Evidence mapPaperPMID 28296232Full record

ArticleCardiovascular therapeutics2017

Inhibitors of soluble epoxide hydrolase minimize ischemia-reperfusion-induced cardiac damage in normal, hypertensive, and diabetic rats.

Oliul Islam, Prashanth Patil, Sumanta K Goswami, Rema Razdan, Mohammed N Inamdar, Mohammed Rizwan, Jubin Mathew, Bora Inceoglu, Kin S Stephen Lee, Sung H Hwang and 1 more

Open access · bronzeAbstract read
In one paragraph

Article in Cardiovascular therapeutics, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 19 papers.

0numbers the graph read from it
0cells of the map it votes in
19citing papers in PubMed
2.2field-weighted citation impact, top 14% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

19 citing papers in PubMed, 25 citations in OpenAlex.

  1. Article
  2. Unraveling "Hot Phases" of Arrhythmogenic Cardiomyopathy.JACC. Basic to translational science · 2025
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  6. Anthracycline derivatives inhibit cardiac CYP2J2.Journal of inorganic biochemistry · 2022
    Article
  7. Article
  8. Article
  9. Review
  10. Review
  11. Article
  12. Review
  13. Drug-Target Residence Time AffectsACS central science · 2019
    Article
  14. Article
  15. Article
  16. Article
  17. Article
  18. Regulation of CYP2J2 and EET Levels in Cardiac Disease and Diabetes.International journal of molecular sciences · 2018
    Review
  19. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 2 institutions in 3 countries.

Oliul IslamDepartment of Pharmacology, Al-Ameen College of Pharmacy, Bangalore, Karnataka, India.
Prashanth PatilDepartment of Pharmacology, Al-Ameen College of Pharmacy, Bangalore, Karnataka, India.
Sumanta K GoswamiDepartment of Pharmacology, Al-Ameen College of Pharmacy, Bangalore, Karnataka, India.
Rema RazdanDepartment of Pharmacology, Al-Ameen College of Pharmacy, Bangalore, Karnataka, India.
Mohammed N InamdarDepartment of Pharmacology, Al-Ameen College of Pharmacy, Bangalore, Karnataka, India.
Mohammed RizwanDepartment of Pharmacology, Al-Ameen College of Pharmacy, Bangalore, Karnataka, India.
Jubin MathewDepartment of Pharmacology, Al-Ameen College of Pharmacy, Bangalore, Karnataka, India.
Bora InceogluDepartment of Entomology and Nematology, and Comprehensive Cancer Center, University of California, Davis, CA, USA.
Kin S Stephen LeeDepartment of Entomology and Nematology, and Comprehensive Cancer Center, University of California, Davis, CA, USA.
Sung H HwangDepartment of Entomology and Nematology, and Comprehensive Cancer Center, University of California, Davis, CA, USA.
Bruce D HammockDepartment of Entomology and Nematology, and Comprehensive Cancer Center, University of California, Davis, CA, USA.ORCID http://orcid.org/0000-0003-1408-8317
Al-Ameen College of Pharmacy · INSylvester Comprehensive Cancer Center · US

Funding

TRANSPORT, TRANSFORMATION &REMEDIATION OF VOCS IN THE VADOSE ZONE &GROUND WATERP42ES004699 · UNIVERSITY OF CALIFORNIA DAVIS · 1987 to 2005
$14.0M
HYDROLYTIC ENZYMES IN THE METABOLISM OF TOXINSR01ES002710 · UNIVERSITY OF CALIFORNIA DAVIS · 1985 to 1997
NIEHS NIH HHS P42 ES004699NIEHS NIH HHS R01 ES002710
6 · The paper itself

Abstract

aimWe designed a study to evaluate the cardioprotective effect of two soluble epoxide hydrolase (sEH) inhibitors, 1-(1-propanoylpiperidin-4-yl)-3-(4-trifluoromethoxy)phenyl)urea (TPPU) and trans-4-{4-[3-(4-trifluoromethoxyphenyl)-ureido]cyclohexyloxy}benzoic acid (t-TUCB), in ischemia-reperfusion (IR) model.

methodsCardioprotective effects of the sEH inhibitors were evaluated against IR-induced myocardial damage in hearts from normal, hypertensive, and diabetic rats using Langendorff's apparatus. In addition, the effect of sEH inhibitors on endothelial function was evaluated in vitro and ex vivo using isolated rat thoracic aorta.

resultsIschemia-reperfusion (IR) increased the myocardial damage in hearts from normal rats. IR-induced myocardial damage was augmented in hearts isolated from hypertensive and diabetic rats. Myocardial damage as evident from increase in the activities of lactate dehydrogenase (LDH) and creatine kinase-MB (CK-MB) in heart perfusate was associated with significant decrease in the heart rate and developed tension, and increase in the resting tension in isolated heart. Both sEH inhibitors protected the heart in normal, hypertensive, and diabetic rats subjected to IR injury. The sEH inhibitor t-TUCB relaxed phenylephrine precontracted aorta from normal rats. Relaxant effect of acetylcholine (ACh) was reduced in aortas from diabetic and hypertensive rats compared to normal rats. Pretreatment of sEH inhibitors to diabetic and hypertensive rats increased relaxant effect of ACh on aortas isolated from these rats.

conclusionsProphylactic treatment with sEH inhibitors decreased myocardial damage due to IR, hypertension and diabetes, and decreased endothelial dysfunction created by diabetes and hypertension. Therefore, inhibitors of sEH are useful probes to study cardiovascular pathology, and inhibition of the sEH is a potential approach in the management of IR-induced cardiac damage and endothelial dysfunction-related cardiovascular disorders.

Indexed as

AnimalsAorta, ThoracicBenzoatesDiabetes Mellitus, ExperimentalDiabetes Mellitus, Type 1Dose-Response Relationship, DrugEnzyme InhibitorsEpoxide HydrolasesHeart RateHypertensionIsolated Heart PreparationMaleMyocardial ContractionMyocardial InfarctionMyocardial Reperfusion InjuryMyocardium1-trifluoromethoxyphenyl-3-(1-propionylpiperidine-4-yl)urea4-(4-(3-(4-trifluoromethoxy-phenyl)ureido)cyclohexyloxy)benzoic acidBenzoatesEnzyme InhibitorsEPHX2 protein, ratEpoxide HydrolasesPhenylurea CompoundsPiperidinesEndothelial dysfunctionIschemia/reperfusion-induced myocardial damageLangendorff's apparatusLisinoprilMetforminsEH inhibitor TPPU and t-TUCB

Identifiers

PMID28296232
PMCPMC5597338
OpenAlexW2600765728

What Socratic holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.