Evidence map›Paper›PMID 28345280›Full record

ArticleJournal of neuroscience research2017

Otoprotective effects of mouse nerve growth factor in DBA/2J mice with early-onset progressive hearing loss.

Qingzhu Wang, Hongchun Zhao, Tihua Zheng, Wenjun Wang, Xiaolin Zhang, Andi Wang, Bo Li, Yanfei Wang, Qingyin Zheng

Open access · greenAbstract read
In one paragraph

Article in Journal of neuroscience research, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed
0.8field-weighted citation impact, top 33% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed, 19 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 2 institutions in 2 countries.

Qingzhu WangTransformative Otology and Neuroscience Center, Binzhou Medical University, Yantai, 264003, Shandong, PR China.ORCID 0000-0002-2685-2303
Hongchun ZhaoDepartment of Otolaryngology/Head and Neck Surgery, Institute of Otolaryngology, Affiliated Hospital of Binzhou Medical University, Binzhou, 256600, Shandong, PR China.
Tihua ZhengTransformative Otology and Neuroscience Center, Binzhou Medical University, Yantai, 264003, Shandong, PR China.
Wenjun WangTransformative Otology and Neuroscience Center, Binzhou Medical University, Yantai, 264003, Shandong, PR China.
Xiaolin ZhangDepartment of Otolaryngology/Head and Neck Surgery, Institute of Otolaryngology, Affiliated Hospital of Binzhou Medical University, Binzhou, 256600, Shandong, PR China.
Andi WangDepartment of Otolaryngology-HNS, Case Western Reserve University, Cleveland, OH, 44106, USA.
Bo LiTransformative Otology and Neuroscience Center, Binzhou Medical University, Yantai, 264003, Shandong, PR China.
Yanfei WangTransformative Otology and Neuroscience Center, Binzhou Medical University, Yantai, 264003, Shandong, PR China.
Qingyin ZhengDepartment of Otolaryngology-HNS, Case Western Reserve University, Cleveland, OH, 44106, USA.
Binzhou Medical University · CNCase Western Reserve University · US

Funding

Deciphering the Usher I protein interactome using a geneticapproachR01DC015111 · NIDCD · CASE WESTERN RESERVE UNIVERSITY · PI ZHENG, QING Y · 2016 to 2020
$1.8M
NIDCD NIH HHS R01 DC015111
6 · The paper itself

Abstract

As it displays progressive hair-cell loss and degeneration of spiral ganglion neurons (SGNs) characterized by early-onset progressive hearing loss (ePHL), DBA/2J is an inbred mouse strain widely used in hearing research. Mouse nerve growth factor (mNGF), as a common exogenous nerve growth factor (NGF), has been studied extensively for its ability to promote neuronal survival and growth. To determine whether mNGF can ameliorate progressive hearing loss (PHL) in DBA/2J mice, saline or mNGF was given to DBA/2J mice of either sex by daily intramuscular injection from the 1st to the 9th week after birth. At 5, 7, and 9 weeks of age, in comparison with vehicle groups, mNGF groups experienced decreased auditory-evoked brainstem response (ABR) thresholds and increased distortion product otoacoustic emission (DPOAE) amplitudes, the prevention of hair cell loss, and the inhibition of apoptosis of SGNs. Downregulation of Bak/Bax and Caspase genes and proteins in cochleae of mice receiving the mNGF treatment was detected by real-time PCR, Western blot, and immunohistochemistry. This suggests that the Bak-dependent mitochondrial apoptosis pathway may be involved in the otoprotective mechanism of mNGF in progressive hearing loss of DBA/2J mice. Our results demonstrate that mNGF can act as an otoprotectant in the DBA/2J mice for the early intervention of PHL and, thus, could become of great value in clinical applications. © 2017 Wiley Periodicals, Inc.

Indexed as

AnimalsEvoked Potentials, Auditory, Brain StemFemaleHair Cells, Auditory, InnerHearing LossMaleMiceMice, Inbred DBANerve Growth FactorNeuroprotective AgentsSpiral GanglionNerve Growth FactorNeuroprotective Agentsapoptosishair cellsmitochondrial pathwaymouse modelmouse nerve growth factorprogressive hearing lossspiral ganglion neurons

Identifiers

PMID28345280
PMCPMC5561496
OpenAlexW2601261090

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.