Evidence map›Paper›PMID 30208317›Full record

ArticleCell reports2018

Sarcolipin Signaling Promotes Mitochondrial Biogenesis and Oxidative Metabolism in Skeletal Muscle.

Santosh K Maurya, Jose L Herrera, Sanjaya K Sahoo, Felipe C G Reis, Rick B Vega, Daniel P Kelly, Muthu Periasamy

Open access · goldAbstract read
In one paragraph

Article in Cell reports, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 70 papers.

0numbers the graph read from it
0cells of the map it votes in
70citing papers in PubMed
5.2field-weighted citation impact, top 4% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

70 citing papers in PubMed, 125 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
  4. Review
  5. Effects of electromyostimulation on sarcolipin and CaBiochemistry and biophysics reports · 2026
    Article
  6. Article
  7. Article
  8. Article
  9. Sarcolipin but not phospholamban responds toFrontiers in physiology · 2026
    Article
  10. Article
  11. Article
  12. Article
  13. Article
  14. Article
  15. Article
  16. Article
  17. Article
  18. Article
  19. Article
  20. Review

10 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 3 institutions in 1 country.

Santosh K MauryaSanford Burnham Prebys Medical Discovery Institute, Orlando, FL 32827, USA.
Jose L HerreraSanford Burnham Prebys Medical Discovery Institute, Orlando, FL 32827, USA.
Sanjaya K SahooSanford Burnham Prebys Medical Discovery Institute, Orlando, FL 32827, USA.
Felipe C G ReisSanford Burnham Prebys Medical Discovery Institute, Orlando, FL 32827, USA.
Rick B VegaSanford Burnham Prebys Medical Discovery Institute, Orlando, FL 32827, USA.
Daniel P KellyPerelman School of Medicine at the University of Pennsylvania, Philadelphia, PA, USA.
Muthu PeriasamySanford Burnham Prebys Medical Discovery Institute, Orlando, FL 32827, USA. Electronic address: mperiasamy@sbpdiscovery.org.
Discovery Institute · USSanford Burnham Prebys Medical Discovery Institute · USUniversity of Pennsylvania · US

Funding

REGULATION OF FATTY ACID OXIDATION DURING CARDIAC GROWTHR01HL058493 · NHLBI · WASHINGTON UNIVERSITY · PI KELLY, DANIEL PATRICK · 1998 to 2024
$10.6M
Molecular Control of Muscle Fuel MetabolismR01DK045416 · NIDDK · WASHINGTON UNIVERSITY · PI KELLY, DANIEL PATRICK · 1996 to 2020
$8.1M
RECRUITMENT OF SKELETAL MUSCLE BASED ON NON-SHIVERING THERMOGENESIS IN HEALTH AND DISEASER01DK098240 · NIDDK · UNIVERSITY OF CENTRAL FLORIDA · PI PERIASAMY, MUTHU · 2013 to 2017
$1.9M
NHLBI NIH HHS R01 HL058493NIDDK NIH HHS R01 DK045416NIDDK NIH HHS R01 DK098240
6 · The paper itself

Abstract

The major objective of this study was to understand the molecular basis of how sarcolipin uncoupling of SERCA regulates muscle oxidative metabolism. Using genetically engineered sarcolipin (SLN) mouse models and primary muscle cells, we demonstrate that SLN plays a crucial role in mitochondrial biogenesis and oxidative metabolism in muscle. Loss of SLN severely compromised muscle oxidative capacity without affecting fiber-type composition. Mice overexpressing SLN in fast-twitch glycolytic muscle reprogrammed mitochondrial phenotype, increasing fat utilization and protecting against high-fat diet-induced lipotoxicity. We show that SLN affects cytosolic Ca

Indexed as

AnimalsCalcium-Calmodulin-Dependent Protein Kinase Type 2Calcium SignalingCells, CulturedEnergy MetabolismMiceMice, KnockoutMitochondriaMuscle Fibers, SkeletalMuscle ProteinsMuscle, SkeletalObesityOrganelle BiogenesisPeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaProteolipidsSignal TransductionCalcium-Calmodulin-Dependent Protein Kinase Type 2Muscle ProteinsPeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaProteolipidssarcolipinCa(2+) signalingCamKIIlipotoxicitymitochondrial biogenesisoxidative metabolismPGC1αprimary muscle myotubessarcolipinSERCAskeletal muscle

Identifiers

PMID30208317
PMCPMC6481681
OpenAlexW2891145500

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.