Evidence mapPaperPMID 28760142Full record

ReviewBMC medicine2017

The renin-angiotensin system: a possible new target for depression.

João Vian, Círia Pereira, Victor Chavarria, Cristiano Köhler, Brendon Stubbs, João Quevedo, Sung-Wan Kim, André F Carvalho, Michael Berk, Brisa S Fernandes

Open access · goldAbstract readReview
In one paragraph

Review in BMC medicine, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 62 papers, 4 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
62citing papers in PubMed, 4 pooled it
4.2field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

62 citing papers in PubMed, 4 syntheses or guidelines pooled it, 136 citations in OpenAlex.

  1. Pooled it
  2. Pooled it
  3. Imaging genetics paradigms in depression research: Systematic review and meta-analysis.Progress in neuro-psychopharmacology & biological psychiatry · 2018
    Pooled it
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  5. Trial
  6. Trial
  7. Trial
  8. Article
  9. Article
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  12. Review
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  17. Comparative Study on the Neuroprotective Effects of Perindopril and Benazepril in Experimentally-induced Chronic Mild Stress in Rats.Journal of neuroimmune pharmacology : the official journal of the Society on NeuroImmune Pharmacology · 2025
    Article
  18. Article
  19. Review
  20. Article

2 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 8 institutions in 7 countries.

João VianPsychiatry and Mental Health Department, Centro Hospitalar Lisboa Norte, Lisbon, Portugal.
Círia PereiraPsychiatry and Mental Health Department, Centro Hospitalar Lisboa Norte, Lisbon, Portugal.
Victor ChavarriaInstitut de Neuropsiquiatria i Adiccions (INAD), Parc de Salut Mar (PSM), Barcelona, Spain.
Cristiano KöhlerTranslational Psychiatry Research Group and Department of Clinical Medicine, Faculty of Medicine, Federal University of Ceará, Fortaleza, Brazil.
Brendon StubbsHealth, Social Care and Education, Anglia Ruskin University, Chelmsford, UK.
João QuevedoTranslational Psychiatry Program, Department of Psychiatry and Behavioral Sciences, McGovern Medical School, The University of Texas Health Science Center at Houston (UTHealth), Houston, TX, USA.
Sung-Wan KimDepartment of Psychiatry, Chonnam National University Medical School, Gwangju, Republic of Korea.
André F CarvalhoTranslational Psychiatry Research Group and Department of Clinical Medicine, Faculty of Medicine, Federal University of Ceará, Fortaleza, Brazil.
Michael BerkDeakin University, IMPACT Strategic Research Centre, School of Medicine, University Hospital Geelong, Barwon Health, Geelong, VIC, Australia.
Brisa S FernandesDeakin University, IMPACT Strategic Research Centre, School of Medicine, University Hospital Geelong, Barwon Health, Geelong, VIC, Australia. brisasf@gmail.com.
Centro Hospitalar Lisboa Norte · PTUniversidade Federal do Ceará · BRBarwon Health · AUChonnam National University · KRKing's College London · GBOrygen Youth Health · AUParc de Salut · ESThe University of Texas Health Science Center at Houston · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Depression remains a debilitating condition with an uncertain aetiology. Recently, attention has been given to the renin-angiotensin system. In the central nervous system, angiotensin II may be important in multiple pathways related to neurodevelopment and regulation of the stress response. Studies of drugs targeting the renin-angiotensin system have yielded promising results. Here, we review the potential beneficial effects of angiotensin blockers in depression and their mechanisms of action. Drugs blocking the angiotensin system have efficacy in several animal models of depression. While no randomised clinical trials were found, case reports and observational studies showed that angiotensin-converting enzyme inhibitors or angiotensin receptor blockers had positive effects on depression, whereas other antihypertensive agents did not. Drugs targeting the renin-angiotensin system act on inflammatory pathways implicated in depression. Both preclinical and clinical data suggest that these drugs possess antidepressant properties. In light of these results, angiotensin system-blocking agents offer new horizons in mood disorder treatment.

Indexed as

Renin-Angiotensin SystemAngiotensin-Converting Enzyme InhibitorsAngiotensin IIAngiotensin Receptor AntagonistsAnimalsAntidepressive AgentsAntihypertensive AgentsDepressionDrug Delivery SystemsHumansAngiotensin-Converting Enzyme InhibitorsAngiotensin IIAngiotensin Receptor AntagonistsAntidepressive AgentsAntihypertensive AgentsAngiotensinAngiotensin-converting enzyme inhibitorsAngiotensin receptor blockersATR1ATR2DepressionInflammationMasPsychiatryRenin–angiotensin system

Identifiers

PMID28760142
PMCPMC5537940
OpenAlexW2742134405

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.