Evidence mapPaperPMID 29580581Full record

ReviewAdvances in chronic kidney disease2018

Acute Kidney Injury and Progression of Diabetic Kidney Disease.

Samuel Mon-Wei Yu, Joseph V Bonventre

Open access · greenAbstract readReview
In one paragraph

Review in Advances in chronic kidney disease, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 87 papers.

0numbers the graph read from it
0cells of the map it votes in
87citing papers in PubMed
12.4field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

87 citing papers in PubMed, 178 citations in OpenAlex.

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  3. Dickkopf-3 (DKK3) in Urine Identifies Patients with Short-Term Risk of eGFR Loss.Journal of the American Society of Nephrology : JASN · 2018
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27 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

Samuel Mon-Wei YuDepartment of Medicine, Jacobi Medical Center/Albert Einstein College of Medicine, Bronx, NY; and Renal Division, Department of Medicine, Brigham and Women's Hospital, Harvard Medical School, Boston, MA.
Joseph V BonventreDepartment of Medicine, Jacobi Medical Center/Albert Einstein College of Medicine, Bronx, NY; and Renal Division, Department of Medicine, Brigham and Women's Hospital, Harvard Medical School, Boston, MA. Electronic address: joseph_bonventre@hms.harvard.edu.
Harvard University · US

Funding

Mechanisms of Ischemic Kidney Injury and RepairR01DK039773 · MASSACHUSETTS GENERAL HOSPITAL · 1987 to 2025
$2.3M
KIDNEY INJURY MOLECULE-1 IN EPITHELIAL REPAIRR01DK072381 · BRIGHAM AND WOMEN'S HOSPITAL · 2005 to 2025
$919k
MECHANISMS OF ISCHEMIC INJURY AND REPAIRR37DK039773 · MASSACHUSETTS GENERAL HOSPITAL · 1993 to 2001
$427k
NIDDK NIH HHS R01 DK039773NIDDK NIH HHS R01 DK072381NIDDK NIH HHS R37 DK039773
6 · The paper itself

Abstract

Diabetic kidney disease, commonly termed diabetic nephropathy (DN), is the most common cause of end-stage kidney disease (ESKD) worldwide. The characteristic histopathology of DN includes glomerular basement membrane thickening, mesangial expansion, nodular glomerular sclerosis, and tubulointerstitial fibrosis. Diabetes is associated with a number of metabolic derangements, such as reactive oxygen species overproduction, hypoxic state, mitochondrial dysfunction, and inflammation. In the past few decades, our knowledge of DN has advanced considerably although much needs to be learned. The traditional paradigm of glomerulus-centered pathophysiology has expanded to the tubule-interstitium, the immune response and inflammation. Biomarkers of proximal tubule injury have been shown to correlate with DN progression, independent of traditional glomerular injury biomarkers such as albuminuria. In this review, we summarize mechanisms of increased susceptibility to acute kidney injury in diabetes mellitus and the roles played by many kidney cell types to facilitate maladaptive responses leading to chronic and end-stage kidney disease.

Indexed as

Acute Kidney InjuryDiabetes Mellitus, Type 2Diabetic NephropathiesKidney Failure, ChronicDisease ProgressionHumansAcute kidney injuryDiabetic kidney diseaseEnd-stage kidney disease

Identifiers

PMID29580581
PMCPMC5898826
OpenAlexW2792844735

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.