Evidence mapPaperPMID 30175474Full record

ArticlePhysiological reports2018

Hydrogen sulfide improves postischemic neoangiogenesis in the hind limb of cystathionine-β-synthase mutant mice via PPAR-γ/VEGF axis.

Avisek Majumder, Mahavir Singh, Akash K George, Jyotirmaya Behera, Neetu Tyagi, Suresh C Tyagi

Open access · goldAbstract read
In one paragraph

Article in Physiological reports, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 29 papers.

0numbers the graph read from it
0cells of the map it votes in
29citing papers in PubMed
3.2field-weighted citation impact, top 8% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

29 citing papers in PubMed, 44 citations in OpenAlex.

  1. Significance of GSH and HRedox report : communications in free radical research · 2026
    Review
  2. Review
  3. HBiomolecules · 2024
    Article
  4. Review
  5. Review
  6. Review
  7. Review
  8. Review
  9. Clinical Potential of Hydrogen Sulfide in Peripheral Arterial Disease.International journal of molecular sciences · 2023
    Review
  10. Article
  11. Review
  12. Article
  13. Article
  14. Review
  15. Article
  16. Review
  17. Hydrogen Sulfide Actions in the Vasculature.Comprehensive Physiology · 2021
    Article
  18. Article
  19. Article
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 1 institution in 1 country.

Avisek MajumderDepartment of Physiology, University of Louisville School of Medicine, Louisville, Kentucky, 40202, USA.
Mahavir SinghDepartment of Physiology, University of Louisville School of Medicine, Louisville, Kentucky, 40202, USA.
Akash K GeorgeDepartment of Physiology, University of Louisville School of Medicine, Louisville, Kentucky, 40202, USA.
Jyotirmaya BeheraDepartment of Physiology, University of Louisville School of Medicine, Louisville, Kentucky, 40202, USA.
Neetu TyagiDepartment of Physiology, University of Louisville School of Medicine, Louisville, Kentucky, 40202, USA.
Suresh C TyagiDepartment of Physiology, University of Louisville School of Medicine, Louisville, Kentucky, 40202, USA.
University of Louisville · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Neoangiogenesis is a fundamental process which helps to meet energy requirements, tissue growth, and wound healing. Although previous studies showed that Peroxisome proliferator-activated receptor (PPAR-γ) regulates neoangiogenesis via upregulation of vascular endothelial growth factor (VEGF), and both VEGF and PPAR-γ expressions were inhibited during hyperhomocysteinemic (HHcy), whether these two processes could trigger pathological effects in skeletal muscle via compromising neoangiogenesis has not been studied yet. Unfortunately, there are no treatment options available to date for ameliorating HHcy-mediated neoangiogenic defects. Hydrogen sulfide (H

Indexed as

Signal TransductionAnimalsCystathionine beta-SynthaseHindlimbHydrogen SulfideHypoxia-Inducible Factor 1, alpha SubunitIschemiaMaleMiceMice, Inbred C57BLMuscle, SkeletalMutationNeovascularization, PhysiologicNitric Oxide Synthase Type IIIPPAR gammaVascular Endothelial Growth Factor ACystathionine beta-SynthaseHif1a protein, mouseHydrogen SulfideHypoxia-Inducible Factor 1, alpha SubunitNitric Oxide Synthase Type IIIPPAR gammaVascular Endothelial Growth Factor AAngiogenesishydrogen sulfidestress response

Identifiers

PMID30175474
PMCPMC6119702
OpenAlexW2890203796

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.