Evidence mapPaperPMID 30670722Full record

ArticleScientific reports2019

Using a Targeted Proteomics Chip to Explore Pathophysiological Pathways for Incident Diabetes- The Malmö Preventive Project.

John Molvin, Manan Pareek, Amra Jujic, Olle Melander, Lennart Råstam, Ulf Lindblad, Bledar Daka, Margrét Leósdóttir, Peter M Nilsson, Michael H Olsen and 1 more

Open access · goldAbstract read
In one paragraph

Article in Scientific reports, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 20 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
20citing papers in PubMed, 1 pooled it
3.1field-weighted citation impact, top 8% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

20 citing papers in PubMed, 1 synthesis or guideline pooled it, 43 citations in OpenAlex.

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  11. Plasma Galectin-4 Levels Are Increased after Stroke in Mice and Humans.International journal of molecular sciences · 2023
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 5 institutions in 3 countries.

John MolvinDepartment of Clinical Sciences, Lund University, Clinical Research Center, Malmö, Sweden. johnmolvin@gmail.com.
Manan PareekCardiology Section, Department of Internal Medicine, Holbæk Hospital, Holbæk, Denmark.
Amra JujicDepartment of Clinical Sciences, Lund University, Clinical Research Center, Malmö, Sweden.
Olle MelanderDepartment of Clinical Sciences, Lund University, Clinical Research Center, Malmö, Sweden.
Lennart RåstamDepartment of Clinical Sciences, Lund University, Clinical Research Center, Malmö, Sweden.
Ulf LindbladInstitute of Medicine, Department of Public Health and Community Medicine, Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden.
Bledar DakaInstitute of Medicine, Department of Public Health and Community Medicine, Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden.
Margrét LeósdóttirDepartment of Clinical Sciences, Lund University, Clinical Research Center, Malmö, Sweden.
Peter M NilssonDepartment of Clinical Sciences, Lund University, Clinical Research Center, Malmö, Sweden.
Michael H OlsenCardiology Section, Department of Internal Medicine, Holbæk Hospital, Holbæk, Denmark.
Martin MagnussonDepartment of Clinical Sciences, Lund University, Clinical Research Center, Malmö, Sweden.
Lund University · SEUniversity of Gothenburg · SEHolbæk Sygehus · DKOdense University Hospital · DKSkåne University Hospital · SE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Multiplex proteomic platforms provide excellent tools for investigating associations between multiple proteins and disease (e.g., diabetes) with possible prognostic, diagnostic, and therapeutic implications. In this study our aim was to explore novel pathophysiological pathways by examining 92 proteins and their association with incident diabetes in a population-based cohort (146 cases of diabetes versus 880 controls) followed over 8 years. After adjusting for traditional risk factors, we identified seven proteins associated with incident diabetes. Four proteins (Scavenger receptor cysteine rich type 1 protein M130, Fatty acid binding protein 4, Plasminogen activator inhibitor 1 and Insulin-like growth factor-binding protein 2) with a previously established association with incident diabetes and 3 proteins (Cathepsin D, Galectin-4, Paraoxonase type 3) with a novel association with incident diabetes. Galectin-4, with an increased risk of diabetes, and Paraoxonase type 3, with a decreased risk of diabetes, remained significantly associated with incident diabetes after adjusting for plasma glucose, implying a glucose independent association with diabetes.

Indexed as

AgedBiomarkersBlood GlucoseCase-Control StudiesDiabetes Mellitus, Type 2FemaleHumansImmunoassayIncidenceMaleMiddle AgedPrognosisProteomicsRisk FactorsSwedenBiomarkersBlood Glucose

Identifiers

PMID30670722
PMCPMC6342982
OpenAlexW2913560469

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.