Evidence mapPaperPMID 28314859Full record

ArticleJournal of lipid research2017

Effects of CETP inhibition with anacetrapib on metabolism of VLDL-TG and plasma apolipoproteins C-II, C-III, and E.

John S Millar, Michael E Lassman, Tiffany Thomas, Rajasekhar Ramakrishnan, Patricia Jumes, Richard L Dunbar, Emil M deGoma, Amanda L Baer, Wahida Karmally, Daniel S Donovan and 11 more

Open access · hybridAbstract read
In one paragraph

Article in Journal of lipid research, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
3.4field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 23 citations in OpenAlex.

  1. Trial
  2. Article
  3. Article
  4. Article
  5. Review
  6. Review
  7. Review
  8. Drugs for rare disorders.British journal of clinical pharmacology · 2017
    Article
  9. Article
  10. Anacetrapib as a potential cardioprotective strategy.Drug design, development and therapy · 2017
    Review
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

21 authors at 4 institutions in 1 country.

John S MillarUniversity of Pennsylvania, Philadelphia, PA 19104 jsmillar@upenn.edu gr2104@cumc.columbia.edu.
Michael E LassmanMerck & Co., Inc., Kenilworth, NJ 07033.
Tiffany ThomasColumbia University, New York, NY 10032.
Rajasekhar RamakrishnanColumbia University, New York, NY 10032.
Patricia JumesMerck & Co., Inc., Kenilworth, NJ 07033.
Richard L DunbarUniversity of Pennsylvania, Philadelphia, PA 19104.
Emil M deGomaUniversity of Pennsylvania, Philadelphia, PA 19104.
Amanda L BaerUniversity of Pennsylvania, Philadelphia, PA 19104.
Wahida KarmallyColumbia University, New York, NY 10032.
Daniel S DonovanColumbia University, New York, NY 10032.
Hashmi RafeekUniversity of Pennsylvania, Philadelphia, PA 19104.
John A WagnerMerck & Co., Inc., Kenilworth, NJ 07033.
Stephen HolleranColumbia University, New York, NY 10032.
Joseph ObunikeNew York City College of Technology, CUNY, Brooklyn, NY 11201.
Yang LiuMerck & Co., Inc., Kenilworth, NJ 07033.
Soumia AoujilUniversity of Pennsylvania, Philadelphia, PA 19104.
Taylor StandifordUniversity of Pennsylvania, Philadelphia, PA 19104.
David E GutsteinMerck & Co., Inc., Kenilworth, NJ 07033.
Henry N GinsbergColumbia University, New York, NY 10032.
Daniel J RaderUniversity of Pennsylvania, Philadelphia, PA 19104.
Gissette Reyes-SofferColumbia University, New York, NY 10032 jsmillar@upenn.edu gr2104@cumc.columbia.edu.
University of Pennsylvania · USColumbia University · USMerck & Co., Inc., Rahway, NJ, USA (United States) · USNew York City College of Technology · US

Funding

Phenotyping Genetic Disorders of Hepatic Lipid and Lipoprotein Metabolism in Cells, Mice, and MenR35HL135833 · NHLBI · COLUMBIA UNIVERSITY HEALTH SCIENCES · PI HENRY N GINSBERG · 2022 to 2023
$2.0M
NCRR NIH HHS UL1 RR024134NHLBI NIH HHS R35 HL135833
6 · The paper itself

Abstract

Cholesteryl ester transfer protein (CETP) mediates the transfer of HDL cholesteryl esters for triglyceride (TG) in VLDL/LDL. CETP inhibition, with anacetrapib, increases HDL-cholesterol, reduces LDL-cholesterol, and lowers TG levels. This study describes the mechanisms responsible for TG lowering by examining the kinetics of VLDL-TG, apoC-II, apoC-III, and apoE. Mildly hypercholesterolemic subjects were randomized to either placebo (N = 10) or atorvastatin 20 mg/qd (N = 29) for 4 weeks (period 1) followed by 8 weeks of anacetrapib, 100 mg/qd (period 2). Following each period, subjects underwent stable isotope metabolic studies to determine the fractional catabolic rates (FCRs) and production rates (PRs) of VLDL-TG and plasma apoC-II, apoC-III, and apoE. Anacetrapib reduced the VLDL-TG pool on a statin background due to an increased VLDL-TG FCR (29%;

Indexed as

Apolipoprotein C-IIApolipoprotein C-IIIApolipoproteinsApolipoproteins ECholesterol Ester Transfer ProteinsDrug InteractionsFemaleHumansHydroxymethylglutaryl-CoA Reductase InhibitorsLipoproteins, VLDLMaleMiddle AgedOxazolidinonesTriglyceridesanacetrapibApolipoprotein C-IIApolipoprotein C-IIIApolipoproteinsApolipoproteins ECholesterol Ester Transfer ProteinsHydroxymethylglutaryl-CoA Reductase InhibitorsLipoproteins, VLDLOxazolidinonesTriglyceridesvery low density lipoprotein triglyceridecholesteryl ester transfer proteindrug therapykineticslipoprotein metabolismplasma lipid transfer proteinsstatinstriglyceridevery low density lipoprotein

Identifiers

PMID28314859
PMCPMC5454510
OpenAlexW2596919763

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.