Evidence mapPaperPMID 30509997Full record

ArticleProceedings of the National Academy of Sciences of the United States of America2018

Inflammation in the hippocampus affects IGF1 receptor signaling and contributes to neurological sequelae in rheumatoid arthritis.

Karin M E Andersson, Caroline Wasén, Lina Juzokaite, Lovisa Leifsdottir, Malin C Erlandsson, Sofia T Silfverswärd, Anna Stokowska, Marcela Pekna, Milos Pekny, Kjell Olmarker and 3 more

Open access · bronzeAbstract read
In one paragraph

Article in Proceedings of the National Academy of Sciences of the United States of America, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 30 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
30citing papers in PubMed, 1 pooled it
2.6field-weighted citation impact, top 9% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

30 citing papers in PubMed, 1 synthesis or guideline pooled it, 51 citations in OpenAlex.

  1. Pooled it
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  3. Observational
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors at 2 institutions in 2 countries.

Karin M E AnderssonDepartment of Rheumatology and Inflammation Research, Institute of Medicine, University of Gothenburg, 40530 Gothenburg, Sweden; karin.andersson@rheuma.gu.se maria.bokarewa@rheuma.gu.se.ORCID 0000-0002-9475-3040
Caroline WasénDepartment of Rheumatology and Inflammation Research, Institute of Medicine, University of Gothenburg, 40530 Gothenburg, Sweden.ORCID 0000-0002-8585-8190
Lina JuzokaiteDepartment of Rheumatology and Inflammation Research, Institute of Medicine, University of Gothenburg, 40530 Gothenburg, Sweden.ORCID 0000-0002-6797-5078
Lovisa LeifsdottirDepartment of Rheumatology and Inflammation Research, Institute of Medicine, University of Gothenburg, 40530 Gothenburg, Sweden.ORCID 0000-0002-1559-3094
Malin C ErlandssonDepartment of Rheumatology and Inflammation Research, Institute of Medicine, University of Gothenburg, 40530 Gothenburg, Sweden.ORCID 0000-0003-1100-7577
Sofia T SilfverswärdDepartment of Rheumatology and Inflammation Research, Institute of Medicine, University of Gothenburg, 40530 Gothenburg, Sweden.ORCID 0000-0002-6641-2759
Anna StokowskaCenter for Brain Repair and Rehabilitation, Department of Clinical Neuroscience, Institute of Neuroscience and Physiology, University of Gothenburg, 40530 Gothenburg, Sweden.ORCID 0000-0001-5237-3341
Marcela PeknaCenter for Brain Repair and Rehabilitation, Department of Clinical Neuroscience, Institute of Neuroscience and Physiology, University of Gothenburg, 40530 Gothenburg, Sweden.ORCID 0000-0003-2734-8237
Milos PeknyCenter for Brain Repair and Rehabilitation, Department of Clinical Neuroscience, Institute of Neuroscience and Physiology, University of Gothenburg, 40530 Gothenburg, Sweden.ORCID 0000-0003-1607-8075
Kjell OlmarkerMusculoskeletal Research, Department of Medical Chemistry and Cell Biology, Institute of Biomedicine, University of Gothenburg, 40530 Gothenburg, Sweden.ORCID 0000-0002-3565-2047
Rolf A HeckemannMed Tech West, Sahlgrenska University Hospital, 41345 Gothenburg, Sweden.ORCID 0000-0003-3582-3683
Marie KalmDepartment of Pharmacology, Institute of Neuroscience and Physiology, University of Gothenburg, 40530 Gothenburg, Sweden.ORCID 0000-0002-0680-5040
Maria I BokarewaDepartment of Rheumatology and Inflammation Research, Institute of Medicine, University of Gothenburg, 40530 Gothenburg, Sweden; karin.andersson@rheuma.gu.se maria.bokarewa@rheuma.gu.se.ORCID 0000-0001-9424-5145
University of Gothenburg · SESahlgrenska University Hospital · SE

Funding

Medical Research Council MR/L01632X/1
6 · The paper itself

Abstract

Rheumatoid arthritis (RA) is an inflammatory joint disease with a neurological component including depression, cognitive deficits, and pain, which substantially affect patients' quality of daily life. Insulin-like growth factor 1 receptor (IGF1R) signaling is one of the factors in RA pathogenesis as well as a known regulator of adult neurogenesis. The purpose of this study was to investigate the association between IGF1R signaling and the neurological symptoms in RA. In experimental RA, we demonstrated that arthritis induced enrichment of IBA1

Indexed as

AdultAgedAnimalsArthritis, RheumatoidBrainDentate GyrusDisease Models, AnimalFemaleGene ExpressionHippocampusHumansInflammationInsulin Receptor Substrate ProteinsInsulin ResistanceMaleMiceIGF1R protein, humanInsulin Receptor Substrate ProteinsReceptor, IGF Type 1Receptors, SomatomedinarthritishippocampusIGF1 receptorMRIpain

Identifiers

PMID30509997
PMCPMC6305002
OpenAlexW2902708622

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.