Evidence mapPaperPMID 29288332Full record

ArticlePflugers Archiv : European journal of physiology2018

Essential role for EGFR tyrosine kinase and ER stress in myocardial infarction in type 2 diabetes.

Vishal Mali, Samuel Haddox, Corey Hornersmith, Khalid Matrougui, Souad Belmadani

Abstract read
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In one paragraph

Article in Pflugers Archiv : European journal of physiology, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 13 papers.

0numbers the graph read from it
0cells of the map it votes in
13citing papers in PubMed
0.8field-weighted citation impact, top 30% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

13 citing papers in PubMed, 25 citations in OpenAlex.

  1. Review
  2. Article
  3. Review
  4. Molecular targets and mechanisms ofWorld journal of diabetes · 2023
    Article
  5. Article
  6. Review
  7. Article
  8. Review
  9. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Vishal MaliDepartment of Physiological Sciences, EVMS, Norfolk, VA, 23501, USA.
Samuel HaddoxDepartment of Physiological Sciences, EVMS, Norfolk, VA, 23501, USA.
Corey HornersmithDepartment of Physiological Sciences, EVMS, Norfolk, VA, 23501, USA.
Khalid MatrouguiDepartment of Physiological Sciences, EVMS, Norfolk, VA, 23501, USA.
Souad BelmadaniDepartment of Physiological Sciences, EVMS, Norfolk, VA, 23501, USA. belmads@evms.edu.
Department of Physiological Sciences · RU

Funding

NHLBI NIH HHS R01 HL095566
6 · The paper itself

Abstract

We previously reported that EGFR tyrosine kinase (EGFRtk) activity and endoplasmic reticulum (ER) stress are enhanced in type 2 diabetic (T2D) mice and cause vascular dysfunction. In the present study, we determined the in vivo contribution of EGFRtk and ER stress in acute myocardial infarction induced by acute ischemia (40 min)-reperfusion (24 h) (I/R) injury in T2D (db

Indexed as

Endoplasmic Reticulum StressAnimalsApoptosisCardiotonic AgentsDiabetes Mellitus, Type 2Diabetic CardiomyopathiesErbB ReceptorsMacrophagesMaleMAP Kinase Signaling SystemMiceMyocardial InfarctionMyocytes, CardiacNeutrophilsProtein Kinase InhibitorsQuinazolinesCardiotonic AgentsEGFR protein, mouseErbB ReceptorsProtein Kinase InhibitorsQuinazolinesRTKI cpdTaurochenodeoxycholic AcidTyrphostinsursodoxicoltaurineEGFR tyrosine kinaseER stressMyocardial infarctionType 2 diabetes

Identifiers

PMID29288332
OpenAlexW2779206059

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.