Evidence mapPaperPMID 30768374Full record

ArticleFASEB journal : official publication of the Federation of American Societies for Experimental Biology2019

Aquaporin-3 deficiency slows cyst enlargement in experimental mouse models of autosomal dominant polycystic kidney disease.

Weiling Wang, Xiaoqiang Geng, Lei Lei, Yingli Jia, Yingjie Li, Hong Zhou, Alan S Verkman, Baoxue Yang

Abstract read
In one paragraph

Article in FASEB journal : official publication of the Federation of American Societies for Experimental Biology, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
2.0field-weighted citation impact, top 13% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 18 citations in OpenAlex.

  1. Review
  2. Article
  3. Article
  4. Review
  5. Aquaporins in Urinary System.Advances in experimental medicine and biology · 2023
    Article
  6. Article
  7. Clinical, cosmetic and investigational dermatology · 2022
    Article
  8. Article
  9. Article
  10. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 4 institutions in 2 countries.

Weiling WangDepartment of Pharmacology, School of Basic Medical Sciences, Peking University, Beijing, China.
Xiaoqiang GengDepartment of Pharmacology, School of Basic Medical Sciences, Peking University, Beijing, China.
Lei LeiDepartment of Pharmacology, School of Basic Medical Sciences, Peking University, Beijing, China.
Yingli JiaDepartment of Pharmacology, School of Basic Medical Sciences, Peking University, Beijing, China.
Yingjie LiDepartment of Pharmacology, School of Basic Medical Sciences, Peking University, Beijing, China.
Hong ZhouDepartment of Pharmacology, School of Basic Medical Sciences, Peking University, Beijing, China.
Alan S VerkmanDepartment of Medicine, University of California-San Francisco, San Francisco, California, USA.
Baoxue YangDepartment of Pharmacology, School of Basic Medical Sciences, Peking University, Beijing, China.
Peking University · CNState Key Laboratory of Natural and Biomimetic DrugsMinistry of Education of the People's Republic of China · CNUniversity of California, San Francisco · US

Funding

NIDDK NIH HHS R01 DK101373
6 · The paper itself

Abstract

Human autosomal dominant polycystic kidney disease (ADPKD) is characterized by bilateral renal cysts that lead to a decline in kidney function. Previous studies reported aquaporin (AQP)-3 expression in cysts derived from collecting ducts in ADPKD. To study the role of AQP3 in cyst development, we generated 2 polycystic kidney disease (PKD) mouse models: kidney-specific

Indexed as

Adenosine TriphosphateAMP-Activated Protein Kinase KinasesAnimalsAquaporin 3DogsFemaleGlucose Transporter Type 1Hypoxia-Inducible Factor 1, alpha SubunitKidney Tubules, CollectingMadin Darby Canine Kidney CellsMaleMiceMice, Inbred C57BLMitogen-Activated Protein Kinase 3Polycystic Kidney DiseasesProtein KinasesAdenosine TriphosphateAMP-Activated Protein Kinase KinasesAqp3 protein, mouseAquaporin 3Glucose Transporter Type 1Hif1a protein, mouseHypoxia-Inducible Factor 1, alpha SubunitMitogen-Activated Protein Kinase 3Protein KinasesSlc2a1 protein, mouseTOR Serine-Threonine KinasesTRPP Cation ChannelsADPKDAMPK/mTOR signalingglucose metabolismHIF1-αwater channel

Identifiers

PMID30768374
PMCPMC6463927
OpenAlexW2914206429

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.