Evidence mapPaperPMID 30496210Full record

Trial reportPloS one2018

Alterations in bone turnover markers in patients with noncardio-embolic ischemic stroke.

K Mathold, P Wanby, L Brudin, S P Von, M Carlsson

Abstract readClinical TrialComparative Study
In one paragraph

Trial report in PloS one, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed, 2 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 2 syntheses or guidelines pooled it.

  1. Pooled it
  2. Pooled it
  3. Article
  4. Review
  5. Article
  6. Article
  7. Exploring the Role of Sclerostin as a Biomarker of Cardiovascular Disease and Mortality: A Scoping Review.International journal of environmental research and public health · 2022
    Article
  8. Review
  9. Crosstalk of Brain and Bone-Clinical Observations and Their Molecular Bases.International journal of molecular sciences · 2020
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

K MatholdDepartment of Geriatric Medicine, County Hospital of Kalmar, Kalmar, Sweden.ORCID 0000-0001-5974-9460
P WanbySection of Endocrinology, Department of Internal Medicine, County Hospital of Kalmar, Kalmar, Sweden.
L BrudinDepartment of Clinical Physiology, County Hospital of Kalmar, Kalmar, Sweden.
S P VonDepartment of Clinical Microbiology and Infectious Diseases, County Hospital of Kalmar, Kalmar, Sweden.
M CarlssonDepartment of Clinical Chemistry, County Hospital of Kalmar, Kalmar, Sweden.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundThe major cause of ischemic stroke is unstable or thrombogenic atherosclerotic plaques. Vascular calcification, a process that appears crucial for plaque stability, shares common features with bone formation. Many bone turnover proteins exhibit metabolic properties, but the evidence is conflicting regarding their possible involvement in vascular disease. Antibodies against sclerostin and dickkopf-1 are currently being evaluated as potential therapy for treating bone disorders. It is important to carefully assess the cardiovascular and metabolic effects of these proteins. The aim of the present study was to explore serum levels of bone turnover markers in patients with acute noncardio-embolic ischemic stroke in comparison with healthy controls.

methodsIn a cross-sectional study, we compared 48 patients aged ≥75 years with noncardio-embolic ischemic stroke and 46 healthy controls. Serum levels of dickkopf-1, sclerostin, osteoprotegerin, osteopontin and osteocalcin were determined by Luminex technique.

resultsWe found clearly increased serum levels of osteoprotegerin, sclerostin, dickkopf-1 and osteopontin in patients with stroke compared with healthy controls. No difference was seen in serum levels of osteocalcin between the two groups.

conclusionOur findings strengthen the hypothesis of bone turnover markers being involved in vascular disease. Whether these proteins can be used as candidate markers for increased stroke risk or prognostic biomarkers remains to be further elucidated.

Indexed as

Bone RemodelingAdaptor Proteins, Signal TransducingAgedAged, 80 and overBiomarkersBone Morphogenetic ProteinsBrain IschemiaFemaleGenetic MarkersHumansIntercellular Signaling Peptides and ProteinsMaleOsteocalcinOsteopontinOsteoprotegerinPlaque, AtheroscleroticAdaptor Proteins, Signal TransducingBGLAP protein, humanBiomarkersBone Morphogenetic ProteinsDKK1 protein, humanGenetic MarkersIntercellular Signaling Peptides and ProteinsOsteocalcinOsteopontinOsteoprotegerinSOST protein, humanSPP1 protein, humanTNFRSF11B protein, human

Identifiers

PMID30496210
PMCPMC6264871

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.