Evidence map›Paper›PMID 30511639›Full record

ArticleeLife2018

Endothelial-specific FoxO1 depletion prevents obesity-related disorders by increasing vascular metabolism and growth.

Martina Rudnicki, Ghoncheh Abdifarkosh, Emmanuel Nwadozi, Sofhia V Ramos, Armin Makki, Diane M Sepa-Kishi, Rolando B Ceddia, Christopher Gr Perry, Emilie Roudier, Tara L Haas

Abstract read
In one paragraph

Article in eLife, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 31 papers.

0numbers the graph read from it
0cells of the map it votes in
31citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

31 citing papers in PubMed.

  1. Trial
  2. mInternational journal of molecular medicine · 2026
    Review
  3. Article
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  8. Review
  9. Article
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  13. iScience · 2023
    Article
  14. Article
  15. Article
  16. Article
  17. Review
  18. Article
  19. Article
  20. Quantitative Methods to Assess Adipose Vasculature.Methods in molecular biology (Clifton, N.J.) · 2022
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors.

Martina RudnickiSchool of Kinesiology and Health Science and the Muscle Health Research Centre, York University, Toronto, Canada.ORCID 0000-0002-7863-5044
Ghoncheh AbdifarkoshSchool of Kinesiology and Health Science and the Muscle Health Research Centre, York University, Toronto, Canada.
Emmanuel NwadoziSchool of Kinesiology and Health Science and the Muscle Health Research Centre, York University, Toronto, Canada.
Sofhia V RamosSchool of Kinesiology and Health Science and the Muscle Health Research Centre, York University, Toronto, Canada.
Armin MakkiSchool of Kinesiology and Health Science and the Muscle Health Research Centre, York University, Toronto, Canada.
Diane M Sepa-KishiSchool of Kinesiology and Health Science and the Muscle Health Research Centre, York University, Toronto, Canada.
Rolando B CeddiaSchool of Kinesiology and Health Science and the Muscle Health Research Centre, York University, Toronto, Canada.
Christopher Gr PerrySchool of Kinesiology and Health Science and the Muscle Health Research Centre, York University, Toronto, Canada.
Emilie RoudierSchool of Kinesiology and Health Science and the Muscle Health Research Centre, York University, Toronto, Canada.ORCID 0000-0003-1014-6620
Tara L HaasSchool of Kinesiology and Health Science and the Muscle Health Research Centre, York University, Toronto, Canada.ORCID 0000-0001-8559-9574

Funding

CIHR MOP-130491
6 · The paper itself

Abstract

Impaired angiogenesis is a hallmark of metabolically dysfunctional adipose tissue in obesity. However, the underlying mechanisms restricting angiogenesis within this context remain ill-defined. Here, we demonstrate that induced endothelial-specific depletion of the transcription factor Forkhead Box O1 (FoxO1) in male mice led to increased vascular density in adipose tissue. Upon high-fat diet feeding, endothelial cell FoxO1-deficient mice exhibited even greater vascular remodeling in the visceral adipose depot, which was paralleled with a healthier adipose tissue expansion, higher glucose tolerance and lower fasting glycemia concomitant with enhanced lactate levels. Mechanistically, FoxO1 depletion increased endothelial proliferative and glycolytic capacities by upregulating the expression of glycolytic markers, which may account for the improvements at the tissue level ultimately impacting whole-body glucose metabolism. Altogether, these findings reveal the pivotal role of FoxO1 in controlling endothelial metabolic and angiogenic adaptations in response to high-fat diet and a contribution of the endothelium to whole-body energy homeostasis.

Indexed as

AnimalsDiet, High-FatEndothelium, VascularForkhead Box Protein O1GlucoseGlycolysisHomeostasisIntra-Abdominal FatMaleMice, KnockoutMicrovesselsModels, BiologicalMuscle, SkeletalObesityOrgan SizeOrgan SpecificityForkhead Box Protein O1Foxo1 protein, mouseGlucoseTriglyceridesangiogenesiscell biologydiet-induced obesityendothelial cellsFoxO1glycolysishuman biologymedicinemouse

Identifiers

PMID30511639
PMCPMC6279348

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.