Evidence mapPaperPMID 28820053Full record

ReviewCurrent neuropharmacology2018

Neurobiological Mechanisms of Stress Resilience and Implications for the Aged Population.

Charlene Faye, Josephine C Mcgowan, Christine A Denny, Denis J David

Open access · bronzeAbstract readReview
In one paragraph

Review in Current neuropharmacology, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 65 papers.

0numbers the graph read from it
0cells of the map it votes in
65citing papers in PubMed
5.5field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

65 citing papers in PubMed, 135 citations in OpenAlex.

  1. Review
  2. Energy Allocation Resilience and Endocrine Integration.International journal of molecular sciences · 2026
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5 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 2 countries.

Charlene FayeCESP/UMR-S 1178, Univ. Paris-Sud, Fac Pharmacie, Inserm, Universite Paris-Saclay, 92296 Chatenay-Malabry, France.
Josephine C McgowanDoctoral Program in Neurobiology and Behavior, Columbia University, New York, NY, United States.
Christine A DennyDepartment of Psychiatry, Columbia University, New York, NY, United States.
Denis J DavidCESP/UMR-S 1178, Univ. Paris-Sud, Fac Pharmacie, Inserm, Universite Paris-Saclay, 92296 Chatenay-Malabry, France.
Centre de recherche en Epidémiologie et Santé des Populations · FRColumbia University · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundStress is a common reaction to an environmental adversity, but a dysregulation of the stress response can lead to psychiatric illnesses such as major depressive disorder (MDD), post-traumatic stress disorder (PTSD), and anxiety disorders. Yet, not all individuals exposed to stress will develop psychiatric disorders; those with enhanced stress resilience mechanisms have the ability to adapt successfully to stress without developing persistent psychopathology. Notably, the potential to enhance stress resilience in at-risk populations may prevent the onset of stress-induced psychiatric disorders. This novel idea has prompted a number of studies probing the mechanisms of stress resilience and how it can be manipulated.

methodsHere, we review the neurobiological factors underlying stress resilience, with particular focus on the serotoninergic (5-HT), glutamatergic, and γ-Aminobutyric acid (GABA) systems, as well as the hypothalamic-pituitary axis (HPA) in rodents and in humans. Finally, we discuss stress resiliency in the context of aging, as the likelihood of mood disorders increases in older adults.

resultsInterestingly, increased resiliency has been shown to slow aging and improved overall health and quality of life. Research in the neurobiology of stress resilience, particularly throughout the aging process, is a nascent, yet, burgeoning field.

conclusionOverall, we consider the possible methods that may be used to induce resilient phenotypes, prophylactically in at-risk populations, such as in military personnel or in older MDD patients. Research in the mechanisms of stress resilience may not only elucidate novel targets for antidepressant treatments, but also provide novel insight about how to prevent these debilitating disorders from developing.

Indexed as

Resilience, PsychologicalAgingAnimalsHumansStress, Psychologicalagingglutamatehypothalamic–pituitary–adrenal axisresilienceserotoninStressγ-aminobutyric acid

Identifiers

PMID28820053
PMCPMC5843978
OpenAlexW2752875392

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.