Evidence mapPaperPMID 30008106Full record

Trial reportEuropean journal of nutrition2019

The insulinotropic effect of a high-protein nutrient preload is mediated by the increase of plasma amino acids in type 2 diabetes.

Domenico Tricò, Silvia Frascerra, Simona Baldi, Alessandro Mengozzi, Lorenzo Nesti, Andrea Mari, Andrea Natali

Registry-linked trialAbstract readRandomized Controlled Trial
PubMed Publisher
In one paragraph

Trial report in European journal of nutrition, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT02342834 (Mechanisms Through Which a Small Protein and Lipid Preload Improves Glucose Tolerance in Subjects With Impaired Glucose Homeostasis), which is not on this map. Cited by 22 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
22citing papers in PubMed, 2 pooled it
4.0field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT02342834 nacompletednot on this map

Mechanisms Through Which a Small Protein and Lipid Preload Improves Glucose Tolerance in Subjects With Impaired Glucose Homeostasis

TypeinterventionalSponsorAzienda Ospedaliero, Universitaria PisanaRan2015 to 2020Enrolled35ConditionsDiabetes Mellitus, Type 2ArmsSmall mixed protein and lipid meal
3 · Its place in the literature

Who cites it

22 citing papers in PubMed, 2 syntheses or guidelines pooled it, 31 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 2 institutions in 1 country.

Domenico TricòDepartment of Clinical and Experimental Medicine, University of Pisa, Via Roma 67, 56126, Pisa, Italy. domenico.trico@for.unipi.it.ORCID http://orcid.org/0000-0002-7633-1346
Silvia FrascerraDepartment of Clinical and Experimental Medicine, University of Pisa, Via Roma 67, 56126, Pisa, Italy.
Simona BaldiDepartment of Clinical and Experimental Medicine, University of Pisa, Via Roma 67, 56126, Pisa, Italy.
Alessandro MengozziDepartment of Clinical and Experimental Medicine, University of Pisa, Via Roma 67, 56126, Pisa, Italy.
Lorenzo NestiDepartment of Clinical and Experimental Medicine, University of Pisa, Via Roma 67, 56126, Pisa, Italy.
Andrea MariInstitute of Neuroscience, National Research Council, Padua, Italy.
Andrea NataliDepartment of Clinical and Experimental Medicine, University of Pisa, Via Roma 67, 56126, Pisa, Italy.
University of Pisa · ITNational Research Council · IT

Funding

University of Pisa Fondi di Ateneo
6 · The paper itself

Abstract

aimsEating protein before carbohydrate reduces postprandial glucose excursions by enhancing insulin and glucagon-like peptide-1 (GLP-1) secretion in type 2 diabetes (T2D). We tested the hypothesis that this insulinotropic effect depends on the elevation of plasma amino acids (AA) after the digestion of food protein.

methodsIn 16 T2D patients, we measured plasma AA levels through the course of two 75-g oral glucose tolerance tests (OGTT) preceded by either 500-ml water or a high-protein nutrient preload (50-g Parmesan cheese, one boiled egg, and 300-ml water). Changes in beta cell function were evaluated by measuring and modelling plasma glucose, insulin, and C-peptide through the OGTT. Changes in incretin hormone secretion were assessed by measuring plasma GLP-1.

resultsPlasma AA levels were 24% higher after the nutrient preload (p < 0.0001). This increment was directly proportional to both the enhancement of beta cell function (r = 0.58, p = 0.02) and the plasma GLP-1 gradients (r = 0.57, p = 0.02) produced by the nutrient preload. Among single AA, glutamine showed the strongest correlation with changes in beta cell function (r = 0.61, p = 0.01), while leucine showed the strongest correlation with GLP-1 responses (r = 0.74, p = 0.001).

conclusionsThe elevation of circulating AA that occurs after a high-protein nutrient preload is associated with an enhancement of beta cell function and GLP-1 secretion in T2D. Manipulating the meal sequence of nutrient ingestion may reduce postprandial hyperglycaemia through a direct and GLP-1-mediated stimulation of insulin secretion by plasma AA. TRIAL REGISTRATION NUMBER: NCT02342834.

Indexed as

AdultAgedAmino AcidsCross-Over StudiesDiabetes Mellitus, Type 2Dietary ProteinsFemaleGlucagon-Like Peptide 1HumansInsulinMaleMiddle AgedPostprandial PeriodAmino AcidsDietary ProteinsGlucagon-Like Peptide 1InsulinBeta cell functionBranched-chain amino acidsGLP-1Glucose toleranceInsulin secretionType 2 diabetes

Identifiers

PMID30008106
OpenAlexW2883564695

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.