglycerides, phospholipids (except for lysophosphatidylcholine) and sphingomyelin in comparison to native LDL. On the other hand, levels of mono-, and diglycerides, lysophosphatidylcholine and free fatty acids were higher in cmLDL than in native LDL. Our study demonstrated that lipid composition of cmLDL from atherosclerotic patients was altered in comparison to healthy subjects. In particular, phospholipid content was decreased, and free fatty acids levels were increased in cmLDL. This strengthens the hypothesis of multiple modification of LDL particles in the bloodstream and underscores the clinical importance of desialylated LDL as a possible marker of atherosclerosis progression.

Indexed as

AdultCase-Control StudiesCholesterolCholesterol EstersCoronary Artery DiseaseFemaleHumansLipoproteins, LDLLysophosphatidylcholinesMaleMiddle AgedPhosphatidylcholinesPhosphatidylethanolaminesSphingomyelinsCholesterolCholesterol EstersLipoproteins, LDLLysophosphatidylcholinesPhosphatidylcholinesphosphatidylethanolaminePhosphatidylethanolaminesSphingomyelinsAtherosclerosisLDLLDL modificationLipid composition of LDLLow density lipoprotein

Identifiers

PMID27558696
PMCPMC4995786
OpenAlexW2510604554

What Socratic holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.