Evidence mapPaperPMID 28246388Full record

ArticleScientific reports2017

Serum microRNA-1 and microRNA-133a levels reflect myocardial steatosis in uncomplicated type 2 diabetes.

D de Gonzalo-Calvo, R W van der Meer, L J Rijzewijk, J W A Smit, E Revuelta-Lopez, L Nasarre, J C Escola-Gil, H J Lamb, V Llorente-Cortes

Open access · goldAbstract read
In one paragraph

Article in Scientific reports, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 80 papers.

0numbers the graph read from it
0cells of the map it votes in
80citing papers in PubMed
7.3field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

80 citing papers in PubMed, 126 citations in OpenAlex.

  1. Unifying and unique roles of non-coding RNA biomarkers in liver and heart fibrosis.Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie · 2026
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  12. Mechanistic insight into the role of cardiac-enriched microRNAs in diabetic heart injury.American journal of physiology. Heart and circulatory physiology · 2025
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20 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 6 institutions in 3 countries.

D de Gonzalo-CalvoBiomedical Research Institute Sant Pau (IIB Sant Pau), Barcelona, Spain. DGonzalo@santpau.cat.
R W van der MeerDepartment of Radiology, Leiden University Medical Center, Leiden, The Netherlands.
L J RijzewijkDepartment of Medicine, Kantonsspital Baden AG, Baden, Switzerland.
J W A SmitDepartment of Internal Medicine, University Medical Center Nijmegen, Nijmegen, The Netherlands.
E Revuelta-LopezBiomedical Research Institute Sant Pau (IIB Sant Pau), Barcelona, Spain.
L NasarreBiomedical Research Institute Sant Pau (IIB Sant Pau), Barcelona, Spain.
J C Escola-GilIIB Sant Pau, Departament de Bioquímica i Biologia Molecular, Universitat Autònoma de Barcelona-CIBER de Diabetes y Enfermedades Metabolicas Asociadas, Barcelona, Spain.
H J LambDepartment of Radiology, Leiden University Medical Center, Leiden, The Netherlands.
V Llorente-CortesBiomedical Research Institute Sant Pau (IIB Sant Pau), Barcelona, Spain. CLlorente@santpau.cat.
Instituto de Salud Carlos III · ESLeiden University Medical Center · NLCentro de Investigación Biomédica en Red Diabetes y Enfermedades Metabólicas Asociadas · ESCentro de Investigación en Red en Enfermedades Cardiovasculares · ESKantonsspital Baden · CHRadboud University Medical Center · NL

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Using in vitro, in vivo and patient-based approaches, we investigated the potential of circulating microRNAs (miRNAs) as surrogate biomarkers of myocardial steatosis, a hallmark of diabetic cardiomyopathy. We analysed the cardiomyocyte-enriched miRNA signature in serum from patients with well-controlled type 2 diabetes and with verified absence of structural heart disease or inducible ischemia, and control volunteers of the same age range and BMI (N = 86), in serum from a high-fat diet-fed murine model, and in exosomes from lipid-loaded HL-1 cardiomyocytes. Circulating miR-1 and miR-133a levels were robustly associated with myocardial steatosis in type 2 diabetes patients, independently of confounding factors in both linear and logistic regression analyses (P < 0.050 for all models). Similar to myocardial steatosis, miR-133a levels were increased in type 2 diabetes patients as compared with healthy subjects (P < 0.050). Circulating miR-1 and miR-133a levels were significantly elevated in high-fat diet-fed mice (P < 0.050), which showed higher myocardial steatosis, as compared with control animals. miR-1 and miR-133a levels were higher in exosomes released from lipid-loaded HL-1 cardiomyocytes (P < 0.050). Circulating miR-1 and miR-133a are independent predictors of myocardial steatosis. Our results highlight the value of circulating miRNAs as diagnostic tools for subclinical diabetic cardiomyopathy.

Indexed as

AgedAnimalsBiomarkersDiabetes Mellitus, Type 2Diabetic CardiomyopathiesDiet, High-FatExosomesHumansMaleMiceMice, Inbred C57BLMicroRNAsMiddle AgedMyocardiumMyocytes, CardiacProton Magnetic Resonance SpectroscopyBiomarkersMicroRNAsMIRN133 microRNA, humanMIRN1 microRNA, human

Identifiers

PMID28246388
PMCPMC5428350
OpenAlexW2593006132

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.